Page last updated: 2024-08-23

substance p and Cot Death

substance p has been researched along with Cot Death in 20 studies

Research

Studies (20)

TimeframeStudies, this research(%)All Research%
pre-19902 (10.00)18.7374
1990's7 (35.00)18.2507
2000's6 (30.00)29.6817
2010's5 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bright, FM; Byard, RW; Duncan, JR; Krous, HF; Paterson, DS; Vink, R1
Bright, F; Byard, RW; Vink, R1
Bright, FM; Byard, RW; Vink, R1
Xu, F; Zang, N; Zhao, L; Zhuang, J1
Lavezzi, AM; Matturri, L; Mehboob, R1
Ozawa, Y; Takashima, S1
Groswasser, J; Kadhim, H; Kahn, A; Nishida, H; Ozawa, Y; Patricia, F; Sawaguchi, T; Sottiaux, M; Takashima, S1
Biondo, B; Bruni, B; Cazzullo, A; Magagnin, S; Matturri, L; Tosi, D1
Bergström, L; Lagercrantz, H; Terenius, L1
Mito, T; Takashima, S; Yamanouchi, H1
Hertzberg, T; Lagercrantz, H; Srinivasan, M1
Becker, LE; Takashima, S; Yamanouchi, H1
Asanuma, M; Becker, LE; Horie, H; Mizuta, R; Obonai, T; Takashima, S; Tanaka, J1
Bouvier, R; Dijoud, F; Jordan, D; Kermadi, I; Kopp, N; Martin, D; Rambaud, C1
Mallard, C; McGregor, H; Rees, S; Tolcos, M; Walker, D1
McGregor, H; Rees, S; Tolcos, M; Walker, D1
Bauman, NM; Luschei, ES; Sandler, AD; Wang, D1
Glaser, S; Knöfel, B; Oehme, P; Rathsack, R; Scheidt, B; Scholle, S; Zwacka, G1
Lagercrantz, H; Prabhakar, N; Srinivasan, M; Yamamoto, Y1
Frenzel, J; Oehme, P; Rathsack, R; Scheidt, B; Scholle, S; Zwacka, G1

Reviews

3 review(s) available for substance p and Cot Death

ArticleYear
The potential role of substance P in brainstem homeostatic control in the pathogenesis of sudden infant death syndrome (SIDS).
    Neuropeptides, 2018, Volume: 70

    Topics: Brain Stem; Homeostasis; Humans; Infant; Infant, Newborn; Receptors, Neurokinin-1; Serotonin; Substance P; Sudden Infant Death

2018
Disturbed chemical neurotransmission and sudden infant death syndrome--the peripheral arterial chemoreceptors as an example.
    Acta paediatrica (Oslo, Norway : 1992). Supplement, 1993, Volume: 82 Suppl 389

    Topics: Animals; Arteries; Catecholamines; Chemoreceptor Cells; Humans; Infant; Infant, Newborn; Neurotransmitter Agents; Substance P; Sudden Infant Death

1993
Functional role of substance P for respiratory control during development.
    Annals of the New York Academy of Sciences, 1991, Volume: 632

    Topics: Aging; Animals; Brain; Humans; Infant, Newborn; Neurotransmitter Agents; Protein Precursors; Respiration; Substance P; Sudden Infant Death; Tachykinins

1991

Other Studies

17 other study(ies) available for substance p and Cot Death

ArticleYear
Abnormalities in substance P neurokinin-1 receptor binding in key brainstem nuclei in sudden infant death syndrome related to prematurity and sex.
    PloS one, 2017, Volume: 12, Issue:9

    Topics: Brain Stem; Cohort Studies; Female; Humans; Infant; Infant, Newborn; Infant, Premature; Male; Medulla Oblongata; Olivary Nucleus; Protein Binding; Receptors, Neurokinin-1; Substance P; Sudden Infant Death

2017
Why is a prone sleeping position dangerous for certain infants?
    Forensic science, medicine, and pathology, 2018, Volume: 14, Issue:1

    Topics: Asphyxia; Bedding and Linens; Brain Stem; Humans; Infant; Prone Position; Risk Factors; Sleep; Sleep Arousal Disorders; Substance P; Sudden Infant Death

2018
Prenatal nicotinic exposure augments cardiorespiratory responses to activation of bronchopulmonary C-fibers.
    American journal of physiology. Lung cellular and molecular physiology, 2015, May-01, Volume: 308, Issue:9

    Topics: Animals; Apnea; Brain-Derived Neurotrophic Factor; Bronchoalveolar Lavage Fluid; Capsaicin; Diaphragm; Electromyography; Female; Ganglia; Humans; Hypoxia; Infant, Newborn; Male; Methacholine Chloride; Nerve Fibers, Unmyelinated; Nerve Growth Factor; Nicotine; Nodose Ganglion; Pregnancy; Prenatal Exposure Delayed Effects; Random Allocation; Rats; Rats, Sprague-Dawley; Receptor, trkA; Receptor, trkB; Recombinant Proteins; Sensory System Agents; Smoking; Substance P; Sudden Infant Death; TRPV Cation Channels

2015
Developmental alterations of the spinal trigeminal nucleus disclosed by substance P immunohistochemistry in fetal and infant sudden unexplained deaths.
    Neuropathology : official journal of the Japanese Society of Neuropathology, 2011, Volume: 31, Issue:4

    Topics: Female; Fetal Death; Humans; Infant; Infant, Newborn; Pregnancy; Substance P; Sudden Infant Death; Trigeminal Nucleus, Spinal

2011
Developmental neurotransmitter pathology in the brainstem of sudden infant death syndrome: a review and sleep position.
    Forensic science international, 2002, Sep-14, Volume: 130 Suppl

    Topics: Astrocytes; Brain Stem; Case-Control Studies; Catecholamines; Glial Fibrillary Acidic Protein; Gliosis; Humans; Hypoxia; Infant; Neural Pathways; Neurons; Neurotransmitter Agents; Prone Position; Receptors, Neurotransmitter; Receptors, Serotonin; Respiration; Risk Factors; Serotonin; Substance P; Sudden Infant Death; Supine Position

2002
Substance P in the midbrains of SIDS victims and its correlation with sleep apnea.
    Early human development, 2003, Volume: 75 Suppl

    Topics: Double-Blind Method; Female; Humans; Immunohistochemistry; Infant; Infant, Newborn; Male; Mesencephalon; Polysomnography; Prospective Studies; Sleep Apnea, Obstructive; Statistics, Nonparametric; Substance P; Sudden Infant Death; Supine Position

2003
Glial and neuronal alterations in the nucleus tractus solitarii of sudden infant death syndrome victims.
    Acta neuropathologica, 2004, Volume: 108, Issue:4

    Topics: Apoptosis; Female; Humans; Immunohistochemistry; In Situ Nick-End Labeling; Infant; Infant, Newborn; Male; Neuroglia; Neurons; Receptors, N-Methyl-D-Aspartate; Solitary Nucleus; Substance P; Sudden Infant Death

2004
Post-mortem analyses of neuropeptides in brains from sudden infant death victims.
    Brain research, 1984, Dec-10, Volume: 323, Issue:2

    Topics: Age Factors; Brain Chemistry; Enkephalin, Methionine; Female; Humans; Infant; Infant, Newborn; Male; Nerve Tissue Proteins; Neuropeptide Y; Postmortem Changes; Sex Factors; Substance P; Sudden Infant Death

1984
Developmental brain-stem pathology in sudden infant death syndrome.
    Acta paediatrica Japonica : Overseas edition, 1994, Volume: 36, Issue:3

    Topics: Brain Stem; Humans; Infant; Infant, Newborn; Substance P; Sudden Infant Death

1994
Correlation of astrogliosis and substance P immunoreactivity in the brainstem of victims of sudden infant death syndrome.
    Neuropediatrics, 1993, Volume: 24, Issue:4

    Topics: Astrocytes; Brain Stem; Female; Glial Fibrillary Acidic Protein; Gliosis; Humans; Immunoenzyme Techniques; Infant; Male; Nerve Fibers; Substance P; Sudden Infant Death; Trigeminal Nucleus, Spinal

1993
Relationship of substance P and gliosis in medulla oblongata in neonatal sudden infant death syndrome.
    Pediatric neurology, 1996, Volume: 15, Issue:3

    Topics: Chi-Square Distribution; Glial Fibrillary Acidic Protein; Gliosis; Humans; Infant, Newborn; Medulla Oblongata; Substance P; Sudden Infant Death

1996
Autoradiographic distribution of brainstem substance P binding sites in humans: ontogenic study and relation to sudden infant death syndrome (SIDS).
    Journal of neural transmission (Vienna, Austria : 1996), 1997, Volume: 104, Issue:10

    Topics: Autoradiography; Binding Sites; Brain Stem; Female; Humans; Infant; Infant, Newborn; Male; Pregnancy; Receptors, Neurokinin-1; Substance P; Sudden Infant Death

1997
Exposure to prenatal carbon monoxide and postnatal hyperthermia: short and long-term effects on neurochemicals and neuroglia in the developing brain.
    Experimental neurology, 2000, Volume: 162, Issue:2

    Topics: Animals; Animals, Newborn; Body Weight; Brain; Brain Chemistry; Carbon Monoxide; Cerebral Cortex; Enkephalin, Methionine; Female; Glial Fibrillary Acidic Protein; Guinea Pigs; Hyperthermia, Induced; Immunohistochemistry; Inhalation Exposure; Maternal Exposure; Medulla Oblongata; Neuroglia; Organ Size; Pregnancy; Prenatal Exposure Delayed Effects; Serotonin; Substance P; Sudden Infant Death; Thalamus; Tyrosine 3-Monooxygenase

2000
Chronic prenatal exposure to carbon monoxide results in a reduction in tyrosine hydroxylase-immunoreactivity and an increase in choline acetyltransferase-immunoreactivity in the fetal medulla: implications for Sudden Infant Death Syndrome.
    Journal of neuropathology and experimental neurology, 2000, Volume: 59, Issue:3

    Topics: Animals; Astrocytes; Blotting, Western; Carbon Monoxide; Choline O-Acetyltransferase; Enkephalin, Methionine; Female; Glial Fibrillary Acidic Protein; Guinea Pigs; Medulla Oblongata; Neurons; Pregnancy; Prenatal Exposure Delayed Effects; Receptor, Muscarinic M2; Receptors, Muscarinic; Smoking; Substance P; Sudden Infant Death; Tyrosine 3-Monooxygenase

2000
Response of the cricothyroid and thyroarytenoid muscles to stereotactic injection of substance P into the region of the nucleus tractus solitarius in developing dogs.
    The Annals of otology, rhinology, and laryngology, 2000, Volume: 109, Issue:12 Pt 1

    Topics: Age Factors; Animals; Disease Models, Animal; Dogs; Electromyography; Humans; Infant, Newborn; Laryngeal Muscles; Laryngismus; Motor Activity; Muscle Development; Solitary Nucleus; Stereotaxic Techniques; Substance P; Sudden Infant Death

2000
Substance P, mean apnoea duration and the sudden infant death syndrome (SIDS).
    Biomedica biochimica acta, 1990, Volume: 49, Issue:4

    Topics: Humans; Infant; Infant, Newborn; Infant, Premature; Respiratory System; Risk; Sleep Apnea Syndromes; Substance P; Sudden Infant Death; Time Factors

1990
Substance P in infants with relation to the sudden infant death syndrome--a preliminary report.
    Die Pharmazie, 1988, Volume: 43, Issue:6

    Topics: Animals; Female; Humans; Infant; Infant, Newborn; Male; Risk Factors; Substance P; Sudden Infant Death

1988