tetradecanoylphorbol acetate has been researched along with Left Ventricular Hypertrophy in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Crozatier, B; Duquesnes, N; Lezoualc'h, F | 1 |
| Goto, K; Iemitsu, M; Irukayama-Tomobe, Y; Kasuya, Y; Miyauchi, T; Ogata, T; Sakai, S; Sudo, T; Takanashi, M; Yamaguchi, I | 1 |
1 review(s) available for tetradecanoylphorbol acetate and Left Ventricular Hypertrophy
| Article | Year |
|---|---|
PKC-delta and PKC-epsilon: foes of the same family or strangers?
Topics: Animals; Apoptosis; Cell Differentiation; Enzyme Activation; Enzyme Inhibitors; Guinea Pigs; Humans; Hypertrophy, Left Ventricular; Isoenzymes; Mice; Myocardial Ischemia; Myocardium; Organ Specificity; Protein Binding; Protein Kinase C-alpha; Protein Kinase C-delta; Protein Kinase C-epsilon; Protein Structure, Tertiary; Rabbits; Rats; Signal Transduction; Tetradecanoylphorbol Acetate | 2011 |
1 other study(ies) available for tetradecanoylphorbol acetate and Left Ventricular Hypertrophy
| Article | Year |
|---|---|
Endothelin-1-induced cardiac hypertrophy is inhibited by activation of peroxisome proliferator-activated receptor-alpha partly via blockade of c-Jun NH2-terminal kinase pathway.
Topics: Animals; Animals, Newborn; Aorta; Cells, Cultured; Disease Models, Animal; Endothelin-1; Fenofibrate; Genes, fos; Genes, jun; Hypertrophy, Left Ventricular; JNK Mitogen-Activated Protein Kinases; Ligation; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Myocytes, Cardiac; Phosphorylation; Protein Processing, Post-Translational; Proto-Oncogene Proteins c-fos; Proto-Oncogene Proteins c-jun; Rats; Rats, Sprague-Dawley; Receptors, Cytoplasmic and Nuclear; RNA, Messenger; Signal Transduction; Tetradecanoylphorbol Acetate; Transcription Factor AP-1; Transcription Factors | 2004 |