Page last updated: 2024-08-22

sodium arsenite and Urinary Bladder Neoplasms

sodium arsenite has been researched along with Urinary Bladder Neoplasms in 8 studies

Research

Studies (8)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (37.50)29.6817
2010's3 (37.50)24.3611
2020's2 (25.00)2.80

Authors

AuthorsStudies
Damoiseaux, R; Gimzewski, JK; Odunola, OA; Olugbami, JO1
Flora, SJS; Sathua, K; Srivastava, S1
Chan, MW; Chen, CJ; Lee, TC; Pu, YS; Wang, HH; Wu, MM1
Black, KE; DiRenzo, J; Fei, DL; Gosse, JA; Hamilton, JW; Karagas, MR; Kozul, CD; Li, H; Martin, KA; Robbins, DJ; Singh, S; Wang, B1
Arnold, LL; Black, M; Choe, KY; Clewell, HJ; Efremenko, A; Gentry, PR; Gill, G; McKim, JM; Pluta, L; Thomas, RS; Wilga, P; Yager, JW1
Hou, MF; Lee, EY; Su, JH; Yang, FW; Yuan, SS; Zhao, S1
Luster, MI; Simeonova, PP1
Cebrián, ME; Córdova, E; Del Razo, LM; Garrido, E; Hernández-Zavala, A1

Reviews

1 review(s) available for sodium arsenite and Urinary Bladder Neoplasms

ArticleYear
Arsenic and urinary bladder cell proliferation.
    Toxicology and applied pharmacology, 2004, Aug-01, Volume: 198, Issue:3

    Topics: Animals; Arsenites; Carcinoma, Transitional Cell; Enzyme Activation; Enzyme Inhibitors; Mice; Mitogen-Activated Protein Kinase Kinases; Sodium Compounds; Urinary Bladder; Urinary Bladder Neoplasms

2004

Other Studies

7 other study(ies) available for sodium arsenite and Urinary Bladder Neoplasms

ArticleYear
Mitigation of aflatoxin B1- and sodium arsenite-induced cytotoxicities in HUC-PC urinary bladder cells by curcumin and Khaya senegalensis.
    Journal of basic and clinical physiology and pharmacology, 2020, Apr-22, Volume: 31, Issue:4

    Topics: Aflatoxin B1; Antineoplastic Agents; Arsenites; Cell Survival; Curcumin; Enzyme Inhibitors; Humans; Meliaceae; Plant Extracts; Primary Cell Culture; Sodium Compounds; Urinary Bladder; Urinary Bladder Neoplasms

2020
MiADMSA ameliorate arsenic induced urinary bladder carcinogenesis in vivo and in vitro.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2020, Volume: 128

    Topics: 8-Hydroxy-2'-Deoxyguanosine; Animals; Anticarcinogenic Agents; Arsenites; Cacodylic Acid; Carcinoma, Transitional Cell; Cell Line, Tumor; Cell Transformation, Neoplastic; Chelating Agents; DNA Damage; Humans; Male; Matrix Metalloproteinase 9; Nitrosative Stress; Rats, Sprague-Dawley; Sodium Compounds; Succimer; Survivin; Urinary Bladder Neoplasms

2020
Long-term low-dose exposure of human urothelial cells to sodium arsenite activates lipocalin-2 via promoter hypomethylation.
    Archives of toxicology, 2014, Volume: 88, Issue:8

    Topics: Acute-Phase Proteins; Arsenites; Azacitidine; Cell Culture Techniques; Cell Line; Cell Proliferation; Chromatin Immunoprecipitation; Culture Media, Serum-Free; Decitabine; DNA Methylation; Dose-Response Relationship, Drug; Environmental Pollutants; Gene Silencing; Genome-Wide Association Study; Humans; Lipocalin-2; Lipocalins; Mutagenesis, Site-Directed; NF-kappa B; Promoter Regions, Genetic; Protein Binding; Proto-Oncogene Proteins; Sodium Compounds; Time Factors; Urinary Bladder Neoplasms; Urothelium

2014
Activation of Hedgehog signaling by the environmental toxicant arsenic may contribute to the etiology of arsenic-induced tumors.
    Cancer research, 2010, Mar-01, Volume: 70, Issue:5

    Topics: Animals; Arsenites; Cattle; Endothelial Cells; Environmental Exposure; Hazardous Substances; Hedgehog Proteins; Humans; Male; Mice; Mice, Inbred C57BL; NIH 3T3 Cells; Signal Transduction; Sodium Compounds; Urinary Bladder Neoplasms

2010
Evaluation of gene expression changes in human primary uroepithelial cells following 24-hr exposures to inorganic arsenic and its methylated metabolites.
    Environmental and molecular mutagenesis, 2013, Volume: 54, Issue:2

    Topics: Adult; Arsenites; Cell Culture Techniques; Cell Survival; Cells, Cultured; Dose-Response Relationship, Drug; Epithelial Cells; Female; Glutathione; Humans; Male; Methylation; Middle Aged; Oligonucleotide Array Sequence Analysis; Risk Assessment; Sodium Compounds; Time Factors; Transcriptome; Urinary Bladder Neoplasms; Urothelium

2013
Arsenic-induced Mre11 phosphorylation is cell cycle-dependent and defective in NBS cells.
    DNA repair, 2002, Feb-28, Volume: 1, Issue:2

    Topics: Arsenites; Carcinoma, Transitional Cell; Cell Cycle; Cell Cycle Proteins; Cell Line; Cell Nucleus; Cells, Cultured; DNA; DNA Damage; DNA Repair; DNA-Binding Proteins; Endodeoxyribonucleases; Exodeoxyribonucleases; Fibroblasts; Fluorescent Antibody Technique; Humans; Immunoblotting; Nuclear Proteins; Phosphorylation; Precipitin Tests; Saccharomyces cerevisiae Proteins; Sodium Compounds; Tumor Cells, Cultured; Urinary Bladder Neoplasms

2002
Effects of arsenite on cell cycle progression in a human bladder cancer cell line.
    Toxicology, 2005, Feb-01, Volume: 207, Issue:1

    Topics: Arsenites; Blotting, Western; Cell Cycle; Cell Cycle Proteins; Cell Line, Tumor; Cell Survival; Cyclin-Dependent Kinase Inhibitor p21; Dose-Response Relationship, Drug; Humans; S Phase; Sodium Compounds; Time Factors; Tumor Suppressor Protein p53; Urinary Bladder Neoplasms

2005