sodium salicylate has been researched along with Adenocarcinoma, Basal Cell in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (50.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bae, YS; Chung, YM; Lee, SY | 1 |
| Choi, JE; Duong, HQ; Han, SI; Kang, HS; Kim, CH; Kim, SM; Lim, SC | 1 |
| Alpert, D; Schwenger, P; Skolnik, EY; Vilcek, J | 1 |
| Callery, MP; Carrigan, RC; McDade, TP; Perugini, RA; Vittimberga, FJ | 1 |
4 other study(ies) available for sodium salicylate and Adenocarcinoma, Basal Cell
| Article | Year |
|---|---|
Molecular ordering of ROS production, mitochondrial changes, and caspase activation during sodium salicylate-induced apoptosis.
Topics: Adenocarcinoma; Anti-Inflammatory Agents, Non-Steroidal; Apoptosis; bcl-X Protein; Caspase 3; Caspase 9; Caspases; Colonic Neoplasms; Cytochrome c Group; Enzyme Activation; Gene Expression; Humans; Membrane Potentials; Mitochondria; NADPH Oxidases; Neoplasms; Proto-Oncogene Proteins c-bcl-2; rac1 GTP-Binding Protein; Reactive Oxygen Species; Sodium Salicylate; Stomach Neoplasms; Transfection; Tumor Cells, Cultured | 2003 |
Sodium salicylate switches glucose depletion-induced necrosis to autophagy and inhibits high mobility group box protein 1 release in A549 lung adenocarcinoma cells.
Topics: Adenocarcinoma; Anti-Inflammatory Agents; Autophagy; Benzimidazoles; Cell Line, Tumor; Cyclooxygenase Inhibitors; Gene Expression Regulation, Neoplastic; Glucose; HMGB1 Protein; Humans; Lung Neoplasms; Necrosis; Sodium Salicylate; Superoxide Dismutase; Tetrazolium Salts; Thiazoles | 2008 |
Cell-type-specific activation of c-Jun N-terminal kinase by salicylates.
Topics: Acetaminophen; Adenocarcinoma; Animals; Anti-Inflammatory Agents, Non-Steroidal; Aspirin; Calcium-Calmodulin-Dependent Protein Kinases; Chlorocebus aethiops; Colonic Neoplasms; COS Cells; Cyclooxygenase Inhibitors; Enzyme Activation; Epidermal Growth Factor; Fibroblasts; Humans; Hypertonic Solutions; Ibuprofen; Indomethacin; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinases; NF-kappa B; Organ Specificity; Osmotic Pressure; p38 Mitogen-Activated Protein Kinases; Proto-Oncogene Proteins c-jun; Recombinant Fusion Proteins; Saline Solution, Hypertonic; Signal Transduction; Sodium Salicylate; Sorbitol; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha | 1999 |
Salicylates inhibit NF-kappaB activation and enhance TNF-alpha-induced apoptosis in human pancreatic cancer cells.
Topics: Adenocarcinoma; Apoptosis; Blotting, Western; Cell Nucleus; DNA Fragmentation; DNA-Binding Proteins; Enzyme-Linked Immunosorbent Assay; Humans; I-kappa B Proteins; NF-kappa B; NF-KappaB Inhibitor alpha; Pancreatic Neoplasms; Phosphorylation; Sodium Salicylate; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha | 1999 |