Page last updated: 2024-08-21

thiazolidines and Autosomal Dominant Cerebellar Ataxia, Type II

thiazolidines has been researched along with Autosomal Dominant Cerebellar Ataxia, Type II in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Galeano, K; Kaczmarek, LK; Khare, S; Nick, HS; Nick, JA; Sampson, J; Subramony, SH; Waters, MF; Zhang, Y	1

Other Studies

1 other study(ies) available for thiazolidines and Autosomal Dominant Cerebellar Ataxia, Type II

Article	Year
C-terminal proline deletions in KCNC3 cause delayed channel inactivation and an adult-onset progressive SCA13 with spasticity. Cerebellum (London, England), 2018, Volume: 17, Issue:5 Topics: Adult; Animals; Bridged Bicyclo Compounds, Heterocyclic; CHO Cells; Cricetulus; Female; Humans; Male; Marine Toxins; Membrane Potentials; Muscle Spasticity; Phenotype; Sequence Deletion; Shaw Potassium Channels; Spinocerebellar Ataxias; Thiazolidines	2018

Article

Year

C-terminal proline deletions in KCNC3 cause delayed channel inactivation and an adult-onset progressive SCA13 with spasticity.

Cerebellum (London, England), 2018, Volume: 17, Issue:5

Topics: Adult; Animals; Bridged Bicyclo Compounds, Heterocyclic; CHO Cells; Cricetulus; Female; Humans; Male; Marine Toxins; Membrane Potentials; Muscle Spasticity; Phenotype; Sequence Deletion; Shaw Potassium Channels; Spinocerebellar Ataxias; Thiazolidines

2018