sodium-bicarbonate and Ureteral-Obstruction

sodium-bicarbonate has been researched along with Ureteral-Obstruction* in 2 studies

Other Studies

2 other study(ies) available for sodium-bicarbonate and Ureteral-Obstruction

ArticleYear
[Percutaneous dissolution of uric acid and cystine stones causing acute ureteral obstruction].
    Hinyokika kiyo. Acta urologica Japonica, 1990, Volume: 36, Issue:11

    Four cases with ureteral obstruction due to uric acid or cystine stones were treated successfully by percutaneous irrigation with sodium bicarbonate or tromethamine-E. These cases underwent percutaneous nephrostomy for the reason of prolonged complete obstruction (case 1), sustained pyelonephritis (case 2) or decreased renal function (case 3 and 4). Two catheters were placed through the nephrostomy tract before irrigation, 6 Fr. ureteral catheter just above the ureteral stone and 10 Fr. pigtail or 12 Fr. Malecot catheter in the renal pelvis. The ureteral stones were markedly reduced in size and passed spontaneously after 6-11 days' duration of irrigation in three cases. In case 2, the remaining stone was removed transurethrally after 14 days' duration of irrigation, and was found to be composed of organic matrix. Percutaneous dissolution is considered to be a safe and reliable method and may be an alternative way of treating uric acid or cystine stone causing acute ureteral obstruction.

    Topics: Acute Disease; Adult; Bicarbonates; Cystine; Female; Humans; Male; Middle Aged; Sodium; Sodium Bicarbonate; Therapeutic Irrigation; Tromethamine; Ureteral Calculi; Ureteral Obstruction; Uric Acid; Urinary Catheterization

1990
Obstructive sodium-losing nephropathy--a case report and review.
    Postgraduate medical journal, 1981, Volume: 57, Issue:663

    The third case in the literature of sodium-losing renal disease due to obstruction is presented. The experimental evidence and limited clinical experience is reviewed which suggests that the sodium loss is due to an inappropriate response in the adaptive processes that are initiated by the loss of functioning nephrons. The immediate treatment is by replacement of sodium but in the long term the condition may be reversed by very cautious reduction in sodium intake. Definitive treatment may be indicated where obstruction is the cause and consequently this should be sought in all cases of salt-losing renal disease.

    Topics: Bicarbonates; Female; Humans; Kidney Failure, Chronic; Kidney Tubules; Middle Aged; Sodium; Sodium Bicarbonate; Sodium Chloride; Ureteral Obstruction

1981