sulfadimethoxine has been researched along with Hyperbilirubinemia, Hereditary in 3 studies
Sulfadimethoxine: A sulfanilamide that is used as an anti-infective agent.
sulfadimethoxine : A sulfonamide consisting of pyrimidine having methoxy substituents at the 2- and 6-positions and a 4-aminobenzenesulfonamido group at the 4-position.
Hyperbilirubinemia, Hereditary: Inborn errors of bilirubin metabolism resulting in excessive amounts of bilirubin in the circulating blood, either because of increased bilirubin production or because of delayed clearance of bilirubin from the blood.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 3 (100.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Jansen, FH | 2 |
| Malvaux, P | 1 |
| Heirwegh, KP | 2 |
| Devriendt, A | 2 |
| Schutta, HS | 1 |
| Johnson, L | 1 |
| De Groote, J | 1 |
3 other studies available for sulfadimethoxine and Hyperbilirubinemia, Hereditary
| Article | Year |
|---|---|
Congenital non-hemolytic jaundice: Crigler-Najjar syndrome.
Topics: Autopsy; Bilirubin; Chemical Phenomena; Chemistry; Chromatography; Female; Glucuronates; Humans; Hyp | 1969 |
Clinical signs and morphologic abnormalities in Gunn rats treated with sulfadimethoxine.
Topics: Age Factors; Animals; Bilirubin; Brain; Brain Damage, Chronic; Disease Models, Animal; Humans; Hyper | 1969 |
[Further biochemical aspects of non-hemolytic congenital jaundice. The Crigler-Najjar syndrome].
Topics: Animals; Glucosyltransferases; Glucuronates; Humans; Hyperbilirubinemia, Hereditary; Liver; Metaboli | 1968 |