Compounds > thiamine
Page last updated: 2024-10-20

thiamine and Diabetes Mellitus

thiamine has been researched along with Diabetes Mellitus in 111 studies

thiamine(1+) : A primary alcohol that is 1,3-thiazol-3-ium substituted by (4-amino-2-methylpyrimidin-5-yl)methyl, methyl and 2-hydroxyethyl groups at positions 3, 4 and 5, respectively.

Diabetes Mellitus: A heterogeneous group of disorders characterized by HYPERGLYCEMIA and GLUCOSE INTOLERANCE.

Research Excerpts

ExcerptRelevanceReference
" Thiamine-responsive megaloblastic anaemia (TRMA), due to mutations in the thiamine transporter SLC19A2, is associated with the classical clinical triad of diabetes, deafness, and megaloblastic anaemia."8.88Recessive SLC19A2 mutations are a cause of neonatal diabetes mellitus in thiamine-responsive megaloblastic anaemia. ( Abdullah, M; Ellard, S; Flanagan, SE; Grulich-Henn, J; Habeb, AM; Hattersley, AT; Hussain, K; Matyka, K; Patch, AM; Pomahacova, R; Shaw-Smith, C, 2012)
" The diabetic states that associate with these genetic variants are MODY 2, thiamine responsive anaemia syndrome (TRAS) and mitochondrial diabetes."8.81Mitochondrial diabetes, diabetes and the thiamine-responsive megaloblastic anaemia syndrome and MODY-2. Diseases with common pathophysiology? ( Maassen, JA, 2002)
" Arrhythmia in thiamine responsive megaloblastic anemia syndrome."7.88Arrhythmia in thiamine responsive megaloblastic anemia syndrome. ( Akın, L; Argun, M; Baykan, A; Hatipoğlu, N; Kurtoğlu, S; Narin, N; Şahin, Y, 2018)
"Solute carrier family 19 member 2 (SLC19A2) gene deficiency is one of the causes of permanent neonatal diabetes mellitus (PNDM) and can be effectively managed by thiamine supplementation."7.88Recovered insulin production after thiamine administration in permanent neonatal diabetes mellitus with a novel solute carrier family 19 member 2 (SLC19A2) mutation. ( Cheng, R; Luo, F; Pei, Z; Sun, B; Sun, C; Yang, L; Zhang, M; Zhao, Z, 2018)
"Thiamine-responsive megaloblastic anemia (TRMA) syndrome is an autosomal recessive inherited disorder characterised by a triad of megaloblastic anemia, diabetes mellitus, and sensorineural deafness."7.88Thiamine Responsive Megaloblastic Anaemia, Diabetes Mellitus and Sensorineural Hearing Loss in a Child. ( Altaf, C; Fatima, S; Khadim, MT; Khurshid, A; Malik, HS; Sajjad, Z, 2018)
"Thiamine-responsive megaloblastic anaemia syndrome (TRMA) is the association of diabetes mellitus, anaemia and deafness, due to mutations in SLC19A2, encoding a thiamine transporter protein."7.73Thiamine-responsive megaloblastic anaemia syndrome: long-term follow-up and mutation analysis of seven families. ( Ariyawansa, I; Barrett, TG; Lo, IF; Minton, JA; Ricketts, CJ; Samuel, J; Wales, JK, 2006)
"Thiamine-responsive megaloblastic anemia (TRMA) syndrome is an autosomal recessive disorder characterized by diabetes mellitus (DM), progressive sensorineural deafness, and thiamine-responsive anemia."7.72Novel mutation in the SLC19A2 gene in an African-American female with thiamine-responsive megaloblastic anemia syndrome. ( Calikoglu, AS; Lagarde, WH; Moats-Staats, BM; Underwood, LE, 2004)
"Thiamine-responsive megaloblastic anaemia with diabetes and deafness (TRMA; MIM 249270) is an autosomal recessive disease thought to be due to a defect in thiamine (vitamin B1) transport."7.70The gene mutated in thiamine-responsive anaemia with diabetes and deafness (TRMA) encodes a functional thiamine transporter. ( Cohen, N; Fleming, JC; Neufeld, EJ; Schorderet, DF; Steinkamp, MP; Tartaglini, E, 1999)
"Thiamine-responsive megaloblastic anaemia syndrome (TRMA; MIM 249270) is an autosomal recessive disorder with features that include megaloblastic anaemia, mild thrombocytopenia and leucopenia, sensorineural deafness and diabetes mellitus."7.70Mutations in a new gene encoding a thiamine transporter cause thiamine-responsive megaloblastic anaemia syndrome. ( Banikazemi, M; Desnick, RJ; Diaz, GA; Gelb, BD; Oishi, K, 1999)
"Thiamine-responsive megaloblastic anaemia (TRMA), also known as Rogers syndrome, is an early onset, autosomal recessive disorder defined by the occurrence of megaloblastic anaemia, diabetes mellitus and sensorineural deafness, responding in varying degrees to thiamine treatment (MIM 249270)."7.70Mutations in SLC19A2 cause thiamine-responsive megaloblastic anaemia associated with diabetes mellitus and deafness. ( Baron, D; Barrett, T; Cohen, N; Gregory, S; Labay, V; Mandel, H; McDonald, L; Nosaka, K; Raz, T; Shalata, A; Szargel, R; Williams, H, 1999)
"We have investigated the cellular pathology of the syndrome called thiamine-responsive megaloblastic anemia (TRMA) with diabetes and deafness."7.70Defective high-affinity thiamine transporter leads to cell death in thiamine-responsive megaloblastic anemia syndrome fibroblasts. ( Baker, MA; Cohen, N; Fleming, JC; Neufeld, EJ; Sakamoto, M; Stagg, AR, 1999)
"Thiamine-responsive megaloblastic anemia (TRMA, also known as Rogers syndrome, OMIM 249270) is a rare autosomal recessive disorder characterized by a triad of megaloblastic anemia, diabetes mellitus, and sensorineural deafness."7.70Refined mapping of the gene for thiamine-responsive megaloblastic anemia syndrome and evidence for genetic homogeneity. ( Barrett, T; Cohen, N; Mandel, H; Neufeld, EJ; Nosaka, K; Raz, T; Szargel, R; Viana, MB, 1998)
"Three brothers with diabetes mellitus, thiamine-responsive megaloblastic anemia, and sensorineural deafness are reported."7.67Diabetes mellitus, thiamine-dependent megaloblastic anemia, and sensorineural deafness associated with deficient alpha-ketoglutarate dehydrogenase activity. ( Abboud, MR; Alexander, D; Najjar, SS, 1985)
"This case demonstrates Leber congenital amaurosis can present as the first clinical feature before systemic manifestations."5.72Leber congenital amaurosis as an initial manifestation in a Chinese patient with thiamine-responsive megaloblastic anemia syndrome. ( Sui, R; Sun, Z; Wu, S; Yao, F; Yuan, Z, 2022)
"Rogers syndrome is an autosomal recessive disorder resulting in megaloblastic anemia, diabetes mellitus, and sensorineural deafness."5.32Disruption of transport activity in a D93H mutant thiamine transporter 1, from a Rogers Syndrome family. ( Aronheim, A; Assaraf, YG; Baron, D; Drori, S, 2003)
"Wolfram syndrome, Kearns-Sayre syndrome, thiamine-responsive megaloblastic anemia, and maternally inherited diabetes and deafness are genetic disorders characterized by diabetes, impaired hearing, and vision."5.22Diabetes Out-of-the-Box: Diabetes Mellitus and Impairment in Hearing and Vision. ( Gruber, N; Pinhas-Hamiel, O, 2022)
"Thiamine-responsive megaloblastic anemia (TRMA), also known as Rogers syndrome, is a rare autosomal recessive disease characterized by three main components: megaloblastic anemia, diabetes mellitus and sensorineural deafness."5.01[Thiamine-responsive megaloblastic anemia or Rogers syndrome: A literature review]. ( Castioni, J; Lu, H; Tran, C; Vaucher, J; Vollenweider, P, 2019)
" Thiamine-responsive megaloblastic anaemia (TRMA), due to mutations in the thiamine transporter SLC19A2, is associated with the classical clinical triad of diabetes, deafness, and megaloblastic anaemia."4.88Recessive SLC19A2 mutations are a cause of neonatal diabetes mellitus in thiamine-responsive megaloblastic anaemia. ( Abdullah, M; Ellard, S; Flanagan, SE; Grulich-Henn, J; Habeb, AM; Hattersley, AT; Hussain, K; Matyka, K; Patch, AM; Pomahacova, R; Shaw-Smith, C, 2012)
" The diabetic states that associate with these genetic variants are MODY 2, thiamine responsive anaemia syndrome (TRAS) and mitochondrial diabetes."4.81Mitochondrial diabetes, diabetes and the thiamine-responsive megaloblastic anaemia syndrome and MODY-2. Diseases with common pathophysiology? ( Maassen, JA, 2002)
"Individuals with thiamine-responsive megaloblastic anemia (TRMA) mainly manifest macrocytic anemia, sensorineural deafness, ocular complications, and nonautoimmune diabetes."4.31An Italian case series' description of thiamine responsive megaloblastic anemia syndrome: importance of early diagnosis and treatment. ( Arrigoni, F; Bonfanti, R; Di Candia, F; Di Iorio, V; Fedi, L; Franzese, A; Iafusco, F; Iovino, C; Malesci, R; Mozzillo, E; Rigamonti, A; Rosanio, FM; Simonelli, F; Tinto, N, 2023)
" Thiamine supplementation corrected anemia and diabetes mellitus but did not improve the hearing defect."4.02Identification of novel compound heterozygous variants in SLC19A2 and the genotype-phenotype associations in thiamine-responsive megaloblastic anemia. ( Li, G; Qiao, Y; Shang, X; Sun, Y; Wang, Z; Zhang, S; Zhuang, J, 2021)
"Thiamine-responsive megaloblastic anemia (TRMA) syndrome is a rare disease comprising a classic triad of megaloblastic anemia, diabetes mellitus, and early-onset sensorineural deafness."3.91An Adult Case of Thiamine-Sensitive Megaloblastic Anemia Syndrome Accidentally Diagnosed Myelodysplastic Syndrome. ( Kutlucan, A, 2019)
"Thiamine-responsive megaloblastic anemia (TRMA) is a rare autosomal recessive inherited disease characterized by the clinical triad of megaloblastic anemia, sensorineural deafness, and diabetes mellitus."3.91TRMA syndrome with a severe phenotype, cerebral infarction, and novel compound heterozygous SLC19A2 mutation: a case report. ( Cheng, Q; Ding, Y; Li, Q; Li, X; Wang, J; Wang, X; Yao, R, 2019)
"Thiamine-responsive megaloblastic anemia (TRMA) syndrome is an autosomal recessive inherited disorder characterised by a triad of megaloblastic anemia, diabetes mellitus, and sensorineural deafness."3.88Thiamine Responsive Megaloblastic Anaemia, Diabetes Mellitus and Sensorineural Hearing Loss in a Child. ( Altaf, C; Fatima, S; Khadim, MT; Khurshid, A; Malik, HS; Sajjad, Z, 2018)
" Arrhythmia in thiamine responsive megaloblastic anemia syndrome."3.88Arrhythmia in thiamine responsive megaloblastic anemia syndrome. ( Akın, L; Argun, M; Baykan, A; Hatipoğlu, N; Kurtoğlu, S; Narin, N; Şahin, Y, 2018)
"A female child presented with Leber's congenital amaurosis at 10 months of age, later diagnosed with hearing impairment at 1 year, diabetes mellitus and megaloblastic anemia at 3 and a half years of age and hence as a case of thiamine responsive megaloblastic anemia."3.80Leber's congenital amaurosis as the retinal degenerative phenotype in thiamine responsive megaloblastic anemia: a case report. ( Arokiasamy, T; Meenakshi, S; Murali, K; Soumittra, N; Srikrupa, NN, 2014)
"Thiamine responsive megaloblastic anemia (TRMA) is characterized by a triad of megaloblastic anemia, non-type 1 diabetes mellitus and sensorineural deafness."3.80Thiamine responsive megaloblastic anemia: the puzzling phenotype. ( Al Zadjali, S; Alrawas, A; Bashir, W; Beshlawi, I; Elshinawy, M; Wali, Y, 2014)
"Thiamine-responsive megaloblastic anemia (TRMA) is a clinical triad characterized by megaloblastic anemia, non-autoimmune diabetes mellitus, and sensory-neural hearing loss."3.79Novel mutation in the SLC19A2 gene in an Iranian family with thiamine-responsive megaloblastic anemia: a series of three cases. ( Abbaszadegan, MR; Baradaran-Heravi, A; Ghaemi, N; Ghahraman, M; Vakili, R, 2013)
"Thiamine-responsive megaloblastic anemia (TRMA) is an autosomal recessive syndrome characterized by early-onset anemia, diabetes, and hearing loss caused by mutations in the SLC19A2 gene."3.79Identification of a SLC19A2 nonsense mutation in Persian families with thiamine-responsive megaloblastic anemia. ( Ellard, S; Haghighi, A; Saleh-Gohari, N; Setoodeh, A, 2013)
"The thiamine-responsive megaloblastic anemia syndrome (TRMA) is an autosomal recessive disorder characterized by diabetes mellitus, megaloblastic anemia and sensorineural hearing loss due to mutations in SLC 19A2 that encodes a thiamine transporter protein."3.78Thiamine-responsive megaloblastic anemia syndrome: a novel mutation. ( Aycan, Z; Bas, VN; Onder, A; Peltek Kendirci, HN; Yilmaz Agladioglu, S, 2012)
"Thiamine-responsive megaloblastic anaemia (TRMA; OMIM 249270) syndrome is an autosomal recessive disorder characterized by diabetes mellitus, megaloblastic anaemia, and sensorineural deafness."3.77Does early treatment prevent deafness in thiamine-responsive megaloblastic anaemia syndrome? ( Akın, L; Akın, MA; Karakükçü, M; Kendirci, M; Kurtoğlu, S, 2011)
"Thiamine-responsive megaloblastic anemia (TRMA) syndrome usually associated with diabetes mellitus, anemia and deafness, due to mutations in SLC19A2, encoding a thiamine transporter protein."3.76Thiamine-responsive megaloblastic anemia syndrome. ( Bay, A; Dai, A; Gumruk, F; Hizli, S; Keskin, M; Uygun, H, 2010)
"Thiamine-responsive megaloblastic anemia is a rare autosomal recessive disorder whose main symptoms are anemia, diabetes mellitus, and sensorineural deafness."3.75Thiamine-responsive megaloblastic anemia syndrome: long term follow-up. ( Azzalli, M; Borgna-Pignatti, C; Pedretti, S, 2009)
"This report describes a female child with thiamine responsive megaloblastic anemia syndrome (Rogers syndrome), presenting with anemia and diabetes mellitus responding to thiamine."3.75Thiamine responsive megaloblastic anemia. ( Mathews, L; Narayanadas, K; Sunil, G, 2009)
"Thiamine-responsive megaloblastic anemia (TRMA) is a rare autosomal recessive disorder characterized by megaloblastic anemia, diabetes mellitus and progressive sensorineural deafness."3.75Diabetic acido-ketosis revealing thiamine-responsive megaloblastic anemia. ( Ben Mansour, F; Bouyahia, O; Khaldi, F; Matoussi, N; Ouderni, M, 2009)
"Thiamine-responsive megaloblastic anaemia (TRMA) is a rare autosomal recessive condition, characterized by megaloblastic anaemia, non-autoimmune diabetes mellitus, and sensorineural hearing loss."3.74Thiamine-responsive megaloblastic anaemia: a cause of syndromic diabetes in childhood. ( Hahnemann, JM; Olsen, BS; Schwartz, M; Østergaard, E, 2007)
"Thiamine-responsive megaloblastic anaemia syndrome (TRMA) is the association of diabetes mellitus, anaemia and deafness, due to mutations in SLC19A2, encoding a thiamine transporter protein."3.73Thiamine-responsive megaloblastic anaemia syndrome: long-term follow-up and mutation analysis of seven families. ( Ariyawansa, I; Barrett, TG; Lo, IF; Minton, JA; Ricketts, CJ; Samuel, J; Wales, JK, 2006)
"Thiamine-responsive megaloblastic anemia (TRMA) syndrome is an autosomal recessive disorder characterized by diabetes mellitus (DM), progressive sensorineural deafness, and thiamine-responsive anemia."3.72Novel mutation in the SLC19A2 gene in an African-American female with thiamine-responsive megaloblastic anemia syndrome. ( Calikoglu, AS; Lagarde, WH; Moats-Staats, BM; Underwood, LE, 2004)
"Thiamine-responsive megaloblastic anemia (TRMA, also known as Rogers syndrome, OMIM 249270) is a rare autosomal recessive disorder characterized by a triad of megaloblastic anemia, diabetes mellitus, and sensorineural deafness."3.70Refined mapping of the gene for thiamine-responsive megaloblastic anemia syndrome and evidence for genetic homogeneity. ( Barrett, T; Cohen, N; Mandel, H; Neufeld, EJ; Nosaka, K; Raz, T; Szargel, R; Viana, MB, 1998)
"We have investigated the cellular pathology of the syndrome called thiamine-responsive megaloblastic anemia (TRMA) with diabetes and deafness."3.70Defective high-affinity thiamine transporter leads to cell death in thiamine-responsive megaloblastic anemia syndrome fibroblasts. ( Baker, MA; Cohen, N; Fleming, JC; Neufeld, EJ; Sakamoto, M; Stagg, AR, 1999)
"Thiamine-responsive megaloblastic anaemia syndrome (TRMA; MIM 249270) is an autosomal recessive disorder with features that include megaloblastic anaemia, mild thrombocytopenia and leucopenia, sensorineural deafness and diabetes mellitus."3.70Mutations in a new gene encoding a thiamine transporter cause thiamine-responsive megaloblastic anaemia syndrome. ( Banikazemi, M; Desnick, RJ; Diaz, GA; Gelb, BD; Oishi, K, 1999)
"Thiamine-responsive megaloblastic anaemia (TRMA), also known as Rogers syndrome, is an early onset, autosomal recessive disorder defined by the occurrence of megaloblastic anaemia, diabetes mellitus and sensorineural deafness, responding in varying degrees to thiamine treatment (MIM 249270)."3.70Mutations in SLC19A2 cause thiamine-responsive megaloblastic anaemia associated with diabetes mellitus and deafness. ( Baron, D; Barrett, T; Cohen, N; Gregory, S; Labay, V; Mandel, H; McDonald, L; Nosaka, K; Raz, T; Shalata, A; Szargel, R; Williams, H, 1999)
"Thiamine-responsive megaloblastic anaemia with diabetes and deafness (TRMA; MIM 249270) is an autosomal recessive disease thought to be due to a defect in thiamine (vitamin B1) transport."3.70The gene mutated in thiamine-responsive anaemia with diabetes and deafness (TRMA) encodes a functional thiamine transporter. ( Cohen, N; Fleming, JC; Neufeld, EJ; Schorderet, DF; Steinkamp, MP; Tartaglini, E, 1999)
"Thiamine responsive megaloblastic anemia (TRMA) is an autosomal recessive disorder with a triad of symptoms: megaloblastic anemia, deafness, and non-type 1 diabetes mellitus."3.70The spectrum of mutations, including four novel ones, in the thiamine-responsive megaloblastic anemia gene SLC19A2 of eight families. ( Anbinder, Y; Baron, D; Barrett, T; Baruchel, A; Cayuela, JM; Cohen, N; Labay, V; Mandel, H; Rabl, W; Raz, T; Szargel, R; Viana, MB, 2000)
"Three brothers with diabetes mellitus, thiamine-responsive megaloblastic anemia, and sensorineural deafness are reported."3.67Diabetes mellitus, thiamine-dependent megaloblastic anemia, and sensorineural deafness associated with deficient alpha-ketoglutarate dehydrogenase activity. ( Abboud, MR; Alexander, D; Najjar, SS, 1985)
"Erythrocyte transketolase activity (ETKA) and the effect of adding thiamine pyrophosphate have been measured in a group of 27 healthy individuals and in 37 patients diagnosed as having diabetes mellitus, anemia, polyneuritis, or malnourishment secondary to vascular disease of the brain."3.65The transketolase assay of thiamine in some diseases. ( Kjosen, B; Seim, SH, 1977)
"Thiamine (vitamin B1) is an essential cofactor in glucose metabolism, but it remains unclear whether thiamine status is lower in individuals with diabetes compared to individuals with normal glucose metabolism."3.01Association between diabetes and thiamine status - A systematic review and meta-analysis. ( Obeid, R; Reiners, K; Strom, A; Ziegler, D, 2023)
"Thiamine is a crucial cofactor involved in the maintenance of carbohydrate metabolism and participates in multiple cellular metabolic processes within the cytosol, mitochondria, and peroxisomes."2.61Genetic defects of thiamine transport and metabolism: A review of clinical phenotypes, genetics, and functional studies. ( Baide-Mairena, H; Marcé-Grau, A; Martí-Sánchez, L; Ortigoza-Escobar, JD; Pérez-Dueñas, B, 2019)
"This case demonstrates Leber congenital amaurosis can present as the first clinical feature before systemic manifestations."1.72Leber congenital amaurosis as an initial manifestation in a Chinese patient with thiamine-responsive megaloblastic anemia syndrome. ( Sui, R; Sun, Z; Wu, S; Yao, F; Yuan, Z, 2022)
"Thiamine is a water-soluble vitamin, rapidly expelled from the body, with no issues of over-dosage or accumulation; unfortunately, it is non-patentable, and neither industry nor independent donors are interested in investing in large-scale randomized controlled clinical trials to investigate its potential in diabetes and its complications."1.62Thiamine and diabetes: back to the future? ( Beltramo, E; Mazzeo, A; Porta, M, 2021)
" Recent evidences have shown that the administration of thiamine or lipid-soluble derivatives, such as benfotiamine (developed to improve the bioavailability of thiamine), has positive effects in the diabetic patient (after thiamine is transformed into TPP)."1.37Could thiamine pyrophosphate be a regulator of the nitric oxide synthesis in the endothelial cell of diabetic patients? ( Alcázar-Leyva, S; Alvarado-Vásquez, N, 2011)
"Treatment with fenofibrate normalizes the altered lipid profile in diabetic rats, whereas benfotiamine treatment has no effect on lipid alteration in diabetic rats."1.35Ameliorative effect of combination of benfotiamine and fenofibrate in diabetes-induced vascular endothelial dysfunction and nephropathy in the rat. ( Balakumar, P; Chakkarwar, VA; Singh, M, 2009)
"Rogers syndrome is an autosomal recessive disorder resulting in megaloblastic anemia, diabetes mellitus, and sensorineural deafness."1.32Disruption of transport activity in a D93H mutant thiamine transporter 1, from a Rogers Syndrome family. ( Aronheim, A; Assaraf, YG; Baron, D; Drori, S, 2003)
"When thiamine was added to cells kept under high glucose conditions, the number of surviving cells was significantly increased and the reduced cell proliferation appeared to be corrected."1.29Thiamine corrects delayed replication and decreases production of lactate and advanced glycation end-products in bovine retinal and human umbilical vein endothelial cells cultured under high glucose conditions. ( Beltramo, E; Bena, E; La Selva, M; Molinatti, GM; Molinatti, PA; Pagnozzi, F; Porta, M, 1996)

Research

Studies (111)

TimeframeStudies, this research(%)All Research%
pre-199037 (33.33)18.7374
1990's8 (7.21)18.2507
2000's20 (18.02)29.6817
2010's29 (26.13)24.3611
2020's17 (15.32)2.80

Authors

AuthorsStudies
Wu, S1
Yuan, Z1
Sun, Z1
Yao, F1
Sui, R1
Wang, Z3
Yu, L1
Chen, Y1
Liu, J3
Allowitz, KV1
Yoo, JJ1
Taylor, JR1
Baloch, OA1
Harames, K1
Ramana, KV1
Mohsen-Pour, N1
Naderi, N1
Ghasemi, S1
Hesami, M1
Maleki, M1
Kalayinia, S1
Gruber, N1
Pinhas-Hamiel, O1
Liu, C2
Meng, Q1
Zu, C1
Li, R1
Yang, S2
He, P1
Li, H1
Zhang, YY1
Zhou, C1
Liu, M1
Ye, Z1
Wu, Q1
Zhang, YJ1
Gan, X1
Qin, X1
Khaliq, S1
Ziegler, D2
Reiners, K1
Strom, A1
Obeid, R2
Dhanani, LY1
Totton, RR1
Jara-Quijada, E1
Pérez-Won, M1
Tabilo-Munizaga, G1
Lemus-Mondaca, R1
González-Cavieres, L1
Palma-Acevedo, A1
Herrera-Lavados, C1
Milovanovic, S1
Grzegorczyk, A1
Świątek, Ł1
Grzęda, A1
Dębczak, A1
Tyskiewicz, K1
Konkol, M1
Li, Y2
Chang, P1
Sankaran, S1
Jang, H1
Nie, Y1
Zeng, A1
Hussain, S1
Wu, JY1
Chen, X2
Shi, L2
Senapati, A1
Chetri, BK1
Mitra, S1
Shelke, RG1
Rangan, L1
Chauhan, AS1
Tiwari, M1
Indoliya, Y1
Mishra, SK1
Lavania, UC1
Chauhan, PS1
Chakrabarty, D1
Tripathi, RD1
Akaputra, R1
Hatta, M1
Massi, MN1
Djaharuddin, I1
Bukhari, A1
Aminuddin, A1
Santoso, A1
Natzir, R1
Bahar, B1
Fachri, M1
Farsida, F1
Fathimah, A1
Ubaidah, FM1
Sridiana, E1
Dwiyanti, R1
Syukri, A1
Junita, AR1
Febrianti, A1
Primaguna, MR1
Azhar, A1
Rajaure, YS1
Thapa, B1
Budhathoki, L1
Rana, SR1
Khadka, M1
Batchu, UR1
Surapaneni, JR1
Cheemalamarri, C1
Mandava, K1
Puvvada, N1
Shetty, PR1
Mv, S1
Ranjbarian, P1
Goudarzi, F1
Akya, A1
Heidarinia, H1
Farasat, A1
Rostamian, M1
Suri, K1
Rajput, N1
Sharma, P1
Omble, AD1
Kulkarni, K1
Gahlay, GK1
Fernandez Garcia, E1
Paudel, U1
Noji, MC1
Bowman, CE1
Rustgi, AK1
Pitarresi, JR1
Wellen, KE1
Arany, Z1
Weissenrieder, JS1
Foskett, JK1
Lee, MS1
Han, HJ1
Choi, TI1
Lee, KH1
Baasankhuu, A1
Kim, HT1
Kim, CH1
Redd, PS1
Payero, L1
Gilbert, DM1
Page, CA1
King, R1
McAssey, EV1
Bodie, D1
Diaz, S1
Hancock, CN1
Lee, HS1
Jung, S1
Lee, SW1
Kim, YT1
Lee, J1
Ren, T1
Yu, Z1
Yu, H1
Deng, K1
Li, X2
Wang, H1
Wang, L2
Xu, Y1
Lascano, J1
Riley, L1
Khodayari, N1
Brantly, M1
Gupta, R1
Pradhan, J1
Haldar, A1
Murapaka, C1
Chandra Mondal, P1
Gao, R1
Dai, TY1
Meng, Z1
Sun, XF1
Liu, DX1
Shi, MM1
Li, HR1
Kang, X1
Bi, B1
Zhang, YT1
Xu, TW1
Yan, JM1
Jiang, Q1
Helmchen, G1
Guo, H1
Xiang, W1
Fang, Y1
Li, J4
Lin, Y1
An, X1
Jiang, D1
Cao, Q1
Xu, F1
Shiigi, H1
Wang, W1
Chen, Z1
Akosman, I1
Kumar, N1
Mortenson, R1
Lans, A1
De La Garza Ramos, R1
Eleswarapu, A1
Yassari, R1
Fourman, MS1
Jana, S1
Evans, EGB1
Jang, HS1
Zhang, S3
Zhang, H1
Rajca, A1
Gordon, SE1
Zagotta, WN1
Stoll, S1
Mehl, RA1
Miller, S1
Lee, DA1
Muhimpundu, S1
Maxwell, CA1
Liu, Z1
Shen, F1
Tong, Q1
Tang, M1
Peng, M1
Jiao, Z1
Jiang, Y1
Ao, L1
Fu, W1
Lv, X1
Jiang, G1
Hou, L1
Tu, WC1
McManamen, AM1
Su, X1
Jeacopello, I1
Takezawa, MG1
Hieber, DL1
Hassan, GW1
Lee, UN1
Anana, EV1
Locknane, MP1
Stephenson, MW1
Shinkawa, VAM1
Wald, ER1
DeMuri, GP1
Adams, KN1
Berthier, E1
Thongpang, S1
Theberge, AB1
Jiang, L1
Li, Z1
Dong, Q1
Rong, X1
Dong, G1
Huang, J1
Liang, Y1
Sun, S1
Zhang, R1
Miao, Z1
Senju, C1
Nakazawa, Y1
Oso, T1
Shimada, M1
Kato, K1
Matsuse, M1
Tsujimoto, M1
Masaki, T1
Miyazaki, Y1
Fukushima, S1
Tateishi, S1
Utani, A1
Murota, H1
Tanaka, K1
Mitsutake, N1
Moriwaki, S1
Nishigori, C1
Ogi, T1
Zhang, X1
Wang, B1
Luo, Z1
Qian, D1
Waterhouse, GIN1
Barbosa, M1
Marques-Sá, J1
Carvalho, C1
Fernandes, V1
Grilli, D1
Smetana, V1
Ahmed, SJ1
Shtender, V1
Pani, M1
Manfrinetti, P1
Mudring, AV1
Kuang, Y1
Yang, D1
Gai, S1
He, F1
An, B1
Yang, P1
Notini, L1
Schulz, K1
Kubeneck, LJ1
Grigg, ARC1
Rothwell, KA1
Fantappiè, G1
ThomasArrigo, LK1
Kretzschmar, R1
Siswanto, FM1
Okukawa, K1
Tamura, A1
Oguro, A1
Imaoka, S1
Kim, CG1
Jung, M1
Kim, HS1
Lee, CK1
Jeung, HC1
Koo, DH1
Bae, WK1
Zang, DY1
Kim, BJ1
Kim, H1
Yun, UJ1
Che, J1
Park, S1
Kim, TS1
Kwon, WS1
Park, J1
Cho, SW1
Nam, CM1
Chung, HC1
Rha, SY1
Colombo, N1
Van Gorp, T1
Matulonis, UA1
Oaknin, A1
Grisham, RN1
Fleming, GF1
Olawaiye, AB1
Nguyen, DD1
Greenstein, AE1
Custodio, JM1
Pashova, HI1
Tudor, IC1
Lorusso, D1
Zhou, F1
Jiang, Z1
Liang, H1
Ru, S1
Bettiol, AA1
Gao, W1
Lipsyc-Sharf, M1
Jain, E1
Collins, LC1
Rosenberg, SM1
Ruddy, KJ1
Tamimi, RM1
Schapira, L1
Come, SE1
Peppercorn, JM1
Borges, VF1
Warner, E1
Snow, C1
Krop, IE1
Kim, D1
Weiss, J1
Zanudo, JGT1
Partridge, AH1
Wagle, N1
Waks, AG1
Moskowitz, A1
Berg, KM1
Grossestreuer, AV1
Balaji, L1
Liu, X1
Cocchi, MN1
Chase, M1
Gong, MN1
Gong, J1
Parikh, SM1
Ngo, L1
Berlin, N1
Donnino, MW1
Zhou, Y1
Chen, Q1
Zhong, S1
Liu, H1
Koh, K1
Chen, H1
He, J1
Chen, J1
Liu, S1
Lin, L1
Zhang, Y1
Xiao, S1
Cao, S1
Yan, B2
Deng, J1
Gu, J1
Tao, Y1
Huang, C1
Lai, C1
Yong, Q1
Shen, Y1
Gong, Z1
Cao, J1
Mao, W1
Yao, Y1
Zhao, J1
Li, Q2
Liu, K1
Liu, B1
Feng, S1
Chandran, V1
Kunjan, C1
Veerapandian, V1
Kannan, R1
Zhang, T1
von Gunten, U1
Caruso, FR1
Goulart, CDL1
Jr, JCB1
de Oliveira, CR1
Mendes, RG1
Arena, R1
Borghi-Silva, A1
Carrasco-Nuñes, N1
Romano, M1
Cabeza, M1
Jiang, B1
Chen, D1
Zhao, C1
Ma, Y1
Yang, W1
Shen, X1
Satheeshkumar, K1
Saravanakumar, P1
Kalavathi, A1
Vennila, KN1
Elango, KP1
Mues Genannt Koers, L1
Prevost, D1
Paulssen, E1
Hoehr, C1
Ulhassan, Z1
He, D1
Khan, AR1
Salam, A1
Azhar, W1
Muhammad, S1
Ali, S1
Hamid, Y1
Khan, I1
Sheteiwy, MS1
Zhou, W1
Wang, X2
Xie, Q1
Wang, Y1
Lü, H3
Fu, M1
Wang, D1
Di Candia, F1
Di Iorio, V1
Tinto, N1
Bonfanti, R1
Iovino, C1
Rosanio, FM1
Fedi, L1
Iafusco, F1
Arrigoni, F1
Malesci, R1
Simonelli, F1
Rigamonti, A1
Franzese, A2
Mozzillo, E2
Alcázar-Leyva, S2
Zapata, E1
Bernal-Alcántara, D1
Gorocica, P1
Alvarado-Vásquez, N2
Stirban, OA2
Zeller-Stefan, H1
Schumacher, J1
Gaus, W1
Schuerholz, T1
Pop-Busui, R1
Nouira, N1
Mansouri, R1
Tlili, R1
Bhouri, I1
Sfaxi, S1
Chtourou, D1
Cheikh, MB1
Warncke, K1
Prinz, N1
Iotova, V1
Dunstheimer, D1
Datz, N1
Karges, B1
Jali, MV1
Linsenmeyer, D1
Olsen, BS2
Seiwald, M1
Prahalad, P1
de Sousa, G1
Pacaud, D1
Qiao, Y1
Zhuang, J1
Sun, Y1
Shang, X1
Li, G1
Frank, J1
Kisters, K1
Lorkowski, S1
Wallert, M1
Egert, S1
Podszun, MC1
Eckert, GP1
Pettersen, JA1
Venturelli, S1
Classen, HG1
Golombek, J1
Beltramo, E2
Mazzeo, A1
Porta, M2
Sun, C1
Pei, Z1
Zhang, M1
Sun, B1
Yang, L1
Zhao, Z1
Cheng, R1
Luo, F1
Xian, X1
Lin, F1
Habeb, AM2
Flanagan, SE3
Zulali, MA1
Abdullah, MA1
Pomahačová, R2
Boyadzhiev, V1
Colindres, LE1
Godoy, GV1
Vasanthi, T1
Al Saif, R1
Setoodeh, A2
Haghighi, A4
Shaalan, Y1
Hattersley, AT2
Ellard, S4
De Franco, E1
Vaucher, J1
Tran, C1
Vollenweider, P1
Castioni, J1
Khurshid, A1
Fatima, S1
Altaf, C1
Malik, HS1
Sajjad, Z1
Khadim, MT1
Argun, M1
Baykan, A1
Hatipoğlu, N1
Akın, L2
Şahin, Y1
Narin, N1
Kurtoğlu, S2
Jungtrakoon, P1
Shirakawa, J1
Buranasupkajorn, P1
Gupta, MK1
De Jesus, DF1
Pezzolesi, MG1
Panya, A1
Hastings, T1
Chanprasert, C1
Mendonca, C1
Kulkarni, RN1
Doria, A1
Marcé-Grau, A1
Martí-Sánchez, L1
Baide-Mairena, H1
Ortigoza-Escobar, JD1
Pérez-Dueñas, B1
Kutlucan, A1
Dalwadi, P1
Joshi, AS1
Thakur, DS1
Bhagwat, NM1
Cheng, Q1
Ding, Y1
Yao, R1
Wang, J1
Saleh-Gohari, N1
Srikrupa, NN1
Meenakshi, S1
Arokiasamy, T1
Murali, K1
Soumittra, N1
Kousar, S1
Sheikh, MA1
Asghar, M1
Ghaemi, N1
Ghahraman, M1
Abbaszadegan, MR1
Baradaran-Heravi, A1
Vakili, R1
Beshlawi, I1
Al Zadjali, S1
Bashir, W1
Elshinawy, M1
Alrawas, A1
Wali, Y1
Liu, G1
Yang, F1
Han, B1
Nie, G1
Wood, MC1
Tsiouris, JA1
Velinov, M1
Mikstiene, V1
Songailiene, J1
Byckova, J1
Rutkauskiene, G1
Jasinskiene, E1
Verkauskiene, R1
Lesinskas, E1
Utkus, A1
Potter, K1
Wu, J1
Lauzon, J1
Ho, J1
Yeşilkaya, E1
Bideci, A1
Temizkan, M1
Kaya, Z1
Camurdan, O1
Koç, A1
Bozkaya, D1
Koçak, U1
Cinaz, P1
Balakumar, P2
Chakkarwar, VA1
Singh, M1
BISCEGLIE, V1
Mathews, L2
Narayanadas, K1
Sunil, G1
Borgna-Pignatti, C1
Azzalli, M1
Pedretti, S1
Bergmann, AK1
Sahai, I1
Falcone, JF1
Fleming, J1
Bagg, A1
Borgna-Pignati, C1
Casey, R1
Fabris, L1
Hexner, E1
Ribeiro, ML1
Wierenga, KJ1
Neufeld, EJ4
Bouyahia, O1
Ouderni, M1
Ben Mansour, F1
Matoussi, N1
Khaldi, F1
Rohilla, A1
Krishan, P1
Solairaj, P1
Thangathirupathi, A1
JANES, RG1
BRADY, JM1
Bay, A1
Keskin, M1
Hizli, S1
Uygun, H1
Dai, A1
Gumruk, F1
Kendirci, M1
Akın, MA1
Karakükçü, M1
Shaw-Smith, C1
Patch, AM1
Grulich-Henn, J1
Hussain, K1
Matyka, K1
Abdullah, M1
Yilmaz Agladioglu, S1
Aycan, Z1
Bas, VN1
Peltek Kendirci, HN1
Onder, A1
Larkin, JR1
Zhang, F1
Godfrey, L1
Molostvov, G1
Zehnder, D1
Rabbani, N1
Thornalley, PJ2
Melis, D1
Falco, M1
Fattorusso, V1
Taurisano, R1
Teodoro, JS1
Gomes, AP1
Varela, AT1
Duarte, FV1
Rolo, AP1
Palmeira, CM1
Oishi, K2
Hofmann, S1
Diaz, GA2
Brown, T1
Manwani, D1
Ng, L1
Young, R1
Vlassara, H1
Ioannou, YA1
Forrest, D1
Gelb, BD2
Maassen, JA1
SILIPRANDI, N1
NAVAZIO, F1
LOVETTI, M1
HAUGEN, HN2
ELLENBERG, M1
BRODERZON, EA1
LUKASHIK, NK1
KOLTUNOVA, VI1
BELLODI, G1
FERSINI, C1
RIPA, R1
SBRIGHI, V1
KUNZ, O1
OKSALA, A1
HUEBER, EF1
REDFERN, WE1
BEBIN, J1
EISENBREY, AB1
Baron, D3
Assaraf, YG1
Drori, S1
Aronheim, A1
JULESZ, M1
ZSOLDOS, I1
SZATMARI, E1
BUTTURINI, U1
LICHTENBELT, HV1
FLORIJN, E1
Lagarde, WH1
Underwood, LE1
Moats-Staats, BM1
Calikoglu, AS1
Ricketts, CJ1
Minton, JA1
Samuel, J1
Ariyawansa, I1
Wales, JK1
Lo, IF1
Barrett, TG1
Małecka, SA1
Poprawski, K1
Bilski, B1
Subramanian, VS1
Marchant, JS1
Said, HM1
Atta, HM1
El-Rehani, MA1
Raheim, SA1
Galal, AM1
Hahnemann, JM1
Schwartz, M1
Østergaard, E1
BRADA, Z1
ABELIN, I1
BARTLETT, PD1
GAEBLER, OH1
BOSE, JP1
Haworth, C1
Evans, DI1
Mitra, J1
Wickramasinghe, SN1
Paoletti, V1
Strazzera, P1
Katsarou, V1
Mammarella, A1
Barlattani, M1
Rieder, HP1
Berger, W1
Fridrich, R1
Lemoine, A1
Le Devehat, C1
Roux, E1
Cirette, B1
Vimeux, M1
Hobara, R1
Ozawa, K1
Okazaki, M1
Yasuhara, H1
La Selva, M1
Pagnozzi, F1
Bena, E1
Molinatti, PA1
Molinatti, GM1
Shehadeh, N1
Gershoni-Baruch, R1
Mandel, H4
Nutenko, I1
Etzioni, A1
Raz, T3
Barrett, T3
Szargel, R3
Nosaka, K2
Viana, MB3
Cohen, N5
Stagg, AR1
Fleming, JC2
Baker, MA1
Sakamoto, M1
Banikazemi, M1
Desnick, RJ1
Labay, V2
Williams, H1
McDonald, L1
Shalata, A1
Gregory, S1
Tartaglini, E1
Steinkamp, MP1
Schorderet, DF1
Tamai, H1
Anbinder, Y1
Rabl, W1
Baruchel, A1
Cayuela, JM1
Jahan, I1
Ng, R1
Li, SQ1
Zhang, QH1
Bappal, B1
Nair, R1
Shaikh, H1
AI Khusaiby, SM1
de Silva, V1
Bellazzi, R1
Guglielmann, R1
Ironi, L1
Patrini, C1
Carvalho, RI1
Kjosen, B1
Seim, SH1
Kolomoĭskaia, MB1
Latypova, GA1
Petrosova, GI1
Saito, N2
Kimura, M2
Kuchiba, A2
Itokawa, Y2
Abboud, MR1
Alexander, D1
Najjar, SS1
Karabun, PM1
Klein, W1
Guy-Grand, B1
Dorf, G1
Duchier, J1
Aimez, P1
Kaĭlakov, AM1
Veselý, KT1
Standl, E2
Haslbeck, H1
Geser, CA2
Mehnert, H2
Haslbeck, M1
Förster, H1

Reviews

13 reviews available for thiamine and Diabetes Mellitus

ArticleYear
Therapeutic potential of vitamin B
    Future medicinal chemistry, 2022, Volume: 14, Issue:11

    Topics: COVID-19 Drug Treatment; Diabetes Mellitus; Humans; Thiamine; Vitamins

2022
Diabetes Out-of-the-Box: Diabetes Mellitus and Impairment in Hearing and Vision.
    Current diabetes reports, 2022, Volume: 22, Issue:9

    Topics: Anemia, Megaloblastic; Deafness; Diabetes Mellitus; Diabetes Mellitus, Type 2; Hearing; Hearing Loss

2022
Association between diabetes and thiamine status - A systematic review and meta-analysis.
    Metabolism: clinical and experimental, 2023, Volume: 144

    Topics: Albuminuria; Diabetes Mellitus; Glucose; Humans; Thiamine; Thiamine Pyrophosphate

2023
The role of biofactors in the prevention and treatment of age-related diseases.
    BioFactors (Oxford, England), 2021, Volume: 47, Issue:4

    Topics: Aged; Cardiovascular Diseases; Dementia; Diabetes Mellitus; Dietary Fiber; Dietary Supplements; Epig

2021
[Defect of thiamine transport and activation and related disease].
    Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics, 2018, Feb-10, Volume: 35, Issue:1

    Topics: Anemia, Megaloblastic; Biological Transport; Diabetes Mellitus; Hearing Loss, Sensorineural; Humans;

2018
[Thiamine-responsive megaloblastic anemia or Rogers syndrome: A literature review].
    La Revue de medecine interne, 2019, Volume: 40, Issue:1

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Diagnosis, Differential; Dietary Supplements; Hearing Loss

2019
Genetic defects of thiamine transport and metabolism: A review of clinical phenotypes, genetics, and functional studies.
    Journal of inherited metabolic disease, 2019, Volume: 42, Issue:4

    Topics: Anemia, Megaloblastic; Biological Transport; Biomarkers; Diabetes Mellitus; Hearing Loss, Sensorineu

2019
The multifaceted therapeutic potential of benfotiamine.
    Pharmacological research, 2010, Volume: 61, Issue:6

    Topics: Animals; Diabetes Mellitus; Glycation End Products, Advanced; Humans; Hyperglycemia; Hypoglycemic Ag

2010
Recessive SLC19A2 mutations are a cause of neonatal diabetes mellitus in thiamine-responsive megaloblastic anaemia.
    Pediatric diabetes, 2012, Volume: 13, Issue:4

    Topics: Anemia, Megaloblastic; Consanguinity; Deafness; Diabetes Mellitus; Genes, Recessive; Homozygote; Hum

2012
Mitochondrial diabetes, diabetes and the thiamine-responsive megaloblastic anaemia syndrome and MODY-2. Diseases with common pathophysiology?
    Panminerva medica, 2002, Volume: 44, Issue:4

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Diabetes Mellitus, Type 2; Humans; Mitochondrial Diseases;

2002
[Prophylactic and therapeutic application of thiamine (vitamin B1)--a new point of view].
    Wiadomosci lekarskie (Warsaw, Poland : 1960), 2006, Volume: 59, Issue:5-6

    Topics: Animals; Cardiovascular Diseases; Comorbidity; Diabetes Mellitus; Diabetic Angiopathies; Humans; Pai

2006
[Diabetes and vitamin levels].
    Nihon rinsho. Japanese journal of clinical medicine, 1999, Volume: 57, Issue:10

    Topics: Ascorbic Acid; Diabetes Mellitus; Humans; Thiamine; Vitamin B 12; Vitamin E

1999
Advances in the development of functional foods from buckwheat.
    Critical reviews in food science and nutrition, 2001, Volume: 41, Issue:6

    Topics: Allergens; Animals; Carrier Proteins; China; Constipation; Diabetes Mellitus; Fagopyrum; Food Hypers

2001

Trials

2 trials available for thiamine and Diabetes Mellitus

ArticleYear
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
    Sexuality research & social policy : journal of NSRC : SR & SP, 2023, Apr-05

    Topics: Acute Kidney Injury; Adult; Aged; Albumins; Alloys; Amides; Amino Acids; Animals; Antineoplastic Com

2023
Treatment with benfotiamine in patients with diabetic sensorimotor polyneuropathy: A double-blind, randomized, placebo-controlled, parallel group pilot study over 12 months.
    Journal of diabetes and its complications, 2020, Volume: 34, Issue:12

    Topics: Diabetes Mellitus; Diabetic Neuropathies; Double-Blind Method; Humans; Pilot Projects; Polyneuropath

2020

Other Studies

96 other studies available for thiamine and Diabetes Mellitus

ArticleYear
Leber congenital amaurosis as an initial manifestation in a Chinese patient with thiamine-responsive megaloblastic anemia syndrome.
    American journal of medical genetics. Part A, 2022, Volume: 188, Issue:3

    Topics: Adolescent; Anemia, Megaloblastic; Child; China; Diabetes Mellitus; Hearing Loss, Sensorineural; Hum

2022
Atypical presentation of thiamine-responsive megaloblastic anemia in a Chinese baby boy.
    International journal of laboratory hematology, 2022, Volume: 44, Issue:4

    Topics: Anemia, Megaloblastic; Asian People; China; Diabetes Mellitus; Humans; Infant; Male; Thiamine

2022
Whole-Exome Sequencing Revealed a Pathogenic Nonsense Variant in the SLC19A2 Gene in an Iranian Family with Thiamine-Responsive Megaloblastic Anemia.
    Laboratory medicine, 2022, Nov-03, Volume: 53, Issue:6

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Exome Sequencing; Female; Humans; Iran; Male; Membrane Tra

2022
U-shaped association between dietary thiamine intake and new-onset diabetes: a nationwide cohort study.
    QJM : monthly journal of the Association of Physicians, 2022, Dec-12, Volume: 115, Issue:12

    Topics: Adult; China; Cohort Studies; Diabetes Mellitus; Diet; Glycated Hemoglobin; Humans; Prospective Stud

2022
Thiamine responsive megaloblastic anaemia complicated with acute Parvovirus infection: A case report.
    JPMA. The Journal of the Pakistan Medical Association, 2023, Volume: 73, Issue:1

    Topics: Adolescent; Anemia, Megaloblastic; Deafness; Diabetes Mellitus; Hearing Loss, Sensorineural; Humans;

2023
An Italian case series' description of thiamine responsive megaloblastic anemia syndrome: importance of early diagnosis and treatment.
    Italian journal of pediatrics, 2023, Nov-30, Volume: 49, Issue:1

    Topics: Adult; Anemia, Megaloblastic; Child; Deafness; Diabetes Mellitus; Early Diagnosis; Hearing Loss, Sen

2023
Thiamine pyrophosphate diminishes nitric oxide synthesis in endothelial cells.
    International journal for vitamin and nutrition research. Internationale Zeitschrift fur Vitamin- und Ernahrungsforschung. Journal international de vitaminologie et de nutrition, 2021, Volume: 91, Issue:5-6

    Topics: Cells, Cultured; Diabetes Mellitus; Endothelial Cells; Glucose; Humans; Infant, Newborn; Nitric Oxid

2021
Relapse of rare diseases during COVID-19 pandemic: bicytopenia in an adult patient with thiamine-responsive megaloblastic anaemia.
    The Pan African medical journal, 2020, Volume: 35, Issue:Suppl 2

    Topics: Acute Coronary Syndrome; Anemia, Megaloblastic; Betacoronavirus; Chest Pain; Coronavirus Infections;

2020
Thiamine-Responsive Megaloblastic Anemia-Related Diabetes: Long-Term Clinical Outcomes in 23 Pediatric Patients From the DPV and SWEET Registries.
    Canadian journal of diabetes, 2021, Volume: 45, Issue:6

    Topics: Adolescent; Anemia, Megaloblastic; Child; Cohort Studies; Diabetes Mellitus; Female; Humans; Male; R

2021
Identification of novel compound heterozygous variants in SLC19A2 and the genotype-phenotype associations in thiamine-responsive megaloblastic anemia.
    Clinica chimica acta; international journal of clinical chemistry, 2021, Volume: 516

    Topics: Anemia, Megaloblastic; Asia; Diabetes Mellitus; Genetic Association Studies; Hearing Loss, Sensorine

2021
Thiamine and diabetes: back to the future?
    Acta diabetologica, 2021, Volume: 58, Issue:11

    Topics: Animals; Diabetes Complications; Diabetes Mellitus; Diabetic Nephropathies; Diabetic Neuropathies; H

2021
Recovered insulin production after thiamine administration in permanent neonatal diabetes mellitus with a novel solute carrier family 19 member 2 (SLC19A2) mutation.
    Journal of diabetes, 2018, Volume: 10, Issue:1

    Topics: Diabetes Mellitus; Homozygote; Humans; Infant; Insulin; Male; Membrane Transport Proteins; Mutation;

2018
Pharmacogenomics in diabetes: outcomes of thiamine therapy in TRMA syndrome.
    Diabetologia, 2018, Volume: 61, Issue:5

    Topics: Alleles; Anemia, Megaloblastic; Child, Preschool; Cohort Studies; Diabetes Mellitus; Female; Genetic

2018
Thiamine Responsive Megaloblastic Anaemia, Diabetes Mellitus and Sensorineural Hearing Loss in a Child.
    Journal of the College of Physicians and Surgeons--Pakistan : JCPSP, 2018, Volume: 28, Issue:9

    Topics: Anemia, Megaloblastic; Blood Glucose; Child, Preschool; Diabetes Mellitus; Female; Glycated Hemoglob

2018
Arrhythmia in thiamine responsive megaloblastic anemia syndrome.
    The Turkish journal of pediatrics, 2018, Volume: 60, Issue:3

    Topics: Anemia, Megaloblastic; Arrhythmias, Cardiac; Child, Preschool; Diabetes Mellitus; Electrocardiograph

2018
Loss-of-Function Mutation in Thiamine Transporter 1 in a Family With Autosomal Dominant Diabetes.
    Diabetes, 2019, Volume: 68, Issue:5

    Topics: Anemia, Megaloblastic; Cell Cycle Checkpoints; Diabetes Mellitus; Humans; Insulin; Membrane Transpor

2019
An Adult Case of Thiamine-Sensitive Megaloblastic Anemia Syndrome Accidentally Diagnosed Myelodysplastic Syndrome.
    Journal of the College of Physicians and Surgeons--Pakistan : JCPSP, 2019, Volume: 29, Issue:6

    Topics: Adult; Anemia, Megaloblastic; Blood Glucose; Diabetes Mellitus; Diabetes Mellitus, Type 1; Diagnosis

2019
Neonatal diabetes mellitus: remission induced by novel therapy.
    BMJ case reports, 2019, Jun-25, Volume: 12, Issue:6

    Topics: Administration, Oral; Anemia, Megaloblastic; Diabetes Mellitus; Diabetic Ketoacidosis; Female; Frame

2019
TRMA syndrome with a severe phenotype, cerebral infarction, and novel compound heterozygous SLC19A2 mutation: a case report.
    BMC pediatrics, 2019, 07-11, Volume: 19, Issue:1

    Topics: Adult; Anemia, Megaloblastic; Arrhythmias, Cardiac; Atrophy; Brain; Cerebral Infarction; Child; Diab

2019
Identification of a SLC19A2 nonsense mutation in Persian families with thiamine-responsive megaloblastic anemia.
    Gene, 2013, May-01, Volume: 519, Issue:2

    Topics: Anemia, Megaloblastic; Child; Child, Preschool; Codon, Nonsense; Diabetes Mellitus; Female; Genetic

2013
Leber's congenital amaurosis as the retinal degenerative phenotype in thiamine responsive megaloblastic anemia: a case report.
    Ophthalmic genetics, 2014, Volume: 35, Issue:2

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Exons; Female; Hearing Loss, Sensorineural; Humans; Infant

2014
Antiglycation activity of thiamin-HCl and benfotiamine in diabetic condition.
    JPMA. The Journal of the Pakistan Medical Association, 2012, Volume: 62, Issue:10

    Topics: Blood Glucose; Chelating Agents; Diabetes Mellitus; Dose-Response Relationship, Drug; Glycation End

2012
Novel mutation in the SLC19A2 gene in an Iranian family with thiamine-responsive megaloblastic anemia: a series of three cases.
    Journal of clinical research in pediatric endocrinology, 2013, Sep-10, Volume: 5, Issue:3

    Topics: Anemia, Megaloblastic; Child, Preschool; Consanguinity; Diabetes Mellitus; Female; Hearing Loss, Sen

2013
Thiamine responsive megaloblastic anemia: the puzzling phenotype.
    Pediatric blood & cancer, 2014, Volume: 61, Issue:3

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Female; Hearing Loss, Sensorineural; Humans; Infant; Male;

2014
Identification of four SLC19A2 mutations in four Chinese thiamine responsive megaloblastic anemia patients without diabetes.
    Blood cells, molecules & diseases, 2014, Volume: 52, Issue:4

    Topics: Amino Acid Substitution; Anemia, Megaloblastic; Child; Child, Preschool; Diabetes Mellitus; DNA Muta

2014
Recurrent psychiatric manifestations in thiamine-responsive megaloblastic anemia syndrome due to a novel mutation c.63_71 delACCGCTC in the gene SLC19A2.
    Psychiatry and clinical neurosciences, 2014, Volume: 68, Issue:6

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Hearing Loss, Sensorineural; Humans; Ketoglutarate Dehydro

2014
Thiamine responsive megaloblastic anemia syndrome: a novel homozygous SLC19A2 gene mutation identified.
    American journal of medical genetics. Part A, 2015, Volume: 167, Issue:7

    Topics: Anemia, Megaloblastic; Base Sequence; Child, Preschool; Diabetes Mellitus; Hearing Loss, Sensorineur

2015
Beta cell function and clinical course in three siblings with thiamine-responsive megaloblastic anemia (TRMA) treated with thiamine supplementation.
    Journal of pediatric endocrinology & metabolism : JPEM, 2017, Feb-01, Volume: 30, Issue:2

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Female; Hearing Loss, Sensorineural; Humans; Infant; Infan

2017
A novel mutation in the SLC19A2 gene in a Turkish female with thiamine-responsive megaloblastic anemia syndrome.
    Journal of tropical pediatrics, 2009, Volume: 55, Issue:4

    Topics: Anemia, Megaloblastic; Child, Preschool; Diabetes Mellitus; Diagnosis, Differential; Female; Genotyp

2009
Ameliorative effect of combination of benfotiamine and fenofibrate in diabetes-induced vascular endothelial dysfunction and nephropathy in the rat.
    Molecular and cellular biochemistry, 2009, Volume: 320, Issue:1-2

    Topics: Angiotensin-Converting Enzyme Inhibitors; Animals; Blood Glucose; Chelating Agents; Diabetes Mellitu

2009
Allossan diabetes; Hepatic and muscle glycognosis in rats with alloxanic diabetes.
    Bollettino della Societa italiana di biologia sperimentale, 1947, Volume: 23, Issue:3

    Topics: Diabetes Mellitus; Glycogen; Liver; Muscles; Thiamine; Thiazoles; Tissues

1947
Thiamine responsive megaloblastic anemia.
    Indian pediatrics, 2009, Volume: 46, Issue:2

    Topics: Anemia, Megaloblastic; Child; Comorbidity; Diabetes Mellitus; Female; Hearing Loss, Sensorineural; H

2009
Thiamine-responsive megaloblastic anemia syndrome: long term follow-up.
    The Journal of pediatrics, 2009, Volume: 155, Issue:2

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Female; Follow-Up Studies; Hearing Loss, Sensorineural; Hu

2009
Thiamine-responsive megaloblastic anemia: identification of novel compound heterozygotes and mutation update.
    The Journal of pediatrics, 2009, Volume: 155, Issue:6

    Topics: Adult; Anemia, Megaloblastic; Child; Child, Preschool; Cohort Studies; Deafness; Diabetes Mellitus;

2009
Diabetic acido-ketosis revealing thiamine-responsive megaloblastic anemia.
    Annales d'endocrinologie, 2009, Volume: 70, Issue:6

    Topics: Anemia, Megaloblastic; Blood Cell Count; Bone Marrow Cells; Diabetes Mellitus; Diabetic Ketoacidosis

2009
Thiamine deficiency in adult normal and diabetic rats as studied under paired-feeding conditions.
    Federation proceedings, 1947, Volume: 6, Issue:1 Pt 2

    Topics: Animals; Diabetes Mellitus; Diabetes Mellitus, Experimental; Diet; Feeding and Eating Disorders; Rat

1947
Thiamine-responsive megaloblastic anemia syndrome.
    International journal of hematology, 2010, Volume: 92, Issue:3

    Topics: Anemia, Megaloblastic; Blood Component Transfusion; Bone Marrow; Diabetes Mellitus; Hearing Loss, Se

2010
Could thiamine pyrophosphate be a regulator of the nitric oxide synthesis in the endothelial cell of diabetic patients?
    Medical hypotheses, 2011, Volume: 76, Issue:5

    Topics: Cell Survival; Diabetes Complications; Diabetes Mellitus; Endothelial Cells; Enzyme Activation; Huma

2011
Does early treatment prevent deafness in thiamine-responsive megaloblastic anaemia syndrome?
    Journal of clinical research in pediatric endocrinology, 2011, Volume: 3, Issue:1

    Topics: Anemia, Megaloblastic; Diabetes Mellitus; Female; Hearing Loss, Sensorineural; Humans; Infant; Ketog

2011
Thiamine-responsive megaloblastic anemia syndrome: a novel mutation.
    Genetic counseling (Geneva, Switzerland), 2012, Volume: 23, Issue:2

    Topics: Anemia, Megaloblastic; Child, Preschool; Diabetes Mellitus; Diagnosis, Differential; Female; Follow-

2012
Glucose-induced down regulation of thiamine transporters in the kidney proximal tubular epithelium produces thiamine insufficiency in diabetes.
    PloS one, 2012, Volume: 7, Issue:12

    Topics: Animals; Cells, Cultured; Diabetes Mellitus; Diabetic Nephropathies; Down-Regulation; Epithelium; Gl

2012
Thiamine responsive megaloblastic anemia: a novel SLC19A2 compound heterozygous mutation in two siblings.
    Pediatric diabetes, 2013, Volume: 14, Issue:5

    Topics: Adult; Anemia, Megaloblastic; Child; Child, Preschool; Diabetes Mellitus; Female; Hearing Loss, Sens

2013
Uncovering the beginning of diabetes: the cellular redox status and oxidative stress as starting players in hyperglycemic damage.
    Molecular and cellular biochemistry, 2013, Volume: 376, Issue:1-2

    Topics: Azaserine; Diabetes Mellitus; Glucose; Glycolysis; Hep G2 Cells; Hexosamines; Humans; Hyperglycemia;

2013
Targeted disruption of Slc19a2, the gene encoding the high-affinity thiamin transporter Thtr-1, causes diabetes mellitus, sensorineural deafness and megaloblastosis in mice.
    Human molecular genetics, 2002, Nov-01, Volume: 11, Issue:23

    Topics: Anemia, Megaloblastic; Animals; Bone Marrow; Brain; Diabetes Mellitus; DNA Primers; Erythrocytes; Ge

2002
[Phosphorylation of thiamine in diabetes mellitus].
    Bollettino della Societa italiana di biologia sperimentale, 1952, Volume: 28, Issue:2

    Topics: Biochemical Phenomena; Diabetes Mellitus; Humans; Phosphorylation; Thiamine

1952
Excretion of thiamine in diabetes: unrelated to glycosuria and polyuria.
    Scandinavian journal of clinical and laboratory investigation, 1961, Volume: 13

    Topics: Diabetes Mellitus; Glycosuria; Humans; Polyuria; Thiamine; Vitamin A

1961
DIABETIC ENTEROPATHY.
    The American journal of gastroenterology, 1963, Volume: 40

    Topics: Anti-Bacterial Agents; Celiac Disease; Diabetes Complications; Diabetes Mellitus; Diagnosis, Differe

1963
[EFFECT OF THIAMINE DIPHOSPHATE ON LIPID METABOLISM IN PATIENTS WITH DIABETES MELLITUS].
    Zdravookhranenie Belorussii, 1963, Volume: 9

    Topics: Adolescent; Child; Cholesterol; Diabetes Mellitus; Diabetes Mellitus, Type 1; Humans; Insulin; Lecit

1963
[ON THE RATIONALITY OF THE HYDROXYCOBALAMINE-THIAMINE MONOPHOSPHORIC ESTER COMBINATION IN TREATMENT].
    Il Policlinico. Sezione medica, 1963, Volume: 70

    Topics: Diabetes Mellitus; Diabetic Neuropathies; Geriatrics; Hepatitis; Hepatitis A; Humans; Hydroxocobalam

1963
[ON THE EFFECTS OF ATP LOAD ON ENDOGENOUS ARTERIOVENOUS BLOOD SUGAR, PYRUVATES AND ALCOHOL IN DIABETIC SUBJECTS].
    L' Arcispedale S. Anna di Ferrara, 1963, Volume: 16

    Topics: Adenosine Triphosphate; Blood Chemical Analysis; Blood Glucose; Diabetes Mellitus; Ethanol; Liver Fu

1963
[TREATMENT OF DIABETIC NEURITIS WITH LIPOPHILIC VITAMIN B1 PREPARATION].
    Munchener medizinische Wochenschrift (1950), 1964, Jan-24, Volume: 106

    Topics: Diabetes Mellitus; Diabetic Neuropathies; Humans; Neuritis; Thiamine

1964
THE BLOOD CONCENTRATION OF THIAMINE IN DIABETES.
    Scandinavian journal of clinical and laboratory investigation, 1964, Volume: 16

    Topics: Blood; Blood Glucose; Diabetes Mellitus; Diabetes Mellitus, Type 1; Drug Therapy; Fasting; Geriatric

1964
CORTISONE THERAPY IN FASCICULITIS OPTICA.
    Ophthalmologica. Journal international d'ophtalmologie. International journal of ophthalmology. Zeitschrift fur Augenheilkunde, 1964, Volume: 148

    Topics: Anti-Bacterial Agents; Cortisone; Diabetes Mellitus; Drug Therapy; Humans; Multiple Sclerosis; Optic

1964
[ON THE USE OF THIANEURONE, A NEW ORAL VITAMIN B-1 PREPARATION, FOR THE THERAPY OF NEURITIC PAINS].
    Wiener klinische Wochenschrift, 1964, Mar-27, Volume: 76

    Topics: Anemia; Arthritis; Arthritis, Rheumatoid; Back Pain; Beriberi; Cardiovascular Diseases; Diabetes Mel

1964
NEUROPATHY OF DIABETES MELLITUS.
    JAMA, 1965, Apr-19, Volume: 192

    Topics: Diabetes Mellitus; Diabetic Neuropathies; Drug Therapy; Hematinics; Humans; Pathology; Peripheral Ne

1965
Disruption of transport activity in a D93H mutant thiamine transporter 1, from a Rogers Syndrome family.
    European journal of biochemistry, 2003, Volume: 270, Issue:22

    Topics: Amino Acid Sequence; Anemia, Megaloblastic; Animals; Aspartic Acid; Biological Transport; Cell Line;

2003
[Hypophysial diabetes and its therapy with vitamin B1].
    Orvosi hetilap, 1951, Oct-28, Volume: 92, Issue:43

    Topics: Diabetes Mellitus; Humans; Thiamine; Vitamin B Complex; Vitamins

1951
[Effect of chlorhydrate of thiamine mono-, di-, and triphosphoric esters on intermediary carbohydrate metabolism in diabetes mellitus].
    Il Progresso medico, 1951, Dec-15, Volume: 7, Issue:23

    Topics: Carbohydrate Metabolism; Diabetes Mellitus; Esters; Humans; Thiamine; Vitamin B Complex; Vitamins

1951
Pyruvate content of blood and phosphorylation of thiamine in diabetic coma.
    Biochimica et biophysica acta, 1952, Volume: 8, Issue:3

    Topics: Biochemical Phenomena; Diabetes Mellitus; Diabetic Coma; Humans; Phosphorylation; Pyruvates; Pyruvic

1952
Novel mutation in the SLC19A2 gene in an African-American female with thiamine-responsive megaloblastic anemia syndrome.
    American journal of medical genetics. Part A, 2004, Mar-15, Volume: 125A, Issue:3

    Topics: Anemia, Megaloblastic; Black or African American; Diabetes Mellitus; DNA Mutational Analysis; Female

2004
Thiamine-responsive megaloblastic anaemia syndrome: long-term follow-up and mutation analysis of seven families.
    Acta paediatrica (Oslo, Norway : 1992), 2006, Volume: 95, Issue:1

    Topics: Adolescent; Adult; Anemia, Megaloblastic; Child; Child, Preschool; Deafness; Diabetes Mellitus; DNA

2006
Targeting and intracellular trafficking of clinically relevant hTHTR1 mutations in human cell lines.
    Clinical science (London, England : 1979), 2007, Volume: 113, Issue:2

    Topics: Anemia, Megaloblastic; Cell Line; Cytoplasm; Deafness; Diabetes Mellitus; DNA Mutational Analysis; F

2007
Lowering homocysteine decreases levels and expression of VEGF(165) and endostatin.
    The Journal of surgical research, 2008, May-15, Volume: 146, Issue:2

    Topics: Adult; Diabetes Mellitus; Down-Regulation; Endostatins; Female; Folic Acid; Gene Expression; Homocys

2008
Thiamine-responsive megaloblastic anaemia: a cause of syndromic diabetes in childhood.
    Pediatric diabetes, 2007, Volume: 8, Issue:4

    Topics: Anemia, Megaloblastic; Child, Preschool; Consanguinity; Diabetes Complications; Diabetes Mellitus; D

2007
About the Influence of the B1 Vitamin on the sugar turnover in alloxan diabetes.
    Experientia, 1948, Oct-15, Volume: 4, Issue:10

    Topics: Alloxan; Animals; Blood Glucose; Diabetes Mellitus; Diabetes Mellitus, Experimental; Thiamine

1948
Phlorhizin diabetes in vitamin deficiencies; conversion of protein to glucose, and serum phosphatase, during deficiencies of thiamine, riboflavin, and pyridoxine.
    The Journal of nutrition, 1949, Jan-10, Volume: 37, Issue:1

    Topics: Avitaminosis; Deficiency Diseases; Diabetes Mellitus; Glucose; Humans; Phlorhizin; Phosphoric Monoes

1949
The role of vitamin B1 in the treatment of diabetes.
    The Indian medical gazette, 1949, Volume: 83, Issue:12

    Topics: Diabetes Mellitus; Humans; Thiamine

1949
Thiamine responsive anaemia: a study of two further cases.
    British journal of haematology, 1982, Volume: 50, Issue:4

    Topics: Anemia, Macrocytic; Bone Marrow; Child; Deafness; Deoxyuridine; Diabetes Mellitus; DNA; Erythroblast

1982
[Use of preparations of group B vitamins also at high dosage and by the parenteral route in a population of hospitalized patients].
    Acta vitaminologica et enzymologica, 1984, Volume: 6, Issue:4

    Topics: Adult; Aged; Diabetes Mellitus; Drug Administration Schedule; Female; Humans; Liver Cirrhosis; Male;

1984
[Vitamin status in diabetic neuropathy (thiamine, riboflavin, pyridoxin, cobalamin and tocopherol)].
    Zeitschrift fur Ernahrungswissenschaft, 1980, Volume: 19, Issue:1

    Topics: Diabetes Complications; Diabetes Mellitus; Diabetic Neuropathies; Diabetic Retinopathy; Female; Huma

1980
[Vitamin status of hospitalized patients. Critical study of methods and results].
    Bibliotheca nutritio et dieta, 1981, Issue:30

    Topics: Ascorbic Acid; Avitaminosis; Clinical Enzyme Tests; Diabetes Mellitus; Female; Gastrointestinal Dise

1981
Relationship between thiamine and glucose levels in diabetes mellitus.
    Japanese journal of pharmacology, 1981, Volume: 31, Issue:6

    Topics: Animals; Blood Glucose; Diabetes Mellitus; Diabetic Neuropathies; Humans; Male; Rabbits; Thiamine

1981
Thiamine corrects delayed replication and decreases production of lactate and advanced glycation end-products in bovine retinal and human umbilical vein endothelial cells cultured under high glucose conditions.
    Diabetologia, 1996, Volume: 39, Issue:11

    Topics: Animals; Cattle; Cell Count; Cell Division; Cells, Cultured; Diabetes Mellitus; Endothelium; Endothe

1996
Congenital permanent diabetes: a different type of diabetes?
    Acta paediatrica (Oslo, Norway : 1992), 1996, Volume: 85, Issue:12

    Topics: Child; Child, Preschool; Diabetes Mellitus; HLA-DR Antigens; Humans; Male; Pedigree; Thiamine; Ultra

1996
Refined mapping of the gene for thiamine-responsive megaloblastic anemia syndrome and evidence for genetic homogeneity.
    Human genetics, 1998, Volume: 103, Issue:4

    Topics: Anemia, Megaloblastic; Child; Child, Preschool; Chromosome Mapping; Chromosomes, Human, Pair 1; Deaf

1998
Defective high-affinity thiamine transporter leads to cell death in thiamine-responsive megaloblastic anemia syndrome fibroblasts.
    The Journal of clinical investigation, 1999, Volume: 103, Issue:5

    Topics: Anemia, Megaloblastic; Apoptosis; Carrier Proteins; Cells, Cultured; Deafness; Diabetes Mellitus; Fi

1999
Mutations in a new gene encoding a thiamine transporter cause thiamine-responsive megaloblastic anaemia syndrome.
    Nature genetics, 1999, Volume: 22, Issue:3

    Topics: Amino Acid Sequence; Anemia, Megaloblastic; Base Sequence; Carrier Proteins; Cloning, Molecular; Dea

1999
Mutations in SLC19A2 cause thiamine-responsive megaloblastic anaemia associated with diabetes mellitus and deafness.
    Nature genetics, 1999, Volume: 22, Issue:3

    Topics: Amino Acid Sequence; Anemia, Megaloblastic; Animals; Base Sequence; Carrier Proteins; Cricetinae; De

1999
The gene mutated in thiamine-responsive anaemia with diabetes and deafness (TRMA) encodes a functional thiamine transporter.
    Nature genetics, 1999, Volume: 22, Issue:3

    Topics: Amino Acid Sequence; Anemia, Megaloblastic; Animals; Base Sequence; Carrier Proteins; Cell Line; Dea

1999
The spectrum of mutations, including four novel ones, in the thiamine-responsive megaloblastic anemia gene SLC19A2 of eight families.
    Human mutation, 2000, Volume: 16, Issue:1

    Topics: Alleles; Anemia, Megaloblastic; Carrier Proteins; Deafness; Diabetes Mellitus; DNA Mutational Analys

2000
Suppression of the accumulation of triosephosphates and increased formation of methylglyoxal in human red blood cells during hyperglycaemia by thiamine in vitro.
    Journal of biochemistry, 2001, Volume: 129, Issue:4

    Topics: Deoxyglucose; Diabetes Complications; Diabetes Mellitus; Erythrocytes; Glucose; Glycolysis; Humans;

2001
Five years followup of diabetes mellitus in two siblings with thiamine responsive megaloblastic anemia.
    Indian pediatrics, 2001, Volume: 38, Issue:11

    Topics: Abnormalities, Multiple; Anemia, Megaloblastic; Blood Glucose; Diabetes Mellitus; Female; Follow-Up

2001
A hybrid input-output approach to model metabolic systems: an application to intracellular thiamine kinetics.
    Journal of biomedical informatics, 2001, Volume: 34, Issue:4

    Topics: Animals; Computational Biology; Diabetes Mellitus; Fuzzy Logic; Humans; Intestinal Absorption; Intra

2001
Thiamine-responsive megaloblastic anemia, sensorineural deafness, and diabetes mellitus: A new syndrome?
    The Journal of pediatrics, 1978, Volume: 93, Issue:2

    Topics: Adult; Anemia, Macrocytic; Anemia, Megaloblastic; Child; Deafness; Diabetes Mellitus; Diabetes Melli

1978
The transketolase assay of thiamine in some diseases.
    The American journal of clinical nutrition, 1977, Volume: 30, Issue:10

    Topics: Anemia, Macrocytic; Anemia, Pernicious; Apoenzymes; Diabetes Mellitus; Diabetes Mellitus, Type 1; Er

1977
[Physical work capacity in diabetes mellitus under treatment].
    Sovetskaia meditsina, 1989, Issue:7

    Topics: Adult; Diabetes Mellitus; Drug Combinations; Exercise Test; Female; Humans; Inosine Diphosphate; Pyr

1989
Blood thiamine levels in outpatients with diabetes mellitus.
    Journal of nutritional science and vitaminology, 1987, Volume: 33, Issue:6

    Topics: Adult; Ambulatory Care; Diabetes Mellitus; Erythrocytes; Female; Humans; Male; Middle Aged; Reflex;

1987
The relationship between blood thiamine levels and dietary thiamine content in diabetic outpatients and healthy subjects.
    Journal of nutritional science and vitaminology, 1987, Volume: 33, Issue:6

    Topics: Adult; Diabetes Mellitus; Diet; Dietary Carbohydrates; Dietary Fats; Dietary Proteins; Energy Intake

1987
Diabetes mellitus, thiamine-dependent megaloblastic anemia, and sensorineural deafness associated with deficient alpha-ketoglutarate dehydrogenase activity.
    The Journal of pediatrics, 1985, Volume: 107, Issue:4

    Topics: Adolescent; Anemia, Macrocytic; Anemia, Megaloblastic; Child; Child, Preschool; Deafness; Diabetes M

1985
[Activity of blood transketolase in patients with diabetes mellitus].
    Vrachebnoe delo, 1973, Volume: 1

    Topics: Adolescent; Adult; Aged; Diabetes Mellitus; Female; Humans; Male; Middle Aged; Thiamine; Transketola

1973
[Diabetic neuropathy].
    Deutsches medizinisches Journal, 1969, Apr-20, Volume: 20, Issue:8

    Topics: Aged; Diabetes Mellitus; Diabetic Neuropathies; Humans; Middle Aged; Thiamine; Thioctic Acid

1969
[Dietetics and nutrition in 1972].
    La Revue du praticien, 1972, Feb-21, Volume: 22, Issue:6

    Topics: Adipose Tissue; Alcoholic Intoxication; Anorexia Nervosa; Caffeine; Calcium; Catecholamines; Coffee;

1972
[Concurrence of diabetes mellitus and hypothyrosis in the diencephalic syndrome].
    Klinicheskaia meditsina, 1971, Volume: 49, Issue:9

    Topics: Adult; Ascorbic Acid; Blood Glucose; Cholesterol; Diabetes Complications; Diabetes Mellitus; Diencep

1971
[Excretion of thiamine and riboflavin in the urine].
    Ceskoslovenska gastroenterologie a vyziva, 1969, Volume: 23, Issue:8

    Topics: Diabetes Mellitus; Food Service, Hospital; Humans; Nutrition Disorders; Nutritional Requirements; Ri

1969
[Studies on the question of an interrelationship between saluretic acting thiazide derivatives and thiamine in diabetes].
    Klinische Wochenschrift, 1968, Nov-01, Volume: 46, Issue:21

    Topics: Blood Glucose; Diabetes Mellitus; Glucose Tolerance Test; Humans; Hydrochlorothiazide; Natriuresis;

1968
[Studies on the influrence of saluretic thiazide derivatives and thiamine on carbohydrate metabolism].
    Verhandlungen der Deutschen Gesellschaft fur Innere Medizin, 1968, Volume: 74

    Topics: Carbohydrate Metabolism; Diabetes Mellitus; Diuretics; Furosemide; Glucose Tolerance Test; Humans; H

1968