sodium-bicarbonate has been researched along with Subarachnoid-Hemorrhage* in 3 studies
3 other study(ies) available for sodium-bicarbonate and Subarachnoid-Hemorrhage
| Article | Year |
|---|---|
Hemorrhagic Encephalopathy From Acute Baking Soda Ingestion.
Baking soda is a readily available household product composed of sodium bicarbonate. It can be used as a home remedy to treat dyspepsia. If used in excessive amounts, baking soda has the potential to cause a variety of serious metabolic abnormalities. We believe this is the first reported case of hemorrhagic encephalopathy induced by baking soda ingestion. Healthcare providers should be aware of the dangers of baking soda misuse and the associated adverse effects. Topics: Adult; Brain Diseases; Emergency Service, Hospital; Humans; Male; Sodium Bicarbonate; Subarachnoid Hemorrhage | 2016 |
Metformin-associated lactic acidosis following contrast media-induced nephrotoxicity.
Topics: Acidosis, Lactic; Angiography, Digital Subtraction; Blood Gas Analysis; Contrast Media; Diabetes Mellitus, Type 2; Fatal Outcome; Heart Arrest; Humans; Hypoglycemic Agents; Intubation, Intratracheal; Iohexol; Male; Metformin; Middle Aged; Postoperative Complications; Radiographic Image Enhancement; Renal Insufficiency; Respiration, Artificial; Respiratory Distress Syndrome; Sodium Bicarbonate; Subarachnoid Hemorrhage; Tomography, X-Ray Computed | 2008 |
Anaerobic change of bloody CSF in subarachnoid haemorrhage. Its relation to cerebral vasospasm.
In our in vitro study of subarachnoid haemorrhage, the anaerobic incubation of CSF-blood mixture led to marked fall in the pH value thereof, which suggested to us that intracranial focal acidosis may play some role in the pathogenesis of cerebral vasospasm or disturbance of consciousness after the haemorrhage. To test this hypothesis, we treated 16 clinical cases of such disorders with carotid injection of 7% sodium bicarbonate solution. The treatment resulted in considerable improvement of the disturbance of consciousness by dilating the cerebral peripheral arteries; this we could observe angiographically 15 minutes after carotid injection. We found no morphological changes, however, in the spastic vessel itself up to 30 minutes after the injection. Given the result both of this study and of our previous experiments, we offer the hypothesis that the synthesizing process of thromboxane A2 and intracranial focal acidosis might play an important role in the pathogenesis of cerebral vasospasm. Topics: Adolescent; Adult; Aged; Anaerobiosis; Bicarbonates; Child, Preschool; Consciousness Disorders; Female; Humans; Intracranial Aneurysm; Ischemic Attack, Transient; Male; Middle Aged; Postoperative Complications; Rupture, Spontaneous; Sodium Bicarbonate; Subarachnoid Hemorrhage | 1981 |