sr 48692 has been researched along with Cancer of Lung in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chan, DC; Jensen, RT; Mantey, SA; Moody, TW; Moreno, P | 1 |
| Casibang, M; Chan, D; Chiles, J; Davis, TP; Moody, E; Moody, TW | 1 |
| Garcia-Marin, L; Jensen, RT; Leyton, J; Moody, TW | 1 |
3 other study(ies) available for sr 48692 and Cancer of Lung
| Article | Year |
|---|---|
SR48692 inhibits non-small cell lung cancer proliferation in an EGF receptor-dependent manner.
Topics: Antineoplastic Agents; Carcinoma, Non-Small-Cell Lung; Cell Line, Tumor; Cell Proliferation; Drug Screening Assays, Antitumor; ErbB Receptors; Extracellular Signal-Regulated MAP Kinases; Gefitinib; Gene Knockdown Techniques; Humans; Lung Neoplasms; Phosphorylation; Protein Processing, Post-Translational; Pyrazoles; Quinazolines; Quinolines; Receptors, Neurotensin; RNA, Small Interfering; Transcriptional Activation; Transforming Growth Factor alpha | 2014 |
SR48692 is a neurotensin receptor antagonist which inhibits the growth of small cell lung cancer cells.
Topics: Animals; Carcinoma, Small Cell; Cell Division; Humans; Lung Neoplasms; Mice; Mice, Nude; Neoplasm Transplantation; Pyrazoles; Quinolines; Receptors, Neurotensin; Signal Transduction; Tumor Cells, Cultured | 2001 |
Neurotensin causes tyrosine phosphorylation of focal adhesion kinase in lung cancer cells.
Topics: Binding, Competitive; Cell Division; Cytochalasin D; Dose-Response Relationship, Drug; Focal Adhesion Kinase 1; Focal Adhesion Protein-Tyrosine Kinases; Humans; Lung Neoplasms; Neurotensin; Nucleic Acid Synthesis Inhibitors; Peptide Fragments; Phosphorylation; Piperidines; Protein-Tyrosine Kinases; Pyrazoles; Quinolines; Receptors, Neurotensin; Tumor Cells, Cultured; Tyrosine | 2002 |