Page last updated: 2024-08-24

simendan and Hypertrophy, Left Ventricular

simendan has been researched along with Hypertrophy, Left Ventricular in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Andersson, KB; Christensen, G; Golz, S; Hillestad, V; Knorr, A; Kramer, F1
Bhavsar, R; Frederiksen, CA; Jakobsen, CJ; Juhl-Olsen, P; Klaaborg, KE; Rasmussen, LA; Sloth, E1
Bech-Hanssen, O; Houltz, E; Jörgensen, K; Ricksten, SE1

Trials

2 trial(s) available for simendan and Hypertrophy, Left Ventricular

ArticleYear
Effects of levosimendan in patients with left ventricular hypertrophy undergoing aortic valve replacement.
    Acta anaesthesiologica Scandinavica, 2015, Volume: 59, Issue:1

    Topics: Aged; Aged, 80 and over; Aortic Valve; Double-Blind Method; Echocardiography; Female; Heart Valve Prosthesis Implantation; Hemodynamics; Humans; Hydrazones; Hypertrophy, Left Ventricular; Male; Middle Aged; Prospective Studies; Pyridazines; Simendan

2015
Effects of levosimendan on left ventricular relaxation and early filling at maintained preload and afterload conditions after aortic valve replacement for aortic stenosis.
    Circulation, 2008, Feb-26, Volume: 117, Issue:8

    Topics: Aged; Aged, 80 and over; Aortic Valve Stenosis; Blood Pressure; Cardiotonic Agents; Diastole; Echocardiography; Female; Heart Valve Prosthesis Implantation; Humans; Hydrazones; Hypertrophy, Left Ventricular; Male; Mitral Valve; Pyridazines; Simendan; Stroke Volume; Treatment Outcome; Vascular Resistance; Ventricular Function, Left

2008

Other Studies

1 other study(ies) available for simendan and Hypertrophy, Left Ventricular

ArticleYear
Long-term levosimendan treatment improves systolic function and myocardial relaxation in mice with cardiomyocyte-specific disruption of the Serca2 gene.
    Journal of applied physiology (Bethesda, Md. : 1985), 2013, Volume: 115, Issue:10

    Topics: Animals; Calcium Signaling; Cardiotonic Agents; Diastole; Disease Models, Animal; Extracellular Matrix Proteins; Fibrosis; Gene Expression Regulation; Heart Failure; Hydrazones; Hypertrophy, Left Ventricular; Mice; Mice, Knockout; Myocytes, Cardiac; Pyridazines; Recovery of Function; Sarcoplasmic Reticulum Calcium-Transporting ATPases; Simendan; Stroke Volume; Systole; Time Factors; Ventricular Dysfunction, Left; Ventricular Function, Left; Ventricular Pressure

2013