sodium bisulfide has been researched along with Brain Ischemia in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 4 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Dong, H; Du, Y; Fu, Y; Huang, X; Liu, H; Liu, Y; Ma, K; Qi, D; Song, Y; Sun, Y; Wen, X; Wu, J; Zhang, F | 1 |
| Ding, Q; Guo, Y; Lu, Z; Sun, X; Tao, L; Yang, L; Yang, Y; Yu, Q | 1 |
| Du, A; Li, D; Mayhan, WG; Ren, C; Sui, J; Zhao, H | 1 |
| Hao, N; Jiang, H; Li, GF; Li, LF; Li, LP; Luo, HK; Wang, WW; Xie, LJ; Zhang, JX; Zhang, QZ | 1 |
4 other study(ies) available for sodium bisulfide and Brain Ischemia
| Article | Year |
|---|---|
H₂S attenuates cognitive deficits through Akt1/JNK3 signaling pathway in ischemic stroke.
Topics: Animals; Brain Ischemia; CA1 Region, Hippocampal; Cell Survival; Chromones; Cognition Disorders; Enzyme Inhibitors; Hydrogen Sulfide; Male; MAP Kinase Kinase Kinase 5; Mitogen-Activated Protein Kinase 10; Morpholines; Neurons; Neuroprotective Agents; Phosphorylation; Proto-Oncogene Proteins c-akt; Rats, Sprague-Dawley; Signal Transduction; Stroke; Sulfides | 2014 |
ROS-Dependent Neuroprotective Effects of NaHS in Ischemia Brain Injury Involves the PARP/AIF Pathway.
Topics: Animals; Antioxidants; Brain; Brain Injuries; Brain Ischemia; Cell Line; Hydrogen Sulfide; Male; Neuroprotective Agents; Rats, Sprague-Dawley; Reactive Oxygen Species; Reperfusion Injury; Sulfides | 2015 |
Dynamic change of hydrogen sulfide during global cerebral ischemia-reperfusion and its effect in rats.
Topics: Animals; Blotting, Western; Brain Ischemia; Central Nervous System Agents; Cerebral Cortex; Cystathionine beta-Synthase; Disease Models, Animal; Hippocampus; Hydrogen Sulfide; Male; Neurons; Photomicrography; Rats; Rats, Sprague-Dawley; Reperfusion Injury; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Sulfides; Time Factors | 2010 |
Dual effects of hydrogen sulphide on focal cerebral ischaemic injury via modulation of oxidative stress-induced apoptosis.
Topics: Animals; Apoptosis; Apoptosis Regulatory Proteins; Brain Infarction; Brain Ischemia; Cerebral Cortex; Dose-Response Relationship, Drug; Hydrogen Sulfide; Infarction, Middle Cerebral Artery; Male; Mitochondria; Mitochondrial Swelling; Nerve Tissue Proteins; Neurons; Neuroprotective Agents; Oxidative Stress; Prodrugs; Random Allocation; Rats; Rats, Sprague-Dawley; Reperfusion Injury; Sulfides | 2012 |