Page last updated: 2024-08-23

simvastatin and Fibrosarcoma

simvastatin has been researched along with Fibrosarcoma in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Kim, YS; Lee, SK	1
Chaisuparat, R; Thunyakitpisal, PD	1

Other Studies

2 other study(ies) available for simvastatin and Fibrosarcoma

Article	Year
Phosphorylation of eIF2α attenuates statin-induced apoptosis by inhibiting the stabilization and translocation of p53 to the mitochondria. International journal of oncology, 2013, Volume: 42, Issue:3 Topics: Animals; Anthracenes; Anticholesteremic Agents; Apoptosis; bcl-2-Associated X Protein; Cell Line, Tumor; Cell Survival; Cinnamates; eIF-2 Kinase; Endoplasmic Reticulum; Endoplasmic Reticulum Chaperone BiP; Endoplasmic Reticulum Stress; Eukaryotic Initiation Factor-2; Fibrosarcoma; Heat-Shock Proteins; Hydroxymethylglutaryl-CoA Reductase Inhibitors; JNK Mitogen-Activated Protein Kinases; Membrane Potential, Mitochondrial; Mevalonic Acid; Mice; Mitochondria; Phosphorylation; Polyisoprenyl Phosphates; Protein Transport; RNA Interference; RNA, Small Interfering; Sesquiterpenes; Signal Transduction; Simvastatin; Thiourea; Transcription Factor CHOP; Tumor Suppressor Protein p53	2013
Simvastatin, an HMG-CoA reductase inhibitor, reduced the expression of matrix metalloproteinase-9 (Gelatinase B) in osteoblastic cells and HT1080 fibrosarcoma cells. Journal of pharmacological sciences, 2004, Volume: 94, Issue:4 Topics: Animals; Animals, Newborn; Blotting, Western; Cell Line, Tumor; Cells, Cultured; Culture Media, Conditioned; Dose-Response Relationship, Drug; Fibrosarcoma; Gene Expression Regulation, Neoplastic; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Matrix Metalloproteinase 9; Osteoblasts; Osteosarcoma; Parietal Bone; Rats; Reverse Transcriptase Polymerase Chain Reaction; Simvastatin; Time Factors; Tumor Necrosis Factor-alpha	2004

Article

Year

Phosphorylation of eIF2α attenuates statin-induced apoptosis by inhibiting the stabilization and translocation of p53 to the mitochondria.

International journal of oncology, 2013, Volume: 42, Issue:3

Topics: Animals; Anthracenes; Anticholesteremic Agents; Apoptosis; bcl-2-Associated X Protein; Cell Line, Tumor; Cell Survival; Cinnamates; eIF-2 Kinase; Endoplasmic Reticulum; Endoplasmic Reticulum Chaperone BiP; Endoplasmic Reticulum Stress; Eukaryotic Initiation Factor-2; Fibrosarcoma; Heat-Shock Proteins; Hydroxymethylglutaryl-CoA Reductase Inhibitors; JNK Mitogen-Activated Protein Kinases; Membrane Potential, Mitochondrial; Mevalonic Acid; Mice; Mitochondria; Phosphorylation; Polyisoprenyl Phosphates; Protein Transport; RNA Interference; RNA, Small Interfering; Sesquiterpenes; Signal Transduction; Simvastatin; Thiourea; Transcription Factor CHOP; Tumor Suppressor Protein p53

2013

Simvastatin, an HMG-CoA reductase inhibitor, reduced the expression of matrix metalloproteinase-9 (Gelatinase B) in osteoblastic cells and HT1080 fibrosarcoma cells.

Journal of pharmacological sciences, 2004, Volume: 94, Issue:4

Topics: Animals; Animals, Newborn; Blotting, Western; Cell Line, Tumor; Cells, Cultured; Culture Media, Conditioned; Dose-Response Relationship, Drug; Fibrosarcoma; Gene Expression Regulation, Neoplastic; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Matrix Metalloproteinase 9; Osteoblasts; Osteosarcoma; Parietal Bone; Rats; Reverse Transcriptase Polymerase Chain Reaction; Simvastatin; Time Factors; Tumor Necrosis Factor-alpha

2004