sorafenib has been researched along with Cancer of Cervix in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 4 (66.67) | 24.3611 |
2020's | 2 (33.33) | 2.80 |
Authors | Studies |
---|---|
He, SL; Li, LJ; Wang, C; Xue, M; Zeng, J | 1 |
El-Khouly, OA; El-Messery, SM; El-Sayed, MA; Henen, MA | 1 |
Al Ghazal, A; Arndt, A; Cronauer, MV; Kraft, K; Müller, J; Schrader, AJ; Schrader, M; Skowronek, P; Steinestel, J; Steinestel, K | 1 |
Berger, W; Dornetshuber-Fleiss, R; Heffeter, P; Heilos, D; Lemmens-Gruber, R; Mohr, T; Novicky, A; Richter, L; Süssmuth, RD; Zlesak, M | 1 |
Chaudary, N; Clarke, B; Fan, S; Fyles, A; Glicksman, R; Haider, M; Hill, RP; Kim, S; MacKay, H; Milosevic, MF; Oza, AM; Pintilie, M; Townsley, CA; Yeung, I | 1 |
Cheng, CW; Hsieh, YH; Lin, CL; Lin, MT; Lin, TY; Tsai, JP; Wu, CC; Yang, SF | 1 |
1 trial(s) available for sorafenib and Cancer of Cervix
Article | Year |
---|---|
Sorafenib Increases Tumor Hypoxia in Cervical Cancer Patients Treated With Radiation Therapy: Results of a Phase 1 Clinical Study.
Topics: Angiogenesis Inhibitors; Antineoplastic Agents; Biomarkers; Brachytherapy; Carcinoma, Squamous Cell; Cell Hypoxia; Chemoradiotherapy; Cisplatin; Drug Administration Schedule; Early Termination of Clinical Trials; Female; Follow-Up Studies; Humans; Niacinamide; Oxygen; Partial Pressure; Phenylurea Compounds; Radiation Tolerance; Sorafenib; Time Factors; Tumor Burden; Uterine Cervical Neoplasms | 2016 |
5 other study(ies) available for sorafenib and Cancer of Cervix
Article | Year |
---|---|
Cdc25A inhibits autophagy-mediated ferroptosis by upregulating ErbB2 through PKM2 dephosphorylation in cervical cancer cells.
Topics: Animals; Autophagy; Carrier Proteins; cdc25 Phosphatases; Cell Line, Tumor; Female; Ferroptosis; Gene Expression Regulation, Neoplastic; Humans; Male; Membrane Proteins; Mice, Nude; Phosphorylation; Receptor, ErbB-2; RNA, Messenger; Signal Transduction; Sorafenib; Thyroid Hormone-Binding Proteins; Thyroid Hormones; Up-Regulation; Uterine Cervical Neoplasms | 2021 |
Design, synthesis and computational study of new benzofuran hybrids as dual PI3K/VEGFR2 inhibitors targeting cancer.
Topics: Antineoplastic Agents; Benzofurans; Breast Neoplasms; Cell Proliferation; Drug Design; Drug Screening Assays, Antitumor; Female; Humans; MCF-7 Cells; Molecular Docking Simulation; Molecular Structure; Phosphatidylinositol 3-Kinases; Protein Kinase Inhibitors; Sorafenib; Structure-Activity Relationship; Uterine Cervical Neoplasms; Vascular Endothelial Growth Factor Receptor-2 | 2022 |
Overexpression of p16(INK4a) in urothelial carcinoma in situ is a marker for MAPK-mediated epithelial-mesenchymal transition but is not related to human papillomavirus infection.
Topics: Aged; Carcinoma in Situ; Cell Line, Tumor; Cyclin-Dependent Kinase Inhibitor p16; Epithelial-Mesenchymal Transition; Female; Humans; Immunohistochemistry; In Situ Hybridization, Fluorescence; Male; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Niacinamide; Papillomaviridae; Papillomavirus Infections; Phenylurea Compounds; Sorafenib; Transfection; Urokinase-Type Plasminogen Activator; Urothelium; Uterine Cervical Dysplasia; Uterine Cervical Neoplasms | 2013 |
The naturally born fusariotoxin enniatin B and sorafenib exert synergistic activity against cervical cancer in vitro and in vivo.
Topics: Animals; Antineoplastic Agents; Cell Line, Tumor; Depsipeptides; Dose-Response Relationship, Drug; Drug Synergism; Female; Human Umbilical Vein Endothelial Cells; Humans; Male; Mice; Mice, SCID; Niacinamide; Phenylurea Compounds; Sorafenib; T-2 Toxin; Uterine Cervical Neoplasms; Xenograft Model Antitumor Assays | 2015 |
Synergistic effect of fisetin combined with sorafenib in human cervical cancer HeLa cells through activation of death receptor-5 mediated caspase-8/caspase-3 and the mitochondria-dependent apoptotic pathway.
Topics: Animals; Apoptosis; Caspase 3; Caspase 8; Cell Line, Tumor; Cell Proliferation; Cell Survival; Disease Models, Animal; Drug Synergism; Female; Flavonoids; Flavonols; HeLa Cells; Humans; Membrane Potential, Mitochondrial; Mitochondria; Niacinamide; Phenylurea Compounds; Receptors, TNF-Related Apoptosis-Inducing Ligand; Signal Transduction; Sorafenib; Tumor Burden; Uterine Cervical Neoplasms; Xenograft Model Antitumor Assays | 2016 |