substance p has been researched along with Injury, Myocardial Reperfusion in 18 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (11.11) | 18.2507 |
| 2000's | 7 (38.89) | 29.6817 |
| 2010's | 8 (44.44) | 24.3611 |
| 2020's | 1 (5.56) | 2.80 |
| Authors | Studies |
|---|---|
| Barbato, E; Bellis, A; Di Gioia, G; Mauro, C; Morisco, C; Sorriento, D; Trimarco, B | 1 |
| Jaggi, AS; Randhawa, PK | 1 |
| Chen, L; Guo, Z; Li, TP; Liu, CJ; Sun, T; Zhao, X | 1 |
| Gu, Q; Qian, Y; Qu, C; Tan, X; Tang, J; Xu, Y; Yu, Z | 1 |
| Cheng, XW; Hong, HS; Kim, S; Kim, W; Lee, KH; Lee, S; Son, Y; Woo, JS | 1 |
| Dehlin, HM; Goldspink, PH; Janicki, JS; Jubair, S; Levick, SP; Li, J; Manteufel, EJ | 1 |
| Cao, C; Chen, H; Gao, Y; Lee, C; Song, J | 1 |
| Wang, DH; Zhong, B | 3 |
| Aust, W; Fitzl, G; Löster, H; Schneider, R; Welt, K | 1 |
| Brazin, J; Corti, F; Levi, R; Morrey, C; Seyedi, N; Silver, RB | 1 |
| Chen, H; Lu, MY; Ren, JY; Song, JX | 1 |
| Kramer, JH; Mak, IT; Phillips, TM; Weglicki, WB | 1 |
| Gams, E; Garcia, SC; Langenbach, MR; Pomblum, V; Schipke, JD | 1 |
| Cassidy, MM; Dickens, BF; Kramer, JH; Mak, IT; Phillips, TM; Stafford, R; Weglicki, WB | 1 |
| Kramer, JH; Phillips, TM; Weglicki, WB | 1 |
| Gonon, A; Gourine, A; Pernow, J; Sjöquist, PO | 1 |
2 review(s) available for substance p and Injury, Myocardial Reperfusion
| Article | Year |
|---|---|
The Rationale of Neprilysin Inhibition in Prevention of Myocardial Ischemia-Reperfusion Injury during ST-Elevation Myocardial Infarction.
Topics: Adrenomedullin; Aminobutyrates; Angiotensin II; Animals; Apelin; Atrial Natriuretic Factor; Biphenyl Compounds; Bradykinin; Cardiotonic Agents; Drug Combinations; Gene Expression Regulation; Humans; Mice; Myocardial Reperfusion Injury; Neprilysin; ST Elevation Myocardial Infarction; Substance P; Survival Analysis; Tetrazoles; Valsartan; Ventricular Remodeling | 2020 |
A Review on Potential Involvement of TRPV
Topics: Animals; Calcitonin Gene-Related Peptide; Cytokines; Free Radicals; Humans; Inflammation Mediators; Myocardial Reperfusion Injury; Reperfusion Injury; Substance P; TRPV Cation Channels | 2018 |
16 other study(ies) available for substance p and Injury, Myocardial Reperfusion
| Article | Year |
|---|---|
Association of down-regulation of calcitonin gene-related peptide and substance P with increase of myocardial vulnerability in diabetic neuropathic rats.
Topics: Animals; Calcitonin Gene-Related Peptide; Diabetes Mellitus, Experimental; Diabetic Neuropathies; Male; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Rats; Substance P | 2017 |
Substance P Attenuates Hypoxia/Reoxygenation-Induced Apoptosis via the Akt Signalling Pathway and the NK1-Receptor in H9C2Cells.
Topics: Animals; Apoptosis; Blotting, Western; Cells, Cultured; Disease Models, Animal; Hypoxia; Immunohistochemistry; Myocardial Reperfusion Injury; Myocytes, Cardiac; Neurotransmitter Agents; Proto-Oncogene Proteins c-akt; Receptors, Neurokinin-1; Signal Transduction; Substance P | 2018 |
Substance-P Prevents Cardiac Ischemia-Reperfusion Injury by Modulating Stem Cell Mobilization and Causing Early Suppression of Injury-Mediated Inflammation.
Topics: Animals; Granulocyte Colony-Stimulating Factor; Hematopoietic Stem Cell Mobilization; Inflammation; Interleukin-10; Lectins, C-Type; Male; Mannose Receptor; Mannose-Binding Lectins; Myocardial Infarction; Myocardial Reperfusion Injury; Rats; Rats, Sprague-Dawley; Receptors, Cell Surface; Substance P; Tumor Necrosis Factor-alpha | 2019 |
Substance P induces cardioprotection in ischemia-reperfusion via activation of AKT.
Topics: Animals; Cardiotonic Agents; Cell Hypoxia; Cells, Cultured; Male; Mice; Mice, Inbred C57BL; Myocardial Reperfusion Injury; Myocytes, Cardiac; Proto-Oncogene Proteins c-akt; Rats; Rats, Sprague-Dawley; Receptors, Neurokinin-1; Signal Transduction; Substance P | 2015 |
TRPV1 activation is involved in the cardioprotection of remote limb ischemic postconditioning in ischemia-reperfusion injury rats.
Topics: Animals; Calcitonin Gene-Related Peptide; Extremities; Ischemic Preconditioning; Male; Myocardial Reperfusion Injury; Rats; Rats, Sprague-Dawley; Substance P; TRPV Cation Channels | 2015 |
N-oleoyldopamine, a novel endogenous capsaicin-like lipid, protects the heart against ischemia-reperfusion injury via activation of TRPV1.
Topics: Animals; Benzophenanthridines; Calcitonin Gene-Related Peptide; Calcitonin Gene-Related Peptide Receptor Antagonists; Coronary Circulation; Dopamine; Isoindoles; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; Neurokinin-1 Receptor Antagonists; Peptide Fragments; Potassium Channel Blockers; Potassium Channels; Protein Kinase C; Protein Kinase Inhibitors; Quaternary Ammonium Compounds; Radioimmunoassay; Receptors, Calcitonin Gene-Related Peptide; Receptors, Neurokinin-1; Substance P; TRPV Cation Channels; Ventricular Function, Left; Ventricular Pressure | 2008 |
Cardiac ischemia and reperfusion in spontaneously diabetic rats with and without application of EGb 761: II. Interstitium and microvasculature.
Topics: Animals; Cardiovascular Agents; Collagen; Diabetes Complications; Diabetes Mellitus, Type 1; Free Radical Scavengers; Ginkgo biloba; Immunohistochemistry; Male; Microcirculation; Microscopy, Electron, Transmission; Myocardial Reperfusion Injury; Myocardium; Nitric Oxide Synthase Type III; Phytotherapy; Plant Extracts; Rats; Rats, Inbred BB; Substance P | 2009 |
Protease-activated receptor 2-mediated protection of myocardial ischemia-reperfusion injury: role of transient receptor potential vanilloid receptors.
Topics: Animals; Calcitonin Gene-Related Peptide; Calcitonin Gene-Related Peptide Receptor Antagonists; Cardiotonic Agents; Coronary Circulation; Cyclic AMP-Dependent Protein Kinase Catalytic Subunits; Disease Models, Animal; Fluorescent Antibody Technique; Isoindoles; Isoquinolines; L-Lactate Dehydrogenase; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardial Contraction; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Neurokinin-1 Receptor Antagonists; Oligopeptides; Peptide Fragments; Protein Kinase C-epsilon; Protein Kinase Inhibitors; Radioimmunoassay; Receptor, PAR-2; Receptors, Calcitonin Gene-Related Peptide; Receptors, Neurokinin-1; Recovery of Function; Substance P; Sulfonamides; TRPV Cation Channels; Ventricular Function, Left; Ventricular Pressure | 2009 |
Interaction between sensory C-fibers and cardiac mast cells in ischemia/reperfusion: activation of a local renin-angiotensin system culminating in severe arrhythmic dysfunction.
Topics: Aldehydes; Animals; Arrhythmias, Cardiac; beta-N-Acetylhexosaminidases; Calcitonin Gene-Related Peptide; Cell Degranulation; Cells, Cultured; Fluorescent Antibody Technique; Guinea Pigs; In Vitro Techniques; Male; Mast Cells; Myocardial Reperfusion Injury; Myocardium; Nerve Endings; Nerve Fibers, Unmyelinated; Norepinephrine; Renin; Renin-Angiotensin System; Sensory Receptor Cells; Substance P; Synaptosomes | 2010 |
Cardioprotection by ischemic postconditioning is lost in isolated perfused heart from diabetic rats: Involvement of transient receptor potential vanilloid 1, calcitonin gene-related peptide and substance P.
Topics: Animals; Blood Flow Velocity; Calcitonin Gene-Related Peptide; Capsaicin; Coronary Vessels; Diabetes Mellitus, Experimental; Heart; In Vitro Techniques; Ischemic Postconditioning; Male; Myocardial Reperfusion Injury; Myocardium; Neurokinin-1 Receptor Antagonists; Peptide Fragments; Rats; Rats, Sprague-Dawley; Substance P; TRPV Cation Channels; Ventricular Function | 2011 |
Dietary magnesium intake influences circulating pro-inflammatory neuropeptide levels and loss of myocardial tolerance to postischemic stress.
Topics: Animals; Disease Models, Animal; Electron Spin Resonance Spectroscopy; Enzyme-Linked Immunosorbent Assay; Erythrocytes; Glutathione; Hemodynamics; Magnesium; Magnesium Deficiency; Male; Malondialdehyde; Myocardial Ischemia; Myocardial Reperfusion Injury; Neurogenic Inflammation; Oxidative Stress; Rats; Rats, Sprague-Dawley; Spin Trapping; Substance P | 2003 |
Independency of myocardial stunning of endothelial stunning?
Topics: Animals; Blood Pressure; Coronary Circulation; Endothelium, Vascular; In Vitro Techniques; Male; Molsidomine; Muscle, Smooth, Vascular; Myocardial Reperfusion Injury; Myocardial Stunning; Rabbits; Research Design; Substance P; Time Factors; Vasodilation; Vasodilator Agents; Ventricular Function | 2007 |
TRPV1 gene knockout impairs preconditioning protection against myocardial injury in isolated perfused hearts in mice.
Topics: Animals; Indoles; Ischemic Preconditioning, Myocardial; Isoindoles; L-Lactate Dehydrogenase; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Neurokinin-1 Receptor Antagonists; Receptors, Calcitonin Gene-Related Peptide; Receptors, Neurokinin-1; Substance P; TRPV Cation Channels; Ventricular Dysfunction, Left | 2007 |
Cytokines, neuropeptides, and reperfusion injury during magnesium deficiency.
Topics: Animals; Calcitonin Gene-Related Peptide; Cardiomyopathies; Cricetinae; Cytokines; Inflammation; Magnesium Deficiency; Myocardial Reperfusion Injury; Neuropeptides; Rats; Reactive Oxygen Species; Substance P; Time Factors; Vitamin E | 1994 |
Magnesium-deficiency-enhanced post-ischemic myocardial injury is reduced by substance P receptor blockade.
Topics: Animals; Cytokines; Drug Evaluation, Preclinical; Hemodynamics; Magnesium Deficiency; Male; Myocardial Reperfusion Injury; Neurokinin-1 Receptor Antagonists; Neuropeptides; Quinuclidines; Rats; Rats, Sprague-Dawley; Substance P | 1997 |
Short-acting calcium antagonist clevidipine protects against reperfusion injury via local nitric oxide-related mechanisms in the jeopardised myocardium.
Topics: Analysis of Variance; Animals; Arginine; Calcium Channel Blockers; Endothelium, Vascular; Enzyme Inhibitors; Female; Male; Myocardial Reperfusion Injury; Myocardium; Nitric Oxide; Nitric Oxide Synthase; omega-N-Methylarginine; Pyridines; Random Allocation; Substance P; Swine; Vascular Resistance | 2001 |