taxifolin has been researched along with Breast Neoplasms in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (25.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Berti, F; Colombo, G; Grazioso, G; Magistrato, A; Martini, S; Pavlin, M; Pennati, M; Sgrignani, J; Spinello, A; Zaffaroni, N | 1 |
Chittiboyina, AG; Cleary, J; Deshpande, R; Liu, Y; Miller, LD; Mo, YY; Sharma, S; Tyagi, A; Wang, Y; Watabe, K; Wu, K; Wu, SY; Xing, F; Yalamanchili, C; Zhao, D | 1 |
Gong, X; Hu, L; Jiang, R; Kuang, G; Li, H; Li, J; Wan, J; Zhou, T | 1 |
Bast, A; Boven, E; Grimbergen, JA; Kramer, K; Kuiper, K; van Acker, SA; van den Berg, DJ; van der Vijgh, WJ | 1 |
4 other study(ies) available for taxifolin and Breast Neoplasms
Article | Year |
---|---|
Rational design of allosteric modulators of the aromatase enzyme: An unprecedented therapeutic strategy to fight breast cancer.
Topics: Allosteric Regulation; Antineoplastic Agents; Aromatase; Breast Neoplasms; Cell Line, Tumor; Cell Proliferation; Dose-Response Relationship, Drug; Drug Design; Drug Screening Assays, Antitumor; Enzyme Inhibitors; Humans; Molecular Dynamics Simulation; Molecular Structure; Structure-Activity Relationship | 2019 |
Epigenetic and Posttranscriptional Modulation of SOS1 Can Promote Breast Cancer Metastasis through Obesity-Activated c-Met Signaling in African-American Women.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Apoptosis; Biomarkers, Tumor; Black or African American; Breast Neoplasms; Cell Proliferation; Epigenesis, Genetic; Female; Gene Expression Regulation, Neoplastic; GRB2 Adaptor Protein; Humans; Lung Neoplasms; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Nude; Obesity; Prognosis; Proto-Oncogene Proteins c-met; Quercetin; SOS1 Protein; Tumor Cells, Cultured; Xenograft Model Antitumor Assays | 2021 |
Taxifolin inhibits breast cancer cells proliferation, migration and invasion by promoting mesenchymal to epithelial transition via β-catenin signaling.
Topics: Animals; beta Catenin; Breast Neoplasms; Cell Line, Tumor; Cell Movement; Cell Proliferation; Epithelial-Mesenchymal Transition; Female; Humans; MCF-7 Cells; Mice; Mice, Inbred BALB C; Neoplasm Invasiveness; Quercetin; Signal Transduction; Xenograft Model Antitumor Assays | 2019 |
Monohydroxyethylrutoside, a dose-dependent cardioprotective agent, does not affect the antitumor activity of doxorubicin.
Topics: Animals; Antibiotics, Antineoplastic; Antioxidants; Breast Neoplasms; Cardiomyopathies; Catechin; Chelation Therapy; Cystadenocarcinoma, Serous; Dose-Response Relationship, Drug; Doxorubicin; Drug Interactions; Electrocardiography; Female; Flavonoids; Flavonols; Free Radical Scavengers; Free Radicals; Humans; Hydroxyethylrutoside; Iron; Iron Chelating Agents; Kaempferols; Mice; Mice, Nude; Molecular Structure; Neoplasm Transplantation; Ovarian Neoplasms; Quercetin; Razoxane; Rutin; Telemetry; Tumor Cells, Cultured; Weight Loss | 1997 |