spermidine has been researched along with Progeria in 1 studies
Progeria: An abnormal congenital condition, associated with defects in the LAMIN TYPE A gene, which is characterized by premature aging in children, where all the changes of cell senescence occur. It is manifested by premature graying; hair loss; hearing loss (DEAFNESS); cataracts (CATARACT); ARTHRITIS; OSTEOPOROSIS; DIABETES MELLITUS; atrophy of subcutaneous fat; skeletal hypoplasia; elevated urinary HYALURONIC ACID; and accelerated ATHEROSCLEROSIS. Many affected individuals develop malignant tumors, especially SARCOMA.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ao, Y | 1 |
| Zhang, J | 1 |
| Liu, Z | 1 |
| Qian, M | 1 |
| Li, Y | 1 |
| Wu, Z | 1 |
| Sun, P | 1 |
| Wu, J | 1 |
| Bei, W | 1 |
| Wen, J | 1 |
| Wu, X | 1 |
| Li, F | 1 |
| Zhou, Z | 1 |
| Zhu, WG | 1 |
| Liu, B | 1 |
| Wang, Z | 1 |
1 other study available for spermidine and Progeria
| Article | Year |
|---|---|
Lamin A buffers CK2 kinase activity to modulate aging in a progeria mouse model.
Topics: Aging; Animals; Casein Kinase II; Cell Nucleus; Cellular Senescence; Disease Models, Animal; DNA Dam | 2019 |