Page last updated: 2024-08-21

thiazoles and Muscular Weakness

thiazoles has been researched along with Muscular Weakness in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Ito, K; Kamiya, K; Matsuda, H; Nakata, Y; Oya, Y; Sano, T; Sato, N; Shigemoto, Y	1
Chan, DC; Chiang, CK; Chiu, CY; Liu, SH; Sheu, ML; Tsai, KS; Yang, RS; Yang, TH	1

Other Studies

2 other study(ies) available for thiazoles and Muscular Weakness

Article	Year
Massive accumulation of 11C-Pittsburg compound B in the occipital lobes of a patient with early-onset dementia accompanied by muscle weakness and hypertonicity. Annals of nuclear medicine, 2013, Volume: 27, Issue:10 Topics: Adult; Aniline Compounds; Benzothiazoles; Dementia; Female; Humans; Muscle Hypertonia; Muscle Weakness; Occipital Lobe; Positron-Emission Tomography; Thiazoles; Tomography, X-Ray Computed	2013
Advanced glycation end-products induce skeletal muscle atrophy and dysfunction in diabetic mice via a RAGE-mediated, AMPK-down-regulated, Akt pathway. The Journal of pathology, 2016, Volume: 238, Issue:3 Topics: Aged; AMP-Activated Protein Kinases; Animals; Atrophy; Case-Control Studies; Diabetes Mellitus; Diabetes Mellitus, Experimental; Down-Regulation; Glycation End Products, Advanced; Humans; Male; MAP Kinase Signaling System; Mice; Muscle Fatigue; Muscle Fibers, Skeletal; Muscle Proteins; Muscle Weakness; Muscle, Skeletal; Muscular Diseases; Receptor for Advanced Glycation End Products; Regeneration; Signal Transduction; SKP Cullin F-Box Protein Ligases; Thiazoles	2016

Article

Year

Massive accumulation of 11C-Pittsburg compound B in the occipital lobes of a patient with early-onset dementia accompanied by muscle weakness and hypertonicity.

Annals of nuclear medicine, 2013, Volume: 27, Issue:10

Topics: Adult; Aniline Compounds; Benzothiazoles; Dementia; Female; Humans; Muscle Hypertonia; Muscle Weakness; Occipital Lobe; Positron-Emission Tomography; Thiazoles; Tomography, X-Ray Computed

2013

Advanced glycation end-products induce skeletal muscle atrophy and dysfunction in diabetic mice via a RAGE-mediated, AMPK-down-regulated, Akt pathway.

The Journal of pathology, 2016, Volume: 238, Issue:3

Topics: Aged; AMP-Activated Protein Kinases; Animals; Atrophy; Case-Control Studies; Diabetes Mellitus; Diabetes Mellitus, Experimental; Down-Regulation; Glycation End Products, Advanced; Humans; Male; MAP Kinase Signaling System; Mice; Muscle Fatigue; Muscle Fibers, Skeletal; Muscle Proteins; Muscle Weakness; Muscle, Skeletal; Muscular Diseases; Receptor for Advanced Glycation End Products; Regeneration; Signal Transduction; SKP Cullin F-Box Protein Ligases; Thiazoles

2016