succinic acid has been researched along with Metabolic Syndrome in 5 studies
Succinic Acid: A water-soluble, colorless crystal with an acid taste that is used as a chemical intermediate, in medicine, the manufacture of lacquers, and to make perfume esters. It is also used in foods as a sequestrant, buffer, and a neutralizing agent. (Hawley's Condensed Chemical Dictionary, 12th ed, p1099; McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed, p1851)
succinic acid : An alpha,omega-dicarboxylic acid resulting from the formal oxidation of each of the terminal methyl groups of butane to the corresponding carboxy group. It is an intermediate metabolite in the citric acid cycle.
Metabolic Syndrome: A cluster of symptoms that are risk factors for CARDIOVASCULAR DISEASES and TYPE 2 DIABETES MELLITUS. The major components of metabolic syndrome include ABDOMINAL OBESITY; atherogenic DYSLIPIDEMIA; HYPERTENSION; HYPERGLYCEMIA; INSULIN RESISTANCE; a proinflammatory state; and a prothrombotic (THROMBOSIS) state.
| Excerpt | Relevance | Reference |
|---|---|---|
| "In this work, we examined whether baicalin (BC), a bioactive flavonoid in Scutellaria baicalensis Georgi, can reduce high-fat diet (HFD)-induced metabolic syndrome (MetS) in mice." | 8.12 | Baicalin attenuate diet-induced metabolic syndrome by improving abnormal metabolism and gut microbiota. ( Huang, TQ; Jiang, ZM; Liang, HT; Lin, Y; Liu, EH; Qin, YQ; Song, Y; Wang, MJ; Wang, ZY, 2022) |
| "In this work, we examined whether baicalin (BC), a bioactive flavonoid in Scutellaria baicalensis Georgi, can reduce high-fat diet (HFD)-induced metabolic syndrome (MetS) in mice." | 4.12 | Baicalin attenuate diet-induced metabolic syndrome by improving abnormal metabolism and gut microbiota. ( Huang, TQ; Jiang, ZM; Liang, HT; Lin, Y; Liu, EH; Qin, YQ; Song, Y; Wang, MJ; Wang, ZY, 2022) |
| "Metabolic syndrome is associated with overactive bladder syndrome (OAB) and increased circulating levels of succinate, an intermediate of the Krebs cycle." | 1.46 | Succinate, increased in metabolic syndrome, activates GPR91 receptor signaling in urothelial cells. ( Cammisotto, PG; Campeau, L; Mossa, AH; Velasquez Flores, M, 2017) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (60.00) | 24.3611 |
| 2020's | 2 (40.00) | 2.80 |
| Authors | Studies |
|---|---|
| Lin, Y | 1 |
| Wang, ZY | 1 |
| Wang, MJ | 1 |
| Jiang, ZM | 1 |
| Qin, YQ | 1 |
| Huang, TQ | 1 |
| Song, Y | 1 |
| Liang, HT | 1 |
| Liu, EH | 1 |
| Karimi, M | 1 |
| Petkova, V | 1 |
| Asara, JM | 1 |
| Griffin, MJ | 1 |
| Sellke, FW | 1 |
| Bishop, AR | 1 |
| Alexandrov, BS | 1 |
| Usheva, A | 1 |
| Mossa, AH | 1 |
| Velasquez Flores, M | 2 |
| Cammisotto, PG | 2 |
| Campeau, L | 2 |
| Mossa, A | 1 |
| Nguyen, H | 1 |
| Gattu, AK | 1 |
| Birkenfeld, AL | 1 |
| Iwakiri, Y | 1 |
| Jay, S | 1 |
| Saltzman, M | 1 |
| Doll, J | 1 |
| Protiva, P | 1 |
| Samuel, VT | 1 |
| Crawford, SE | 1 |
| Chung, C | 1 |
5 other studies available for succinic acid and Metabolic Syndrome
| Article | Year |
|---|---|
Baicalin attenuate diet-induced metabolic syndrome by improving abnormal metabolism and gut microbiota.
Topics: Animals; Diet, High-Fat; DNA, Ribosomal; Flavonoids; Gastrointestinal Microbiome; Metabolic Syndrome | 2022 |
Metabolomics and the pig model reveal aberrant cardiac energy metabolism in metabolic syndrome.
Topics: Animals; Biomarkers; Citric Acid Cycle; Diet, High-Fat; Disease Models, Animal; Energy Metabolism; G | 2020 |
Succinate, increased in metabolic syndrome, activates GPR91 receptor signaling in urothelial cells.
Topics: Animals; Cells, Cultured; Female; Metabolic Syndrome; Rats, Sprague-Dawley; Receptors, G-Protein-Cou | 2017 |
Beta-3 Adrenoceptor Signaling Pathways in Urothelial and Smooth Muscle Cells in the Presence of Succinate.
Topics: Acetanilides; Animals; Cyclic AMP; Female; Metabolic Syndrome; Muscle Relaxation; Muscle, Smooth; My | 2018 |
Pigment epithelium-derived factor (PEDF) suppresses IL-1β-mediated c-Jun N-terminal kinase (JNK) activation to improve hepatocyte insulin signaling.
Topics: Adipocytes; Animals; Eye Proteins; Gene Expression Regulation; Glucose Tolerance Test; Hepatocytes; | 2014 |