thiamine has been researched along with Pantothenate Kinase-Associated Neurodegeneration in 1 studies
thiamine(1+) : A primary alcohol that is 1,3-thiazol-3-ium substituted by (4-amino-2-methylpyrimidin-5-yl)methyl, methyl and 2-hydroxyethyl groups at positions 3, 4 and 5, respectively.
Pantothenate Kinase-Associated Neurodegeneration: A rare autosomal recessive degenerative disorder which usually presents in late childhood or adolescence. Clinical manifestations include progressive MUSCLE SPASTICITY; hyperreflexia; MUSCLE RIGIDITY; DYSTONIA; DYSARTHRIA; and intellectual deterioration which progresses to severe dementia over several years. (From Adams et al., Principles of Neurology, 6th ed, p972; Davis & Robertson, Textbook of Neuropathology, 2nd ed, pp972-929)
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 1 (100.00) | 2.80 |
Authors | Studies |
---|---|
Álvarez-Córdoba, M | 1 |
Reche-López, D | 1 |
Cilleros-Holgado, P | 1 |
Talaverón-Rey, M | 1 |
Villalón-García, I | 1 |
Povea-Cabello, S | 1 |
Suárez-Rivero, JM | 1 |
Suárez-Carrillo, A | 1 |
Munuera-Cabeza, M | 1 |
Piñero-Pérez, R | 1 |
Sánchez-Alcázar, JA | 1 |
1 other study available for thiamine and Pantothenate Kinase-Associated Neurodegeneration
Article | Year |
---|---|
Therapeutic approach with commercial supplements for pantothenate kinase-associated neurodegeneration with residual PANK2 expression levels.
Topics: Carbon-Sulfur Lyases; Humans; Iron; Pantothenate Kinase-Associated Neurodegeneration; Phosphotransfe | 2022 |