substance p has been researched along with Diabetic Nephropathies in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (50.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Akerman, KE; Chapman, H; Hukkanen, M; Koivisto, A; Kuokkanen, K; Lindstedt, K; Pertovaara, A; Saarnilehto, M; Viisanen, H; Wei, H | 1 |
Arai, D; Bloom, DC; Lam, H; Maidment, NT; Matsuka, Y; Spigelman, I; Tran, C; Walwyn, WM | 1 |
2 other study(ies) available for substance p and Diabetic Nephropathies
Article | Year |
---|---|
Inhibiting TRPA1 ion channel reduces loss of cutaneous nerve fiber function in diabetic animals: sustained activation of the TRPA1 channel contributes to the pathogenesis of peripheral diabetic neuropathy.
Topics: Animals; Calcium Channels; Calcium Signaling; Diabetes Mellitus, Experimental; Diabetic Nephropathies; HEK293 Cells; Humans; Male; Membrane Potentials; Nerve Fibers; Nerve Tissue Proteins; Neural Conduction; Neurons, Afferent; Pain Threshold; Rats; Reflex; Sensory System Agents; Skin; Substance P; Time Factors; Transfection; Transient Receptor Potential Channels; TRPA1 Cation Channel; TRPC Cation Channels | 2012 |
HSV-1-mediated NGF delivery delays nociceptive deficits in a genetic model of diabetic neuropathy.
Topics: Action Potentials; Age Factors; Animals; Behavior, Animal; Blotting, Northern; Cell Count; Diabetic Nephropathies; Disease Models, Animal; Dose-Response Relationship, Radiation; Electric Stimulation; Ganglia, Spinal; Genetic Therapy; Genetic Vectors; Immunohistochemistry; Male; Mice; Mice, Inbred C57BL; Nerve Fibers; Nerve Growth Factors; Pain Measurement; Reaction Time; Receptor, Nerve Growth Factor; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Simplexvirus; Substance P | 2006 |