sodium-bicarbonate and Hyperuricemia

sodium-bicarbonate has been researched along with Hyperuricemia* in 2 studies

Reviews

1 review(s) available for sodium-bicarbonate and Hyperuricemia

Article	Year
[Gouty kidney]. Nihon rinsho. Japanese journal of clinical medicine, 2003, Volume: 61 Suppl 1 Topics: Animals; Diet; Gout; Humans; Hyperuricemia; Kidney Diseases; Sodium Bicarbonate; Tartrates; Uric Acid; Uricosuric Agents	2003

Other Studies

1 other study(ies) available for sodium-bicarbonate and Hyperuricemia

Article	Year
Partial hypoxanthine-guanine phosphoribosyltransferase deficiency presenting as acute renal failure. Pediatric nephrology (Berlin, Germany), 2005, Volume: 20, Issue:12 Hyperuricemia and secondary urate nephropathy are uncommon in the paediatric setting outside of tumour lysis syndrome. We describe the case of a 12-year-old boy who presented at 3 years of age with acute renal failure. The cause of this remained unknown until the development of uric acid renal calculi 9 years later. This, and the availability of the previously unknown family history, provided the subsequent diagnosis of partial hypoxanthine-guanine phosphoribosyltransferase (HPRT) deficiency. Detailed family history is important for early detection of this heterogeneous group of disorders. Early treatment may minimise long-term renal morbidity and mortality from renal insufficiency. Topics: Acute Kidney Injury; Age of Onset; Allopurinol; Buffers; Cells, Cultured; Child; Enzyme Inhibitors; Erythrocytes; Female; Fibroblasts; Follow-Up Studies; Genetic Linkage; Greece; Heterozygote; Humans; Hyperuricemia; Hypoxanthine Phosphoribosyltransferase; Male; Pedigree; Skin; Sodium Bicarbonate; Time Factors; Treatment Outcome; Ultrasonography; Urinary Calculi	2005

Article

Year

Partial hypoxanthine-guanine phosphoribosyltransferase deficiency presenting as acute renal failure.

Pediatric nephrology (Berlin, Germany), 2005, Volume: 20, Issue:12

Hyperuricemia and secondary urate nephropathy are uncommon in the paediatric setting outside of tumour lysis syndrome. We describe the case of a 12-year-old boy who presented at 3 years of age with acute renal failure. The cause of this remained unknown until the development of uric acid renal calculi 9 years later. This, and the availability of the previously unknown family history, provided the subsequent diagnosis of partial hypoxanthine-guanine phosphoribosyltransferase (HPRT) deficiency. Detailed family history is important for early detection of this heterogeneous group of disorders. Early treatment may minimise long-term renal morbidity and mortality from renal insufficiency.

Topics: Acute Kidney Injury; Age of Onset; Allopurinol; Buffers; Cells, Cultured; Child; Enzyme Inhibitors; Erythrocytes; Female; Fibroblasts; Follow-Up Studies; Genetic Linkage; Greece; Heterozygote; Humans; Hyperuricemia; Hypoxanthine Phosphoribosyltransferase; Male; Pedigree; Skin; Sodium Bicarbonate; Time Factors; Treatment Outcome; Ultrasonography; Urinary Calculi

2005