substance p has been researched along with Carcinoma, Ductal, Pancreatic in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Askan, G; Bailey, JM; Baker, L; Basturk, O; Fu, YY; Grbovic-Huezo, O; Grimont, A; Joo, MG; Ketcham, M; Kurtz, RC; Lafaro, K; Leach, SD; Melchor, JP; Park, Y; Pasricha, PJ; Saglimbeni, JA; Sinha, S; Tuveson, D; Yang, IH; Zhong, Y | 1 |
| Burdick, M; Eibl, G; Fan, RS; Guha, S; Hines, OJ; Kisfalvi, K; Reber, H; Rozengurt, E; Strieter, R | 1 |
2 other study(ies) available for substance p and Carcinoma, Ductal, Pancreatic
| Article | Year |
|---|---|
PanIN Neuroendocrine Cells Promote Tumorigenesis via Neuronal Cross-talk.
Topics: 3T3 Cells; Animals; Carcinogenesis; Carcinoma, Pancreatic Ductal; Disease Models, Animal; Ganglia, Spinal; HEK293 Cells; Humans; Mice; Mice, Inbred C57BL; Neuroendocrine Cells; Pancreas; Pancreatic Neoplasms; Precancerous Conditions; Sensory Receptor Cells; STAT3 Transcription Factor; Substance P | 2017 |
Broad-spectrum G protein-coupled receptor antagonist, [D-Arg1,D-Trp5,7,9,Leu11]SP: a dual inhibitor of growth and angiogenesis in pancreatic cancer.
Topics: Animals; Apoptosis; Carcinoma, Pancreatic Ductal; Cell Growth Processes; Cornea; DNA, Neoplasm; Humans; Interleukin-8; Ki-67 Antigen; Male; Mice; Mice, Nude; Neovascularization, Pathologic; Neovascularization, Physiologic; Pancreatic Neoplasms; Rats; Receptors, G-Protein-Coupled; Substance P; Xenograft Model Antitumor Assays | 2005 |