tetradecanoylphorbol acetate has been researched along with Autosomal Dominant Cerebellar Ataxia, Type II in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Goedhart, J; Jezierska, J; Kampinga, HH; Reits, EA; Verbeek, DS | 1 |
| Bruinsma, L; Goedhart, J; Reits, EA; Sinke, RJ; Verbeek, DS | 1 |
2 other study(ies) available for tetradecanoylphorbol acetate and Autosomal Dominant Cerebellar Ataxia, Type II
| Article | Year |
|---|---|
SCA14 mutation V138E leads to partly unfolded PKCĪ³ associated with an exposed C-terminus, altered kinetics, phosphorylation and enhanced insolubilization.
Topics: Animals; Blotting, Western; Carcinogens; Chlorocebus aethiops; COS Cells; DNA; Fluorescence Resonance Energy Transfer; HEK293 Cells; Humans; Kinetics; Mutation, Missense; Phosphorylation; Polyethylene Glycols; Protein Folding; Protein Kinase C; Protein Serine-Threonine Kinases; Pyruvate Dehydrogenase Acetyl-Transferring Kinase; Solubility; Solvents; Spinocerebellar Ataxias; Tetradecanoylphorbol Acetate | 2014 |
PKC gamma mutations in spinocerebellar ataxia type 14 affect C1 domain accessibility and kinase activity leading to aberrant MAPK signaling.
Topics: Amino Acid Sequence; Cell Membrane; Cell Nucleus; Cell Survival; Enzyme Activation; HeLa Cells; Humans; Kinetics; MAP Kinase Signaling System; Mitogen-Activated Protein Kinase 1; Molecular Sequence Data; Mutation; Protein Kinase C; Protein Structure, Tertiary; Protein Transport; Spinocerebellar Ataxias; Subcellular Fractions; Tetradecanoylphorbol Acetate | 2008 |