spermine has been researched along with Acute Brain Injuries in 9 studies
| Excerpt | Relevance | Reference |
|---|---|---|
| " Here, we show that extracellular spermine, one of the endogenous polyamines, exacerbates ischemic neuronal injury through sensitization of ASIC1a channels to extracellular acidosis." | 7.77 | Extracellular spermine exacerbates ischemic neuronal injury through sensitization of ASIC1a channels to extracellular acidosis. ( Cao, H; Chu, XP; Duan, B; Hansen, J; Huang, Y; Simon, RP; Wang, YZ; Xiong, ZG; Xu, TL; Yang, T; Yu, Y; Zhu, MX, 2011) |
| " Here, we show that extracellular spermine, one of the endogenous polyamines, exacerbates ischemic neuronal injury through sensitization of ASIC1a channels to extracellular acidosis." | 3.77 | Extracellular spermine exacerbates ischemic neuronal injury through sensitization of ASIC1a channels to extracellular acidosis. ( Cao, H; Chu, XP; Duan, B; Hansen, J; Huang, Y; Simon, RP; Wang, YZ; Xiong, ZG; Xu, TL; Yang, T; Yu, Y; Zhu, MX, 2011) |
| "Kukoamine A (KuA) has the ability of anti-oxidative stress and anti-apoptosis in vitro." | 1.43 | Neuroprotective Effects of Kukoamine a against Radiation-induced Rat Brain Injury through Inhibition of Oxidative Stress and Neuronal Apoptosis. ( Cheng, Z; Ma, H; Meng, W; Wang, C; Zhang, Y; Zhao, Q, 2016) |
| "Spermine oxidase is a FAD-containing enzyme involved in polyamines catabolism, selectively oxidizing spermine to produce H2O2, spermidine, and 3-aminopropanal." | 1.39 | A New Transgenic Mouse Model for Studying the Neurotoxicity of Spermine Oxidase Dosage in the Response to Excitotoxic Injury. ( Amendola, R; Bellavia, G; Berger, J; Cavallucci, V; Cecconi, F; Cervelli, M; D'Amelio, M; Marcoli, M; Mariottini, P; Maura, G; Moreno, S; Nardacci, R; Piacentini, M, 2013) |
| "Spermine levels were normal at 6 h and decreased slightly at 24 h, but were normal again by 72 h post-injury." | 1.36 | Polyamine catabolism is enhanced after traumatic brain injury. ( Casero, RA; Huttinger, F; Morrison, R; Murray-Stewart, T; Strauss, KI; Zahedi, K, 2010) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (22.22) | 29.6817 |
| 2010's | 7 (77.78) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cervelli, M | 1 |
| Bellavia, G | 1 |
| D'Amelio, M | 1 |
| Cavallucci, V | 1 |
| Moreno, S | 1 |
| Berger, J | 1 |
| Nardacci, R | 1 |
| Marcoli, M | 1 |
| Maura, G | 1 |
| Piacentini, M | 1 |
| Amendola, R | 1 |
| Cecconi, F | 1 |
| Mariottini, P | 1 |
| Hu, XL | 1 |
| Gao, LY | 1 |
| Niu, YX | 1 |
| Tian, X | 1 |
| Wang, J | 1 |
| Meng, WH | 1 |
| Zhang, Q | 1 |
| Cui, C | 1 |
| Han, L | 1 |
| Zhao, QC | 1 |
| Yan, H | 1 |
| Hao, S | 1 |
| Sun, X | 1 |
| Zhang, D | 1 |
| Gao, X | 1 |
| Yu, Z | 1 |
| Li, K | 1 |
| Hang, CH | 1 |
| Zhang, Y | 1 |
| Cheng, Z | 1 |
| Wang, C | 1 |
| Ma, H | 1 |
| Meng, W | 1 |
| Zhao, Q | 1 |
| Zahedi, K | 1 |
| Huttinger, F | 1 |
| Morrison, R | 1 |
| Murray-Stewart, T | 1 |
| Casero, RA | 1 |
| Strauss, KI | 1 |
| Duan, B | 1 |
| Wang, YZ | 1 |
| Yang, T | 1 |
| Chu, XP | 1 |
| Yu, Y | 1 |
| Huang, Y | 1 |
| Cao, H | 1 |
| Hansen, J | 1 |
| Simon, RP | 1 |
| Zhu, MX | 1 |
| Xiong, ZG | 1 |
| Xu, TL | 1 |
| Yamashita, S | 1 |
| Hasuo, H | 1 |
| Tokutomi, T | 1 |
| Shigemori, M | 1 |
| Akasu, T | 1 |
| Seiler, N | 1 |
| Adibhatla, RM | 1 |
| Hatcher, JF | 1 |
| Sailor, K | 1 |
| Dempsey, RJ | 1 |
1 review available for spermine and Acute Brain Injuries
| Article | Year |
|---|---|
Oxidation of polyamines and brain injury.
Topics: Amine Oxidase (Copper-Containing); Animals; Brain; Brain Injuries; Cattle; Enzyme Inhibitors; Humans | 2000 |
8 other studies available for spermine and Acute Brain Injuries
| Article | Year |
|---|---|
A New Transgenic Mouse Model for Studying the Neurotoxicity of Spermine Oxidase Dosage in the Response to Excitotoxic Injury.
Topics: Animals; Brain Injuries; Disease Models, Animal; Gene Dosage; HeLa Cells; Humans; Mice; Mice, Inbred | 2013 |
Neuroprotection by Kukoamine A against oxidative stress may involve N-methyl-D-aspartate receptors.
Topics: Apoptosis; Apoptosis Regulatory Proteins; Brain Injuries; Calcium; Cell Line, Tumor; Humans; Hydroge | 2015 |
Blockage of mitochondrial calcium uniporter prevents iron accumulation in a model of experimental subarachnoid hemorrhage.
Topics: Animals; Apoptosis; Brain Injuries; Calcium Channels; Disease Models, Animal; Homeostasis; Iron; Mal | 2015 |
Neuroprotective Effects of Kukoamine a against Radiation-induced Rat Brain Injury through Inhibition of Oxidative Stress and Neuronal Apoptosis.
Topics: Animals; Antioxidants; Apoptosis; Apoptosis Regulatory Proteins; Brain Injuries; Disease Models, Ani | 2016 |
Polyamine catabolism is enhanced after traumatic brain injury.
Topics: Acetyltransferases; Animals; Biogenic Polyamines; Biomarkers; Brain; Brain Chemistry; Brain Injuries | 2010 |
Extracellular spermine exacerbates ischemic neuronal injury through sensitization of ASIC1a channels to extracellular acidosis.
Topics: Acid Sensing Ion Channels; Acidosis; Amino Acid Transport System y+; Animals; Biophysics; Brain Inju | 2011 |
Edaravone attenuates impairment of synaptic plasticity in granule cell layer of the dentate gyrus following traumatic brain injury.
Topics: Animals; Antipyrine; Brain Injuries; Dentate Gyrus; Edaravone; Electric Stimulation; Excitatory Post | 2011 |
Polyamines and central nervous system injury: spermine and spermidine decrease following transient focal cerebral ischemia in spontaneously hypertensive rats.
Topics: Animals; Brain; Brain Injuries; Gerbillinae; Ischemic Attack, Transient; Putrescine; Rats; Rats, Inb | 2002 |