Page last updated: 2024-09-05

sorafenib and Diffuse Mixed Small and Large Cell Lymphoma

sorafenib has been researched along with Diffuse Mixed Small and Large Cell Lymphoma in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Chen, Y; Gao, J; He, Z; Jia, Y; Tao, H; Wang, F; Wu, P; Zhang, X; Zhou, Z1
Chapuy, B; Dohm, A; Hand, E; Panse, M; Schroers, R; Schuelper, N; Truemper, L; Wulf, GG1
Adjei, AA; Halling, T; Haug, JL; Kimlinger, TK; Kumar, S; Rajkumar, SV; Ramakrishnan, V; Timm, M; Wellik, LE; Witzig, TE1

Other Studies

3 other study(ies) available for sorafenib and Diffuse Mixed Small and Large Cell Lymphoma

ArticleYear
Artesunate synergistically promotes sorafenib‑induced apoptosis and ferroptosis in non‑Hodgkin lymphoma cells through inhibition of the STAT3 pathway.
    Oncology reports, 2023, Volume: 50, Issue:1

    Topics: Apoptosis; Artesunate; Cell Line, Tumor; Ferroptosis; Humans; Lymphoma, Non-Hodgkin; Sorafenib; STAT3 Transcription Factor

2023
Multikinase inhibitor sorafenib exerts cytocidal efficacy against Non-Hodgkin lymphomas associated with inhibition of MAPK14 and AKT phosphorylation.
    British journal of haematology, 2011, Volume: 152, Issue:4

    Topics: Antineoplastic Agents; Apoptosis; Benzenesulfonates; Cell Cycle; Cell Death; Cell Proliferation; Dose-Response Relationship, Drug; Drug Evaluation, Preclinical; Humans; Lymphoma, Non-Hodgkin; Mitogen-Activated Protein Kinase 14; Niacinamide; Phenylurea Compounds; Phosphorylation; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Pyridines; Signal Transduction; Sorafenib; Tumor Cells, Cultured; Tumor Stem Cell Assay

2011
Sorafenib, a multikinase inhibitor, is effective in vitro against non-Hodgkin lymphoma and synergizes with the mTOR inhibitor rapamycin.
    American journal of hematology, 2012, Volume: 87, Issue:3

    Topics: Angiogenesis Inhibitors; Antineoplastic Agents; Apoptosis; Benzenesulfonates; Cell Line, Tumor; Drug Screening Assays, Antitumor; Drug Synergism; Gene Expression Regulation, Neoplastic; Humans; In Vitro Techniques; Interleukin-6; Lymphoma, Non-Hodgkin; Neoplasm Proteins; Niacinamide; Phenylurea Compounds; Pyridines; raf Kinases; Receptors, Vascular Endothelial Growth Factor; Sirolimus; Sorafenib; TOR Serine-Threonine Kinases

2012