technetium-tc-99m-bicisate and Apraxias

technetium-tc-99m-bicisate has been researched along with Apraxias* in 2 studies

Other Studies

2 other study(ies) available for technetium-tc-99m-bicisate and Apraxias

Article	Year
Progressive apraxic agraphia with micrographia presenting as corticobasal syndrome showing extensive Pittsburgh compound B uptake. Journal of neurology, 2013, Volume: 260, Issue:8 A 65-year-old woman developed progressive apraxic agraphia, characterized by poorly formed graphemes, a kanji (Japanese morphograms) recall impairment, relatively preserved oral spelling of kanji characters, and incorrect stroke sequences on writing accompanied by micrographia over a 3-year period. She also showed minor degrees of rigidity, limb-kinetic apraxia, and ideomotor apraxia of the left hand. Although asymmetric rigidity and limb-kinetic apraxia strongly suggested corticobasal degeneration, (11)C-Pittsburgh compound B positron emission tomography (PiB-PET) showed the predominantly right-sided accumulation of amyloid β in the cortices and striatum. (18)F-fluoro-deoxy-glucose PET and single photon emission computed tomography with a (99m)Tc-ethylcysteinate dimer (ECD-SPECT) also revealed predominantly right-sided hypometabolism and hypoperfusion in the primary sensorimotor cortex, posterior cingulate gyrus, temporoparietal cortices, frontal cortices, thalamus, and basal ganglia, a pattern characteristic of both corticobasal degeneration and Alzheimer's disease. The findings suggest that progressive apraxic agraphia with micrographia presenting as corticobasal syndrome can show an Alzheimer's disease pathology. It is also suggested that ideomotor apraxia of the left hand can occur without a callosal lesion, and is caused by hypometabolism or hypoperfusion in the right frontal and parietal cortices, as revealed by PET and SPECT. Topics: Aged; Agraphia; Aniline Compounds; Apolipoproteins E; Apraxias; Basal Ganglia Diseases; Brain; Cerebral Cortex; Cysteine; Disease Progression; Executive Function; Female; Handwriting; Humans; Image Processing, Computer-Assisted; Intelligence Tests; Neurodegenerative Diseases; Neurologic Examination; Neuropsychological Tests; Organotechnetium Compounds; Positron-Emission Tomography; Radiopharmaceuticals; Reading; Thiazoles; Tomography, Emission-Computed, Single-Photon	2013
Abnormal cerebral blood flow findings in transplant patients with posttransplant apraxia of speech. Transplantation proceedings, 2001, Volume: 33, Issue:4 Topics: Apraxias; Basal Ganglia; Brain; Brain Ischemia; Cerebral Cortex; Cerebrovascular Circulation; Cysteine; Female; Glial Fibrillary Acidic Protein; Heart Transplantation; Humans; Liver Transplantation; Magnetic Resonance Imaging; Male; Middle Aged; Organotechnetium Compounds; Postoperative Complications; Radiopharmaceuticals; Speech; Thalamus; Tomography, Emission-Computed, Single-Photon	2001

Article

Year

Progressive apraxic agraphia with micrographia presenting as corticobasal syndrome showing extensive Pittsburgh compound B uptake.

Journal of neurology, 2013, Volume: 260, Issue:8

A 65-year-old woman developed progressive apraxic agraphia, characterized by poorly formed graphemes, a kanji (Japanese morphograms) recall impairment, relatively preserved oral spelling of kanji characters, and incorrect stroke sequences on writing accompanied by micrographia over a 3-year period. She also showed minor degrees of rigidity, limb-kinetic apraxia, and ideomotor apraxia of the left hand. Although asymmetric rigidity and limb-kinetic apraxia strongly suggested corticobasal degeneration, (11)C-Pittsburgh compound B positron emission tomography (PiB-PET) showed the predominantly right-sided accumulation of amyloid β in the cortices and striatum. (18)F-fluoro-deoxy-glucose PET and single photon emission computed tomography with a (99m)Tc-ethylcysteinate dimer (ECD-SPECT) also revealed predominantly right-sided hypometabolism and hypoperfusion in the primary sensorimotor cortex, posterior cingulate gyrus, temporoparietal cortices, frontal cortices, thalamus, and basal ganglia, a pattern characteristic of both corticobasal degeneration and Alzheimer's disease. The findings suggest that progressive apraxic agraphia with micrographia presenting as corticobasal syndrome can show an Alzheimer's disease pathology. It is also suggested that ideomotor apraxia of the left hand can occur without a callosal lesion, and is caused by hypometabolism or hypoperfusion in the right frontal and parietal cortices, as revealed by PET and SPECT.

Topics: Aged; Agraphia; Aniline Compounds; Apolipoproteins E; Apraxias; Basal Ganglia Diseases; Brain; Cerebral Cortex; Cysteine; Disease Progression; Executive Function; Female; Handwriting; Humans; Image Processing, Computer-Assisted; Intelligence Tests; Neurodegenerative Diseases; Neurologic Examination; Neuropsychological Tests; Organotechnetium Compounds; Positron-Emission Tomography; Radiopharmaceuticals; Reading; Thiazoles; Tomography, Emission-Computed, Single-Photon

2013

Abnormal cerebral blood flow findings in transplant patients with posttransplant apraxia of speech.

Transplantation proceedings, 2001, Volume: 33, Issue:4

Topics: Apraxias; Basal Ganglia; Brain; Brain Ischemia; Cerebral Cortex; Cerebrovascular Circulation; Cysteine; Female; Glial Fibrillary Acidic Protein; Heart Transplantation; Humans; Liver Transplantation; Magnetic Resonance Imaging; Male; Middle Aged; Organotechnetium Compounds; Postoperative Complications; Radiopharmaceuticals; Speech; Thalamus; Tomography, Emission-Computed, Single-Photon

2001