tetradecanoylphorbol acetate has been researched along with Aldrich Syndrome in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (25.00) | 18.2507 |
| 2000's | 3 (75.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Candotti, F; Dong, X; Patino-Lopez, G; Shaw, S | 1 |
| Aruffo, A; Miki, H; Nonoyama, S; Ochs, HD; Takenawa, T; Zhu, Q | 1 |
| Boxer, LA; Gunning, WT; Keil, ML; Lachant, NA; Ochs, HD; Omann, GM; Rengan, R; Sweet, LI | 1 |
| Abo, A; Belisle, B; Silvin, C | 1 |
4 other study(ies) available for tetradecanoylphorbol acetate and Aldrich Syndrome
| Article | Year |
|---|---|
Structure-function analysis of the WIP role in T cell receptor-stimulated NFAT activation: evidence that WIP-WASP dissociation is not required and that the WIP NH2 terminus is inhibitory.
Topics: Actins; CD3 Complex; Cytoskeletal Proteins; Humans; Interleukin-2; Intracellular Signaling Peptides and Proteins; Jurkat Cells; Kinetics; Models, Biological; NFATC Transcription Factors; Protein Structure, Tertiary; Receptors, Antigen, T-Cell; Structure-Activity Relationship; Tetradecanoylphorbol Acetate; Transcription, Genetic; Wiskott-Aldrich Syndrome; Wiskott-Aldrich Syndrome Protein | 2007 |
Tyrosine kinase signaling regulates Wiskott-Aldrich syndrome protein function, which is essential for megakaryocyte differentiation.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; Amino Acid Sequence; Animals; Antibodies; Benzoquinones; Cell Differentiation; Cell Line; Humans; Lactams, Macrocyclic; Megakaryocytes; Molecular Sequence Data; Protein Kinase C; Protein-Tyrosine Kinases; Proteins; Quinones; Rabbits; Rifabutin; Signal Transduction; Tetradecanoylphorbol Acetate; Wiskott-Aldrich Syndrome; Wiskott-Aldrich Syndrome Protein | 1997 |
Actin cytoskeletal function is spared, but apoptosis is increased, in WAS patient hematopoietic cells.
Topics: Actins; Adenosine Diphosphate; Apoptosis; Blood Platelets; Cell Survival; Cytochalasin B; Cytoskeleton; Dinoprost; fas Receptor; Humans; In Vitro Techniques; Kinetics; Leukocytes; Lymphocytes; Mutation, Missense; N-Formylmethionine Leucyl-Phenylalanine; Necrosis; Point Mutation; Proteins; Reference Values; src Homology Domains; Tetradecanoylphorbol Acetate; Thrombocytopenia; Wiskott-Aldrich Syndrome; Wiskott-Aldrich Syndrome Protein | 2000 |
A role for Wiskott-Aldrich syndrome protein in T-cell receptor-mediated transcriptional activation independent of actin polymerization.
Topics: Actins; Cell Line; DNA-Binding Proteins; Genes, Reporter; Humans; Ionomycin; Jurkat Cells; Kinetics; Luciferases; Mitogen-Activated Protein Kinases; Mutagenesis; NFATC Transcription Factors; Nuclear Proteins; Proteins; Receptors, Antigen, T-Cell; Recombinant Proteins; Sequence Deletion; T-Lymphocytes; Tetradecanoylphorbol Acetate; Transcription Factors; Transcription, Genetic; Transcriptional Activation; Transfection; Wiskott-Aldrich Syndrome; Wiskott-Aldrich Syndrome Protein | 2001 |