sulforaphane has been researched along with Cerebral Infarction, Middle Cerebral Artery in 3 studies
sulforaphane: from Cardaria draba L.
sulforaphane : An isothiocyanate having a 4-(methylsulfinyl)butyl group attached to the nitrogen.
Excerpt | Relevance | Reference |
---|---|---|
"As sulforaphane pretreatment led to a moderate increase in peroxynitrite generation, we suggest that hormetic preconditioning underlies sulforaphane-mediated protection against stroke." | 1.39 | Sulforaphane preconditioning of the Nrf2/HO-1 defense pathway protects the cerebral vasculature against blood-brain barrier disruption and neurological deficits in stroke. ( Alfieri, A; Cash, D; Duchen, MR; Fraser, PA; Mann, GE; Modo, M; Siow, RCM; Srivastava, S; Williams, SCR, 2013) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 1 (33.33) | 2.80 |
Authors | Studies |
---|---|
Li, Q | 1 |
Fadoul, G | 1 |
Ikonomovic, M | 1 |
Yang, T | 1 |
Zhang, F | 1 |
Srivastava, S | 2 |
Alfieri, A | 2 |
Siow, RC | 1 |
Mann, GE | 2 |
Fraser, PA | 2 |
Siow, RCM | 1 |
Cash, D | 1 |
Modo, M | 1 |
Duchen, MR | 1 |
Williams, SCR | 1 |
3 other studies available for sulforaphane and Cerebral Infarction, Middle Cerebral Artery
Article | Year |
---|---|
Sulforaphane promotes white matter plasticity and improves long-term neurological outcomes after ischemic stroke via the Nrf2 pathway.
Topics: Animals; Brain Injuries; Female; Infarction, Middle Cerebral Artery; Ischemic Stroke; Isothiocyanate | 2022 |
Temporal and spatial distribution of Nrf2 in rat brain following stroke: quantification of nuclear to cytoplasmic Nrf2 content using a novel immunohistochemical technique.
Topics: Animals; Brain; Cell Line; Cell Nucleus; Cytoplasm; Infarction, Middle Cerebral Artery; Isothiocyana | 2013 |
Sulforaphane preconditioning of the Nrf2/HO-1 defense pathway protects the cerebral vasculature against blood-brain barrier disruption and neurological deficits in stroke.
Topics: Animals; Blood-Brain Barrier; Cerebral Arteries; Gene Expression; Heme Oxygenase-1; Infarction, Midd | 2013 |