sodium-bicarbonate and Colitis--Ulcerative

sodium-bicarbonate has been researched along with Colitis--Ulcerative* in 2 studies

Other Studies

2 other study(ies) available for sodium-bicarbonate and Colitis--Ulcerative

ArticleYear
[A case of drug induced urolithiasis composed of acetyl sulphapyridine associated with ulcerative colitis].
    Nihon Hinyokika Gakkai zasshi. The japanese journal of urology, 1999, Volume: 90, Issue:3

    A 26-year-old female visited our hospital complaining left flank pain and macroscopic hematuria. She had been suffering ulcerative colitis and administered salazosulphapyridine and predonisolone from 17-year-old. Intravenous urography showed radiolucent multiple stones in the left renal pelvis. Three sessions of extracorporeal shock wave lithotripsy were performed after ureteral stenting. Although disintegration and discharge of the stones were satisfactory, bladder stone induced by ureteral stent was complicated. The extracted bladder stone showed a yellowish brown color and the surface was granular shape. Composition of the stone was acetyl sulphapyridine which was a metabolite of salazosulphapyridine. After maintenance of the urinary pH ranges between 6.5 and 7.5 by medication of sodium bicarbonate, the patient remains free of stone for 3 years. Drug induced urolithiasis originated from salazosulphapyridine is extremely rare. Satisfactory oral fluid intake and urinary alkalization are important for prevention of sulpha drugs calculi of urinary tract.

    Topics: Adult; Anti-Inflammatory Agents, Non-Steroidal; Colitis, Ulcerative; Female; Gastrointestinal Agents; Humans; Lithotripsy; Sodium Bicarbonate; Sulfapyridine; Sulfasalazine; Urinary Calculi

1999
Prevention of uric acid stone formation by sodium bicarbonate in an ileostomy patient--a case report.
    The Japanese journal of surgery, 1988, Volume: 18, Issue:4

    A 38 year old female underwent a proctocolectomy and ileostomy for ulcerative colitis in February, 1974. For 8 year post-operatively, she excreted innumerable renal stones, mainly composed of uric acid. Her urine was highly acidic and hyperuricosuric with a low concentration of sodium. Sodium bicarbonate 4 gm/day, t.i.d., was started in October 1985, after which her renal stone excretion completely ceased (up until March, 1987), except for one incidence of stone excretion when she discontinued therapy for a week. During the sodium bicarbonate therapy, her urinary pH and Na concentration were elevated. Furthermore, sodium bicarbonate significantly elevated the urinary pH and Na concentration of other ileostomy patients. Thus, sodium bicarbonate could be used for the possible prophylaxis of uric acid formation in selected ileostomy patients.

    Topics: Adult; Bicarbonates; Colitis, Ulcerative; Female; Humans; Ileostomy; Kidney Calculi; Sodium; Sodium Bicarbonate; Uric Acid

1988