sulforaphane has been researched along with Blood Poisoning in 5 studies
sulforaphane: from Cardaria draba L.
sulforaphane : An isothiocyanate having a 4-(methylsulfinyl)butyl group attached to the nitrogen.
| Excerpt | Relevance | Reference |
|---|---|---|
| "To explore the effects and molecular mechanisms of sulforaphane (SFN) on sepsis-associated inflammation and sarcopenia." | 8.31 | Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways. ( Gan, L; Liu, M; Teng, Z; Wang, M; Wu, X; Xu, W; Zhang, Y, 2023) |
| "As sepsis is associated with a 50% increase in mortality, sepsis-induced cardiomyopathy has become a critical topic." | 5.91 | Effect of Sulforaphane on cardiac injury induced by sepsis in a mouse model: Role of toll-like receptor 4. ( Altoraihi, K; Ghafil, FA; Hadi, NR; Hadi, SMH; Majeed, S, 2023) |
| "To explore the effects and molecular mechanisms of sulforaphane (SFN) on sepsis-associated inflammation and sarcopenia." | 4.31 | Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways. ( Gan, L; Liu, M; Teng, Z; Wang, M; Wu, X; Xu, W; Zhang, Y, 2023) |
| "As sepsis is associated with a 50% increase in mortality, sepsis-induced cardiomyopathy has become a critical topic." | 1.91 | Effect of Sulforaphane on cardiac injury induced by sepsis in a mouse model: Role of toll-like receptor 4. ( Altoraihi, K; Ghafil, FA; Hadi, NR; Hadi, SMH; Majeed, S, 2023) |
| "Sepsis is often characterized by an acute brain inflammation and dysfunction, which is associated with increased morbidity and mortality worldwide." | 1.43 | Sulforaphane induces neurovascular protection against a systemic inflammatory challenge via both Nrf2-dependent and independent pathways. ( Alexander, JS; Becker, F; Evans, PC; Gavins, FNE; Gillespie, S; Holloway, PM; Nguyen, V; Vital, SA, 2016) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (60.00) | 24.3611 |
| 2020's | 2 (40.00) | 2.80 |
| Authors | Studies |
|---|---|
| Wang, M | 3 |
| Liu, M | 3 |
| Xu, W | 3 |
| Teng, Z | 3 |
| Wu, X | 3 |
| Gan, L | 3 |
| Zhang, Y | 3 |
| Hadi, SMH | 1 |
| Majeed, S | 1 |
| Ghafil, FA | 1 |
| Altoraihi, K | 1 |
| Hadi, NR | 1 |
| Lee, IC | 1 |
| Kim, DY | 1 |
| Bae, JS | 2 |
| Ku, SK | 1 |
| Han, MS | 1 |
| Holloway, PM | 1 |
| Gillespie, S | 1 |
| Becker, F | 1 |
| Vital, SA | 1 |
| Nguyen, V | 1 |
| Alexander, JS | 1 |
| Evans, PC | 1 |
| Gavins, FNE | 1 |
5 other studies available for sulforaphane and Blood Poisoning
| Article | Year |
|---|---|
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Sulforaphane reduces lipopolysaccharide-induced inflammation and enhances myogenic differentiation of mouse embryonic myoblasts via the toll-like receptor 4 and NLRP3 pathways.
Topics: Animals; Cell Differentiation; Inflammasomes; Inflammation; Lipopolysaccharides; Mice; Myoblasts; NL | 2023 |
Effect of Sulforaphane on cardiac injury induced by sepsis in a mouse model: Role of toll-like receptor 4.
Topics: Animals; Cardiomyopathies; Heart Injuries; Male; Mice; Sepsis; Toll-Like Receptor 4 | 2023 |
Sulforaphane Reduces HMGB1-Mediated Septic Responses and Improves Survival Rate in Septic Mice.
Topics: Animals; Anti-Inflammatory Agents; Brassicaceae; Cell Movement; Disease Models, Animal; HMGB1 Protei | 2017 |
Sulforaphane inhibits endothelial protein C receptor shedding in vitro and in vivo.
Topics: ADAM Proteins; ADAM17 Protein; Animals; Blood Coagulation Factors; Cecum; Disease Models, Animal; Hu | 2014 |
Sulforaphane induces neurovascular protection against a systemic inflammatory challenge via both Nrf2-dependent and independent pathways.
Topics: Adolescent; Adult; Animals; Anti-Inflammatory Agents; Antioxidants; Brain; Cell Line; Disease Models | 2016 |