sulforaphane has been researched along with Cardiomyopathies, Primary in 4 studies
sulforaphane: from Cardaria draba L.
sulforaphane : An isothiocyanate having a 4-(methylsulfinyl)butyl group attached to the nitrogen.
Excerpt | Relevance | Reference |
---|---|---|
"We reported previously that nuclear factor erythroid 2-related factor 2 (Nrf2) and metallothionein (MT) play critical roles in preventing intermittent hypoxia (IH)-induced cardiomyopathy." | 7.88 | Nrf2 expression and function, but not MT expression, is indispensable for sulforaphane-mediated protection against intermittent hypoxia-induced cardiomyopathy in mice. ( Cai, J; Cai, L; Cui, T; Liu, Q; Wang, J; Xin, Y; Yin, X; Zhang, Z; Zheng, Y; Zhou, S, 2018) |
"As sepsis is associated with a 50% increase in mortality, sepsis-induced cardiomyopathy has become a critical topic." | 5.91 | Effect of Sulforaphane on cardiac injury induced by sepsis in a mouse model: Role of toll-like receptor 4. ( Altoraihi, K; Ghafil, FA; Hadi, NR; Hadi, SMH; Majeed, S, 2023) |
"We reported previously that nuclear factor erythroid 2-related factor 2 (Nrf2) and metallothionein (MT) play critical roles in preventing intermittent hypoxia (IH)-induced cardiomyopathy." | 3.88 | Nrf2 expression and function, but not MT expression, is indispensable for sulforaphane-mediated protection against intermittent hypoxia-induced cardiomyopathy in mice. ( Cai, J; Cai, L; Cui, T; Liu, Q; Wang, J; Xin, Y; Yin, X; Zhang, Z; Zheng, Y; Zhou, S, 2018) |
"As sepsis is associated with a 50% increase in mortality, sepsis-induced cardiomyopathy has become a critical topic." | 1.91 | Effect of Sulforaphane on cardiac injury induced by sepsis in a mouse model: Role of toll-like receptor 4. ( Altoraihi, K; Ghafil, FA; Hadi, NR; Hadi, SMH; Majeed, S, 2023) |
" Previous studies have shown that oxidative stress caused by DOX is one of the primary mechanisms for its toxic effects on the heart." | 1.42 | Sulforaphane protects the heart from doxorubicin-induced toxicity. ( Allen, CD; Beneš, H; McElhanon, K; Megyesi, JK; Sharma, R; Singh, P; Singh, SP, 2015) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 2 (50.00) | 2.80 |
Authors | Studies |
---|---|
Hadi, SMH | 1 |
Majeed, S | 1 |
Ghafil, FA | 1 |
Altoraihi, K | 1 |
Hadi, NR | 1 |
Su, X | 1 |
Wang, S | 1 |
Zhang, H | 1 |
Yang, G | 1 |
Bai, Y | 1 |
Liu, P | 1 |
Meng, L | 1 |
Jiang, X | 1 |
Xin, Y | 2 |
Zhou, S | 1 |
Wang, J | 1 |
Yin, X | 1 |
Zhang, Z | 1 |
Cui, T | 1 |
Cai, J | 1 |
Zheng, Y | 1 |
Liu, Q | 1 |
Cai, L | 1 |
Singh, P | 1 |
Sharma, R | 1 |
McElhanon, K | 1 |
Allen, CD | 1 |
Megyesi, JK | 1 |
Beneš, H | 1 |
Singh, SP | 1 |
4 other studies available for sulforaphane and Cardiomyopathies, Primary
Article | Year |
---|---|
Effect of Sulforaphane on cardiac injury induced by sepsis in a mouse model: Role of toll-like receptor 4.
Topics: Animals; Cardiomyopathies; Heart Injuries; Male; Mice; Sepsis; Toll-Like Receptor 4 | 2023 |
Sulforaphane prevents angiotensin II-induced cardiomyopathy by activation of Nrf2 through epigenetic modification.
Topics: Angiotensin II; Animals; Cardiomyopathies; Epigenesis, Genetic; Gene Expression Regulation; Isothioc | 2021 |
Nrf2 expression and function, but not MT expression, is indispensable for sulforaphane-mediated protection against intermittent hypoxia-induced cardiomyopathy in mice.
Topics: Animals; Cardiomyopathies; Cardiotonic Agents; Gene Expression Regulation; Hypoxia; Isothiocyanates; | 2018 |
Sulforaphane protects the heart from doxorubicin-induced toxicity.
Topics: Animals; Antibiotics, Antineoplastic; Cardiomyopathies; Cardiotonic Agents; Caspase 3; Cell Line; Do | 2015 |