temozolomide has been researched along with Carcinogenesis in 34 studies
Carcinogenesis: The origin, production or development of cancer through genotypic and phenotypic changes which upset the normal balance between cell proliferation and cell death. Carcinogenesis generally requires a constellation of steps, which may occur quickly or over a period of many years.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Although temozolomide (TMZ) is recommended for glioblastoma (GBM) treatment, patients treated with TMZ usually develop TMZ resistance." | 8.12 | Oncogenic Forkhead box D3 antisense RNA 1 promotes cell survival and confers temozolomide resistance in glioblastoma cells through the miR-128-3p/WEE1 G2 checkpoint kinase axis. ( Ling, Z; Liu, Q; Zhang, J, 2022) |
| "It is necessary to elucidate the individual effects of temozolomide (TMZ) on carcinogenesis and tumor resistance to chemotherapy mechanisms." | 8.12 | The Different Temozolomide Effects on Tumorigenesis Mechanisms of Pediatric Glioblastoma PBT24 and SF8628 Cell Tumor in CAM Model and on Cells In Vitro. ( Alonso, MM; Balnytė, I; Damanskienė, E; Preikšaitis, A; Stakišaitis, D; Valančiūtė, A, 2022) |
| "A generally used chemotherapeutic drug for glioma, a frequently diagnosed brain tumour, is temozolomide (TMZ)." | 7.96 | miR-152-5p suppresses glioma progression and tumorigenesis and potentiates temozolomide sensitivity by targeting FBXL7. ( Cao, Y; Fang, Y; He, R; Kong, S; Wang, B; Zhao, Z, 2020) |
| "Glioma is a brain tumour that is often diagnosed, and temozolomide (TMZ) is a common chemotherapeutic drug used in glioma." | 7.96 | MiR-3116 sensitizes glioma cells to temozolomide by targeting FGFR1 and regulating the FGFR1/PI3K/AKT pathway. ( Cao, Y; Kong, S; Li, X; Li, Z; Meng, Y; Xin, Y, 2020) |
| "Resistance of glioblastoma to the chemotherapeutic compound temozolomide is associated with the presence of glioblastoma stem cells in glioblastoma and is a key obstacle for the poor prognosis of glioblastoma." | 7.96 | Phospholipase D1 inhibition sensitizes glioblastoma to temozolomide and suppresses its tumorigenicity. ( Hwang, WC; Kang, DW; Min, DS; Noh, YN; Park, KS, 2020) |
| "Glioma is a frequently diagnosed brain tumors and Temozolomide (TMZ) is a common chemotherapeutic drug for glioma." | 7.91 | MicroRNA-34a-5p suppresses tumorigenesis and progression of glioma and potentiates Temozolomide-induced cytotoxicity for glioma cells by targeting HMGA2. ( Fu, T; Gao, M; Ma, S; Zhao, S, 2019) |
| "Temozolomide (TMZ) is an alkylating agent widely used to treat cancer, resistance to this drug is often found." | 5.51 | Impact of extremely low-frequency electromagnetic field (100 Hz, 100 G) exposure on human glioblastoma U87 cells during Temozolomide administration. ( Ahmadi-Zeidabadi, M; Akbarnejad, Z; Eskandary, H; Esmaeeli, M; Masoumi-Ardakani, Y; Mohammadipoor-Ghasemabad, L, 2019) |
| "Highly malignant gliomas are characterized by pronounced intra‑ and intertumoral heterogeneity." | 5.48 | APOBEC3B is expressed in human glioma, and influences cell proliferation and temozolomide resistance. ( Hattermann, K; Held-Feindt, J; Lucius, R; Schmitt, C; Synowitz, M, 2018) |
| "Although temozolomide (TMZ) is recommended for glioblastoma (GBM) treatment, patients treated with TMZ usually develop TMZ resistance." | 4.12 | Oncogenic Forkhead box D3 antisense RNA 1 promotes cell survival and confers temozolomide resistance in glioblastoma cells through the miR-128-3p/WEE1 G2 checkpoint kinase axis. ( Ling, Z; Liu, Q; Zhang, J, 2022) |
| "It is necessary to elucidate the individual effects of temozolomide (TMZ) on carcinogenesis and tumor resistance to chemotherapy mechanisms." | 4.12 | The Different Temozolomide Effects on Tumorigenesis Mechanisms of Pediatric Glioblastoma PBT24 and SF8628 Cell Tumor in CAM Model and on Cells In Vitro. ( Alonso, MM; Balnytė, I; Damanskienė, E; Preikšaitis, A; Stakišaitis, D; Valančiūtė, A, 2022) |
| " The present study evaluates the effects of AT101, alone or in combination with temozolomide (TMZ), in a microenvironmental glioma stem cell niche model of two GBM cell lines (U251MG and U87MG)." | 4.02 | Effects of the Anti-Tumorigenic Agent AT101 on Human Glioblastoma Cells in the Microenvironmental Glioma Stem Cell Niche. ( Caylioglu, D; Held-Feindt, J; Hellmold, D; Kubelt, C; Meyer, RJ; Synowitz, M, 2021) |
| "A generally used chemotherapeutic drug for glioma, a frequently diagnosed brain tumour, is temozolomide (TMZ)." | 3.96 | miR-152-5p suppresses glioma progression and tumorigenesis and potentiates temozolomide sensitivity by targeting FBXL7. ( Cao, Y; Fang, Y; He, R; Kong, S; Wang, B; Zhao, Z, 2020) |
| "Glioma is a brain tumour that is often diagnosed, and temozolomide (TMZ) is a common chemotherapeutic drug used in glioma." | 3.96 | MiR-3116 sensitizes glioma cells to temozolomide by targeting FGFR1 and regulating the FGFR1/PI3K/AKT pathway. ( Cao, Y; Kong, S; Li, X; Li, Z; Meng, Y; Xin, Y, 2020) |
| "Resistance of glioblastoma to the chemotherapeutic compound temozolomide is associated with the presence of glioblastoma stem cells in glioblastoma and is a key obstacle for the poor prognosis of glioblastoma." | 3.96 | Phospholipase D1 inhibition sensitizes glioblastoma to temozolomide and suppresses its tumorigenicity. ( Hwang, WC; Kang, DW; Min, DS; Noh, YN; Park, KS, 2020) |
| "Glioma is a frequently diagnosed brain tumors and Temozolomide (TMZ) is a common chemotherapeutic drug for glioma." | 3.91 | MicroRNA-34a-5p suppresses tumorigenesis and progression of glioma and potentiates Temozolomide-induced cytotoxicity for glioma cells by targeting HMGA2. ( Fu, T; Gao, M; Ma, S; Zhao, S, 2019) |
| "Glioblastoma is devastating cancer with a high frequency of occurrence and poor survival rate and it is urgent to discover novel glioblastoma-specific antigens for the therapy." | 1.56 | Suppressing Dazl modulates tumorigenicity and stemness in human glioblastoma cells. ( Liu, C; Liu, R; Lu, Y; Zhang, F; Zhang, H, 2020) |
| "Temozolomide (TMZ) is an alkylating agent widely used to treat cancer, resistance to this drug is often found." | 1.51 | Impact of extremely low-frequency electromagnetic field (100 Hz, 100 G) exposure on human glioblastoma U87 cells during Temozolomide administration. ( Ahmadi-Zeidabadi, M; Akbarnejad, Z; Eskandary, H; Esmaeeli, M; Masoumi-Ardakani, Y; Mohammadipoor-Ghasemabad, L, 2019) |
| " In addition, TMZ could increase the levels of miR-505 and combination with pri-miR-505 and TMZ promoted the suppressive role mediated by miR-505 in GBM cells." | 1.48 | Combination with TMZ and miR-505 inhibits the development of glioblastoma by regulating the WNT7B/Wnt/β-catenin signaling pathway. ( Fu, C; Liu, X; Yang, X; Zhang, C, 2018) |
| "Highly malignant gliomas are characterized by pronounced intra‑ and intertumoral heterogeneity." | 1.48 | APOBEC3B is expressed in human glioma, and influences cell proliferation and temozolomide resistance. ( Hattermann, K; Held-Feindt, J; Lucius, R; Schmitt, C; Synowitz, M, 2018) |
| "Malignant gliomas are a group of aggressive neoplasms among human cancers." | 1.46 | Hyperthermia with different temperatures inhibits proliferation and promotes apoptosis through the EGFR/STAT3 pathway in C6 rat glioma cells. ( An, TT; Chen, YD; Dong, TX; Liu, PF; Xu, YT; Yang, XH; Zhang, W; Zhang, Y, 2017) |
| "Glioma is the most frequent primary central nervous system tumor." | 1.46 | β-Elemene Selectively Inhibits the Proliferation of Glioma Stem-Like Cells Through the Downregulation of Notch1. ( Chen, FR; Chen, ZP; Feng, HB; Guo, CC; Jiang, HR; Mei, X; Qu, Y; Sai, K; Wang, J; Yang, QY; Zhang, ZP; Zhao, YY, 2017) |
| "Glioblastoma is the most prevalent primary brain tumor and is essentially universally fatal within 2 years of diagnosis." | 1.40 | Molecular targeting of TRF2 suppresses the growth and tumorigenesis of glioblastoma stem cells. ( Bai, Y; Flavahan, W; Lathia, JD; Mattson, MP; Rich, JN; Zhang, P, 2014) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 16 (47.06) | 24.3611 |
| 2020's | 18 (52.94) | 2.80 |
| Authors | Studies |
|---|---|
| Li, T | 1 |
| Mehraein-Ghomi, F | 1 |
| Forbes, ME | 1 |
| Namjoshi, SV | 1 |
| Ballard, EA | 1 |
| Song, Q | 1 |
| Chou, PC | 1 |
| Wang, X | 7 |
| Parker Kerrigan, BC | 1 |
| Lang, FF | 1 |
| Lesser, G | 1 |
| Debinski, W | 1 |
| Yang, X | 4 |
| Zhang, W | 2 |
| Ling, Z | 1 |
| Zhang, J | 4 |
| Liu, Q | 2 |
| Damanskienė, E | 1 |
| Balnytė, I | 1 |
| Valančiūtė, A | 1 |
| Alonso, MM | 1 |
| Preikšaitis, A | 1 |
| Stakišaitis, D | 1 |
| Elmaci, İ | 1 |
| Altinoz, MA | 1 |
| Kahraman Ozlu, EB | 1 |
| Sari, R | 1 |
| Er, O | 1 |
| Gokhan Ekmekci, C | 1 |
| Turkgenc, B | 1 |
| Ozpinar, A | 2 |
| Hacker, E | 1 |
| Sadeghipour, N | 1 |
| Kumar, SU | 1 |
| Massoud, TF | 1 |
| Paulmurugan, R | 1 |
| Feng, SW | 1 |
| Wu, ZS | 1 |
| Chiu, YL | 1 |
| Huang, SM | 1 |
| Aguado, T | 1 |
| Romero-Revilla, JA | 1 |
| Granados, R | 1 |
| Campuzano, S | 1 |
| Torrente-Rodríguez, RM | 1 |
| Cuesta, ÁM | 1 |
| Albiñana, V | 1 |
| Botella, LM | 1 |
| Santamaría, S | 1 |
| Garcia-Sanz, JA | 1 |
| Pingarrón, JM | 1 |
| Sánchez-Sancho, F | 1 |
| Sánchez-Puelles, JM | 1 |
| Honorato, JR | 1 |
| Hauser-Davis, RA | 1 |
| Saggioro, EM | 1 |
| Correia, FV | 1 |
| Sales-Junior, SF | 1 |
| Soares, LOS | 1 |
| Lima, LDR | 1 |
| Moura-Neto, V | 1 |
| Lopes, GPF | 1 |
| Spohr, TCLS | 1 |
| Kong, S | 2 |
| Fang, Y | 1 |
| Wang, B | 1 |
| Cao, Y | 2 |
| He, R | 1 |
| Zhao, Z | 2 |
| Li, X | 11 |
| Li, Z | 4 |
| Xin, Y | 1 |
| Meng, Y | 2 |
| Zhang, F | 2 |
| Liu, R | 1 |
| Zhang, H | 2 |
| Liu, C | 5 |
| Lu, Y | 2 |
| Kang, DW | 1 |
| Hwang, WC | 1 |
| Noh, YN | 1 |
| Park, KS | 1 |
| Min, DS | 1 |
| Yang, K | 2 |
| Wei, M | 1 |
| Yang, Z | 1 |
| Fu, Z | 1 |
| Xu, R | 1 |
| Cheng, C | 1 |
| Chen, X | 2 |
| Chen, S | 3 |
| Dammer, E | 1 |
| Le, W | 1 |
| Nguépy Keubo, FR | 1 |
| Mboua, PC | 1 |
| Djifack Tadongfack, T | 1 |
| Fokouong Tchoffo, E | 1 |
| Tasson Tatang, C | 1 |
| Ide Zeuna, J | 1 |
| Noupoue, EM | 1 |
| Tsoplifack, CB | 1 |
| Folefack, GO | 1 |
| Kettani, M | 1 |
| Bandelier, P | 1 |
| Huo, J | 1 |
| Li, H | 4 |
| Yu, D | 1 |
| Arulsamy, N | 1 |
| AlAbbad, S | 1 |
| Sardot, T | 1 |
| Lekashvili, O | 1 |
| Decato, D | 1 |
| Lelj, F | 1 |
| Alexander Ross, JB | 1 |
| Rosenberg, E | 1 |
| Nazir, H | 1 |
| Muthuswamy, N | 1 |
| Louis, C | 1 |
| Jose, S | 1 |
| Prakash, J | 1 |
| Buan, MEM | 1 |
| Flox, C | 1 |
| Chavan, S | 1 |
| Shi, X | 1 |
| Kauranen, P | 1 |
| Kallio, T | 1 |
| Maia, G | 1 |
| Tammeveski, K | 1 |
| Lymperopoulos, N | 1 |
| Carcadea, E | 1 |
| Veziroglu, E | 1 |
| Iranzo, A | 1 |
| M Kannan, A | 1 |
| Arunamata, A | 1 |
| Tacy, TA | 1 |
| Kache, S | 1 |
| Mainwaring, RD | 1 |
| Ma, M | 1 |
| Maeda, K | 1 |
| Punn, R | 1 |
| Noguchi, S | 1 |
| Hahn, S | 3 |
| Iwasa, Y | 3 |
| Ling, J | 2 |
| Voccio, JP | 2 |
| Kim, Y | 3 |
| Song, J | 3 |
| Bascuñán, J | 2 |
| Chu, Y | 1 |
| Tomita, M | 1 |
| Cazorla, M | 1 |
| Herrera, E | 1 |
| Palomeque, E | 1 |
| Saud, N | 1 |
| Hoplock, LB | 1 |
| Lobchuk, MM | 1 |
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| Henson, MA | 1 |
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| Swaroop, S | 1 |
| Nagornov, KO | 1 |
| Kozhinov, AN | 1 |
| Tsybin, YO | 1 |
| Kuang, S | 1 |
| Laskin, J | 1 |
| Zin, NNINM | 1 |
| Mohamad, MN | 1 |
| Roslan, K | 1 |
| Abdul Wafi, S | 1 |
| Abdul Moin, NI | 1 |
| Alias, A | 1 |
| Zakaria, Y | 1 |
| Abu-Bakar, N | 1 |
| Naveed, A | 1 |
| Jilani, K | 1 |
| Siddique, AB | 1 |
| Akbar, M | 1 |
| Riaz, M | 1 |
| Mushtaq, Z | 1 |
| Sikandar, M | 1 |
| Ilyas, S | 1 |
| Bibi, I | 1 |
| Asghar, A | 1 |
| Rasool, G | 1 |
| Irfan, M | 1 |
| Li, XY | 1 |
| Zhao, S | 2 |
| Fan, XH | 1 |
| Chen, KP | 1 |
| Hua, W | 1 |
| Liu, ZM | 1 |
| Xue, XD | 1 |
| Zhou, B | 1 |
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| Xing, YL | 1 |
| Chen, MA | 1 |
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| Wu, XT | 1 |
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| Huang, W | 1 |
| Cui, Y | 1 |
| Yang, QQ | 1 |
| Li, HW | 1 |
| Zhao, XQ | 1 |
| Hossein Rashidi, B | 1 |
| Tarafdari, A | 1 |
| Ghazimirsaeed, ST | 1 |
| Shahrokh Tehraninezhad, E | 1 |
| Keikha, F | 1 |
| Eslami, B | 1 |
| Ghazimirsaeed, SM | 1 |
| Jafarabadi, M | 1 |
| Silvani, Y | 1 |
| Lovita, AND | 1 |
| Maharani, A | 1 |
| Wiyasa, IWA | 1 |
| Sujuti, H | 1 |
| Ratnawati, R | 1 |
| Raras, TYM | 1 |
| Lemin, AS | 1 |
| Rahman, MM | 1 |
| Pangarah, CA | 1 |
| Kiyu, A | 1 |
| Zeng, C | 2 |
| Du, H | 1 |
| Lin, D | 1 |
| Jalan, D | 1 |
| Rubagumya, F | 1 |
| Hopman, WM | 1 |
| Vanderpuye, V | 1 |
| Lopes, G | 1 |
| Seruga, B | 1 |
| Booth, CM | 1 |
| Berry, S | 1 |
| Hammad, N | 1 |
| Sajo, EA | 1 |
| Okunade, KS | 1 |
| Olorunfemi, G | 1 |
| Rabiu, KA | 1 |
| Anorlu, RI | 1 |
| Xu, C | 2 |
| Xiang, Y | 1 |
| Xu, X | 1 |
| Zhou, L | 3 |
| Dong, X | 1 |
| Tang, S | 1 |
| Gao, XC | 1 |
| Wei, CH | 1 |
| Zhang, RG | 1 |
| Cai, Q | 1 |
| He, Y | 1 |
| Tong, F | 1 |
| Dong, JH | 1 |
| Wu, G | 2 |
| Dong, XR | 1 |
| Tang, X | 1 |
| Tao, F | 1 |
| Xiang, W | 1 |
| Zhao, Y | 2 |
| Jin, L | 1 |
| Tao, H | 1 |
| Lei, Y | 1 |
| Gan, H | 1 |
| Huang, Y | 1 |
| Chen, Y | 3 |
| Chen, L | 3 |
| Shan, A | 1 |
| Zhao, H | 2 |
| Wu, M | 3 |
| Ma, Q | 2 |
| Wang, J | 6 |
| Zhang, E | 1 |
| Li, Y | 5 |
| Xue, F | 1 |
| Deng, L | 1 |
| Liu, L | 2 |
| Yan, Z | 2 |
| Wang, Y | 3 |
| Meng, J | 1 |
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| Anastassiadou, M | 1 |
| Bernasconi, G | 1 |
| Brancato, A | 1 |
| Carrasco Cabrera, L | 1 |
| Greco, L | 1 |
| Jarrah, S | 1 |
| Kazocina, A | 1 |
| Leuschner, R | 1 |
| Magrans, JO | 1 |
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| Nave, S | 1 |
| Pedersen, R | 1 |
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| Rojas, A | 1 |
| Sacchi, A | 1 |
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| Theobald, A | 1 |
| Vagenende, B | 1 |
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| Zhuang, T | 1 |
| Tu, J | 1 |
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| Yao, H | 1 |
| Lee, DF | 1 |
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| Wen, L | 1 |
| Huang, G | 2 |
| Xie, X | 1 |
| Zhao, Q | 1 |
| Hu, W | 1 |
| Zhang, Y | 5 |
| Wu, X | 1 |
| Lu, J | 2 |
| Li, M | 1 |
| Li, W | 3 |
| Wu, W | 1 |
| Du, F | 1 |
| Ji, H | 1 |
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| Liao, J | 1 |
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| Na, KJ | 1 |
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| Chung, DH | 1 |
| Paeng, JC | 1 |
| Park, IK | 1 |
| Kang, CH | 1 |
| Cheon, GJ | 1 |
| Kang, KW | 1 |
| Lee, DS | 1 |
| Kim, YT | 1 |
| Pajuelo-Lozano, N | 1 |
| Alcalá, S | 1 |
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| Perona, R | 1 |
| Sanchez-Perez, I | 1 |
| Logotheti, S | 1 |
| Marquardt, S | 1 |
| Gupta, SK | 1 |
| Richter, C | 1 |
| Edelhäuser, BAH | 1 |
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| Brenmoehl, J | 1 |
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| Alpers, M | 1 |
| Singh, KP | 1 |
| Wolkenhauer, O | 1 |
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4 reviews available for temozolomide and Carcinogenesis
| Article | Year |
|---|---|
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Oncogenesis, Microenvironment Modulation and Clinical Potentiality of FAP in Glioblastoma: Lessons Learned from Other Solid Tumors.
Topics: Animals; Biomarkers; Biomarkers, Tumor; Brain Neoplasms; Cancer-Associated Fibroblasts; Carcinogenes | 2021 |
Molecular mechanism of adenomatous polyposis coli-induced blockade of base excision repair pathway in colorectal carcinogenesis.
Topics: Adenomatous Polyposis Coli; Adenomatous Polyposis Coli Protein; Animals; Antineoplastic Agents; Carc | 2015 |
O
Topics: Antineoplastic Agents, Alkylating; Brain Neoplasms; Carcinogenesis; Carcinoma, Non-Small-Cell Lung; | 2017 |
1 trial available for temozolomide and Carcinogenesis
| Article | Year |
|---|---|
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
Psychological distress among health care professionals of the three COVID-19 most affected Regions in Cameroon: Prevalence and associated factors.
Topics: 3' Untranslated Regions; 5'-Nucleotidase; A549 Cells; Accidental Falls; Acetylcholinesterase; Acryli | 2021 |
30 other studies available for temozolomide and Carcinogenesis
| Article | Year |
|---|---|
HSP90-CDC37 functions as a chaperone for the oncogenic FGFR3-TACC3 fusion.
Topics: Carcinogenesis; Cell Cycle Proteins; Cell Line, Tumor; Chaperonins; Glioblastoma; Glioma; HSP90 Heat | 2022 |
Oncogenic Forkhead box D3 antisense RNA 1 promotes cell survival and confers temozolomide resistance in glioblastoma cells through the miR-128-3p/WEE1 G2 checkpoint kinase axis.
Topics: Apoptosis; Carcinogenesis; Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; Cell Survival; | 2022 |
The Different Temozolomide Effects on Tumorigenesis Mechanisms of Pediatric Glioblastoma PBT24 and SF8628 Cell Tumor in CAM Model and on Cells In Vitro.
Topics: Animals; Antineoplastic Agents, Alkylating; Apoptosis; Brain Neoplasms; Carcinogenesis; Cell Line, T | 2022 |
MGMT in glial carcinogenesis. Roles from prevention to treatment.
Topics: Brain Neoplasms; Carcinogenesis; Carcinogens; Dacarbazine; DNA; DNA Methylation; DNA Modification Me | 2022 |
A rationally identified panel of microRNAs targets multiple oncogenic pathways to enhance chemotherapeutic effects in glioblastoma models.
Topics: Animals; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Drug Resistance, Neoplasm; Gene Expressi | 2022 |
Exploring the Functional Roles of Telomere Maintenance 2 in the Tumorigenesis of Glioblastoma Multiforme and Drug Responsiveness to Temozolomide.
Topics: Adult; Antineoplastic Agents, Alkylating; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cell Tr | 2023 |
11PS04 is a new chemical entity identified by microRNA-based biosensing with promising therapeutic potential against cancer stem cells.
Topics: Animals; Antineoplastic Agents; Biosensing Techniques; Carcinogenesis; Cell Line, Tumor; Cell Prolif | 2019 |
Role of Sonic hedgehog signaling in cell cycle, oxidative stress, and autophagy of temozolomide resistant glioblastoma.
Topics: Animals; Antineoplastic Agents, Alkylating; Autophagy; Beclin-1; Carcinogenesis; Cell Cycle; Cell Li | 2020 |
miR-152-5p suppresses glioma progression and tumorigenesis and potentiates temozolomide sensitivity by targeting FBXL7.
Topics: Animals; Apoptosis; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Disease Progression; Drug | 2020 |
MiR-3116 sensitizes glioma cells to temozolomide by targeting FGFR1 and regulating the FGFR1/PI3K/AKT pathway.
Topics: Animals; Apoptosis; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Dacarbazine; Drug Resistan | 2020 |
Suppressing Dazl modulates tumorigenicity and stemness in human glioblastoma cells.
Topics: Animals; Antineoplastic Agents; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cell Movement; Ce | 2020 |
Phospholipase D1 inhibition sensitizes glioblastoma to temozolomide and suppresses its tumorigenicity.
Topics: Animals; Antineoplastic Agents, Alkylating; Biomarkers, Tumor; Brain Neoplasms; Carcinogenesis; Cell | 2020 |
Activation of dopamine receptor D1 inhibits glioblastoma tumorigenicity by regulating autophagic activity.
Topics: Animals; Autophagy; Calcium; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Glioblastoma; Hum | 2020 |
EIF4A3-induced circular RNA ASAP1 promotes tumorigenesis and temozolomide resistance of glioblastoma via NRAS/MEK1/ERK1-2 signaling.
Topics: Adaptor Proteins, Signal Transducing; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cell Prolif | 2021 |
RPN2 is targeted by miR-181c and mediates glioma progression and temozolomide sensitivity via the wnt/β-catenin signaling pathway.
Topics: Animals; Antineoplastic Agents, Alkylating; Apoptosis; beta Catenin; Carcinogenesis; Cell Line, Tumo | 2020 |
The novel roles of virus infection-associated gene CDKN1A in chemoresistance and immune infiltration of glioblastoma.
Topics: Antineoplastic Agents, Alkylating; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cyclin-Depende | 2021 |
Effects of the Anti-Tumorigenic Agent AT101 on Human Glioblastoma Cells in the Microenvironmental Glioma Stem Cell Niche.
Topics: Antineoplastic Combined Chemotherapy Protocols; Brain; Brain Neoplasms; Carcinogenesis; Cell Line, T | 2021 |
Targeting PTPRZ inhibits stem cell-like properties and tumorigenicity in glioblastoma cells.
Topics: Animals; Antineoplastic Agents, Alkylating; Carcinogenesis; Enzyme Inhibitors; Female; Glioblastoma; | 2017 |
Hyperthermia with different temperatures inhibits proliferation and promotes apoptosis through the EGFR/STAT3 pathway in C6 rat glioma cells.
Topics: Animals; Apoptosis; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Dacarbazi | 2017 |
Combination with TMZ and miR-505 inhibits the development of glioblastoma by regulating the WNT7B/Wnt/β-catenin signaling pathway.
Topics: Animals; Antineoplastic Agents, Alkylating; Base Sequence; beta Catenin; Binding Sites; Brain Neopla | 2018 |
APOBEC3B is expressed in human glioma, and influences cell proliferation and temozolomide resistance.
Topics: Biomarkers, Tumor; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Cytidine Deaminase; Drug Re | 2018 |
MicroRNA-34a-5p suppresses tumorigenesis and progression of glioma and potentiates Temozolomide-induced cytotoxicity for glioma cells by targeting HMGA2.
Topics: Animals; Base Sequence; Carcinogenesis; Cell Line, Tumor; Cell Movement; Cell Proliferation; Disease | 2019 |
Interleukin-8/CXCR2 signaling regulates therapy-induced plasticity and enhances tumorigenicity in glioblastoma.
Topics: Animals; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cell Plasticity; Drug Resistance, Neopla | 2019 |
Impact of extremely low-frequency electromagnetic field (100 Hz, 100 G) exposure on human glioblastoma U87 cells during Temozolomide administration.
Topics: Antineoplastic Agents; Apoptosis; Calcium; Carcinogenesis; Cell Differentiation; Cell Line, Tumor; C | 2019 |
Glioma initiating cells form a differentiation niche via the induction of extracellular matrices and integrin αV.
Topics: Animals; Brain Neoplasms; Carcinogenesis; Cell Adhesion; Cell Differentiation; Cell Movement; Cell S | 2013 |
Molecular targeting of TRF2 suppresses the growth and tumorigenesis of glioblastoma stem cells.
Topics: Animals; Antineoplastic Agents, Alkylating; Brain Neoplasms; Carcinogenesis; Cell Differentiation; C | 2014 |
A complex mechanism for HDGF-mediated cell growth, migration, invasion, and TMZ chemosensitivity in glioma.
Topics: Adolescent; Adult; Aged; Antineoplastic Agents, Alkylating; Apoptosis; Brain Neoplasms; Cadherins; C | 2014 |
Suppressing H19 Modulates Tumorigenicity and Stemness in U251 and U87MG Glioma Cells.
Topics: Antineoplastic Agents, Alkylating; Apoptosis; Biomarkers, Tumor; Carcinogenesis; Cell Line, Tumor; C | 2016 |
Chemical Screening Identifies EUrd as a Novel Inhibitor Against Temozolomide-Resistant Glioblastoma-Initiating Cells.
Topics: 5'-Nucleotidase; Animals; Brain Neoplasms; Carcinogenesis; Cell Cycle Checkpoints; Cell Death; Cell | 2016 |
β-Elemene Selectively Inhibits the Proliferation of Glioma Stem-Like Cells Through the Downregulation of Notch1.
Topics: Animals; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Disease Models, Animal; Down-Regulati | 2017 |