taurocholic acid has been researched along with Cancer of Esophagus in 5 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (40.00) | 29.6817 |
| 2010's | 3 (60.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Hylemon, PB; Li, X; Liu, R; Zhou, H | 1 |
| Kanai, S; Mukaisho, KI; Sugihara, H; Taniura, N; Yoshida, S | 1 |
| Hattori, T; Mukaisho, K; Saito, S; Sato, S; Sugihara, H; Yamamoto, G; Yamamoto, H | 1 |
| Geng, J; Gong, J; Song, Y; Wu, J | 1 |
| Hémon, B; Leteurtre, E; Mariette, C; Piessen, G; Triboulet, JP; Van Seuningen, I | 1 |
5 other study(ies) available for taurocholic acid and Cancer of Esophagus
| Article | Year |
|---|---|
Conjugated Bile Acids Promote Invasive Growth of Esophageal Adenocarcinoma Cells and Cancer Stem Cell Expansion via Sphingosine 1-Phosphate Receptor 2-Mediated Yes-Associated Protein Activation.
Topics: Adaptor Proteins, Signal Transducing; Adenocarcinoma; Cholagogues and Choleretics; Esophageal Neoplasms; Gene Expression Regulation, Neoplastic; Humans; Neoplasm Invasiveness; Neoplastic Stem Cells; Phosphoproteins; Receptors, Lysosphingolipid; Sphingosine-1-Phosphate Receptors; Taurocholic Acid; Transcription Factors; Tumor Cells, Cultured; YAP-Signaling Proteins | 2018 |
Host factors influence Barrett's carcinogenesis: findings from a mouse gastroduodenal reflux model.
Topics: Animals; Barrett Esophagus; Bile Acids and Salts; Carcinogenesis; Cell Proliferation; Diet, High-Fat; Disease Models, Animal; Esophageal Neoplasms; Esophagogastric Junction; Esophagostomy; Esophagus; Gastroesophageal Reflux; Glucuronosyltransferase; Humans; Jejunostomy; Male; Mice; Mice, Inbred C57BL; Minor Histocompatibility Antigens; Rats; Taurocholic Acid | 2019 |
Continuous taurocholic acid exposure promotes esophageal squamous cell carcinoma progression due to reduced cell loss resulting from enhanced vascular development.
Topics: Animals; Carcinoma, Squamous Cell; Cell Cycle; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cell Shape; Disease Progression; Esophageal Neoplasms; Esophageal Squamous Cell Carcinoma; Flow Cytometry; Gene Expression Regulation, Neoplastic; Human Umbilical Vein Endothelial Cells; Humans; Mice; Mice, Nude; Neoplasm Invasiveness; Neovascularization, Pathologic; Rats; RNA, Messenger; Taurocholic Acid; Transforming Growth Factor beta1; Vascular Endothelial Growth Factor A; Xenograft Model Antitumor Assays | 2014 |
Deoxycholic acid induces the overexpression of intestinal mucin, MUC2, via NF-kB signaling pathway in human esophageal adenocarcinoma cells.
Topics: Adenocarcinoma; Analysis of Variance; Aspirin; Cell Line, Tumor; Chenodeoxycholic Acid; Cholagogues and Choleretics; Cyclic AMP-Dependent Protein Kinases; Deoxycholic Acid; Esophageal Neoplasms; Gastrointestinal Agents; Gene Expression Regulation, Neoplastic; Humans; Mucin-2; Naphthalenes; NF-kappa B; Protein Kinase C; RNA, Small Interfering; Signal Transduction; Taurocholic Acid | 2008 |
Activation of MUC1 mucin expression by bile acids in human esophageal adenocarcinomatous cells and tissues is mediated by the phosphatidylinositol 3-kinase.
Topics: Adenocarcinoma; Bile Acids and Salts; Cell Line, Tumor; Deoxycholic Acid; Esophageal Neoplasms; Esophagus; Humans; Immunohistochemistry; In Vitro Techniques; Mucin 5AC; Mucin-1; Mucins; Mucous Membrane; Phosphatidylinositol 3-Kinases; Promoter Regions, Genetic; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Signal Transduction; Taurocholic Acid; Transfection; Up-Regulation | 2008 |