sevoflurane has been researched along with Infarction, Middle Cerebral Artery in 23 studies
Sevoflurane: A non-explosive inhalation anesthetic used in the induction and maintenance of general anesthesia. It does not cause respiratory irritation and may also prevent PLATELET AGGREGATION.
sevoflurane : An ether compound having fluoromethyl and 1,1,1,3,3,3-hexafluoroisopropyl as the two alkyl groups.
Infarction, Middle Cerebral Artery: NECROSIS occurring in the MIDDLE CEREBRAL ARTERY distribution system which brings blood to the entire lateral aspects of each CEREBRAL HEMISPHERE. Clinical signs include impaired cognition; APHASIA; AGRAPHIA; weak and numbness in the face and arms, contralaterally or bilaterally depending on the infarction.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Sevoflurane (sevo) has been reported to be an effective neuroprotective agent in cerebral ischemia/reperfusion injury (CIRI)." | 7.91 | Sevoflurane prevents miR-181a-induced cerebral ischemia/reperfusion injury. ( Ai, Y; Shan, Z; Zhang, Y; Zhao, Y, 2019) |
| "We aim to investigate the effects of sevoflurane on the ATPase activity of the hippocampal neurons in rats with cerebral ischemia-reperfusion injury (IRI) via the cyclic adenosine monophosphate (cAMP) and protein kinase A (PKA) signaling pathway." | 7.88 | Effect of sevoflurane on the ATPase activity of hippocampal neurons in a rat model of cerebral ischemia-reperfusion injury via the cAMP-PKA signaling pathway. ( Cheng, AB; Gao, XZ; Liu, TJ; Tan, ZB; Wang, JJ; Zhang, JC; Zhang, PP; Zhang, SB, 2018) |
| "Potential additive effects of estrogens and sevoflurane against cerebral infarction after transient or permanent middle cerebral artery occlusion (MCAO) have not been addressed." | 7.83 | Protective effects of estrogen combined with sevoflurane in an experimental model of cerebral infarction and focal cerebral ischemia-reperfusion injury. ( Liu, JJ; Pan, SY, 2016) |
| "Postconditioning with sevoflurane has been shown to protect against focal cerebral ischemia and reperfusion injury." | 7.81 | Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore. ( Liu, XZ; Wang, JK; Wu, HF; Yang, B; Zhou, H, 2015) |
| " The purpose of this study was to determine if sevoflurane preconditioning offers durable neuroprotection against cerebral ischemia." | 7.75 | Sevoflurane preconditioning against focal cerebral ischemia: inhibition of apoptosis in the face of transient improvement of neurological outcome. ( Bruder, NJ; Codaccioni, JL; Guillet, BA; Moubarik, C; Pisano, PS; Velly, LJ, 2009) |
| "Sevoflurane (sevo) has been reported to be an effective neuroprotective agent in cerebral ischemia/reperfusion injury (CIRI)." | 3.91 | Sevoflurane prevents miR-181a-induced cerebral ischemia/reperfusion injury. ( Ai, Y; Shan, Z; Zhang, Y; Zhao, Y, 2019) |
| "We aim to investigate the effects of sevoflurane on the ATPase activity of the hippocampal neurons in rats with cerebral ischemia-reperfusion injury (IRI) via the cyclic adenosine monophosphate (cAMP) and protein kinase A (PKA) signaling pathway." | 3.88 | Effect of sevoflurane on the ATPase activity of hippocampal neurons in a rat model of cerebral ischemia-reperfusion injury via the cAMP-PKA signaling pathway. ( Cheng, AB; Gao, XZ; Liu, TJ; Tan, ZB; Wang, JJ; Zhang, JC; Zhang, PP; Zhang, SB, 2018) |
| "Potential additive effects of estrogens and sevoflurane against cerebral infarction after transient or permanent middle cerebral artery occlusion (MCAO) have not been addressed." | 3.83 | Protective effects of estrogen combined with sevoflurane in an experimental model of cerebral infarction and focal cerebral ischemia-reperfusion injury. ( Liu, JJ; Pan, SY, 2016) |
| "Postconditioning with sevoflurane has been shown to protect against focal cerebral ischemia and reperfusion injury." | 3.81 | Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore. ( Liu, XZ; Wang, JK; Wu, HF; Yang, B; Zhou, H, 2015) |
| "Sevoflurane can produce delayed protection against cerebral ischemia-reperfusion injury by down-regulating TNF-alpha, IL-1beta protein, and mRNA expression." | 3.75 | [Sevoflurane preconditioning induced delayed neuroprotection against focal cerebral ischemia in rats]. ( Guo, Q; Pan, Y; Shi, M; Wang, E; Ye, Z, 2009) |
| " The purpose of this study was to determine if sevoflurane preconditioning offers durable neuroprotection against cerebral ischemia." | 3.75 | Sevoflurane preconditioning against focal cerebral ischemia: inhibition of apoptosis in the face of transient improvement of neurological outcome. ( Bruder, NJ; Codaccioni, JL; Guillet, BA; Moubarik, C; Pisano, PS; Velly, LJ, 2009) |
| "The rat model of middle cerebral artery occlusion (MCAO) was established by thread embolization." | 1.72 | Sevoflurane up-regulates miR-7a to protect against ischemic brain injury in rats by down-regulating ATG7 and reducing neuronal autophagy. ( Lin, L; Tan, J; Wu, Z; Yuan, W; Zhang, W, 2022) |
| "The rat middle cerebral artery occlusion model was established." | 1.62 | Sevoflurane improves nerve regeneration and repair of neurological deficit in brain damage rats via microRNA-490-5p/CDK1 axis. ( Ding, L; Ke, H; Lu, B; Ning, J; Wang, Q, 2021) |
| "After SPC, mice were subjected to middle cerebral artery occlusion (MCAO)." | 1.56 | Role of Metallothionein-1 and Metallothionein-2 in the Neuroprotective Mechanism of Sevoflurane Preconditioning in Mice. ( Liu, J; Long, Y; Luo, J; Mei, X; Tan, S; Tang, Y; Wang, Y, 2020) |
| "Sevoflurane treatment also caused increased phosphorylation of p38 MAPK at 24 and 72 h after reperfusion." | 1.38 | Delayed neuroprotection induced by sevoflurane via opening mitochondrial ATP-sensitive potassium channels and p38 MAPK phosphorylation. ( Guo, Q; Wang, E; Wang, N; Xia, P; Ye, Z; Yuan, Y, 2012) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (13.04) | 29.6817 |
| 2010's | 13 (56.52) | 24.3611 |
| 2020's | 7 (30.43) | 2.80 |
| Authors | Studies |
|---|---|
| Song, J | 1 |
| He, K | 1 |
| Yang, L | 1 |
| Shen, J | 1 |
| Wu, Z | 1 |
| Tan, J | 1 |
| Lin, L | 1 |
| Zhang, W | 1 |
| Yuan, W | 1 |
| Liu, J | 1 |
| Tan, S | 1 |
| Wang, Y | 1 |
| Luo, J | 1 |
| Long, Y | 1 |
| Mei, X | 1 |
| Tang, Y | 1 |
| Deng, Y | 1 |
| Shi, C | 1 |
| Gu, Y | 1 |
| Yang, N | 1 |
| Xu, M | 1 |
| Xu, T | 1 |
| Guo, X | 1 |
| Yu, F | 1 |
| Tong, LJ | 1 |
| Cai, DS | 1 |
| Ding, L | 1 |
| Ning, J | 1 |
| Wang, Q | 1 |
| Lu, B | 1 |
| Ke, H | 1 |
| Cai, M | 1 |
| Sun, S | 1 |
| Wang, J | 1 |
| Dong, B | 1 |
| Yang, Q | 1 |
| Tian, L | 2 |
| Dong, H | 2 |
| Wang, S | 1 |
| Hou, W | 1 |
| Liu, TJ | 1 |
| Zhang, JC | 1 |
| Gao, XZ | 1 |
| Tan, ZB | 1 |
| Wang, JJ | 1 |
| Zhang, PP | 1 |
| Cheng, AB | 1 |
| Zhang, SB | 1 |
| Zhang, Y | 4 |
| Shan, Z | 1 |
| Zhao, Y | 1 |
| Ai, Y | 1 |
| Wang, JK | 1 |
| Wu, HF | 1 |
| Zhou, H | 1 |
| Yang, B | 1 |
| Liu, XZ | 1 |
| Li, B | 1 |
| Sun, J | 1 |
| Lv, G | 1 |
| Yu, Y | 2 |
| Wang, G | 1 |
| Xie, K | 1 |
| Jiao, Y | 1 |
| Chen, Y | 1 |
| Nie, H | 1 |
| Tong, L | 1 |
| Deng, J | 1 |
| Xiong, L | 1 |
| Wang, H | 1 |
| Shi, H | 1 |
| Yu, Q | 1 |
| Chen, J | 2 |
| Zhang, F | 1 |
| Gao, Y | 1 |
| Liu, JJ | 1 |
| Pan, SY | 1 |
| Ye, Z | 3 |
| Guo, Q | 2 |
| Wang, E | 3 |
| Shi, M | 1 |
| Pan, Y | 1 |
| Codaccioni, JL | 1 |
| Velly, LJ | 1 |
| Moubarik, C | 1 |
| Bruder, NJ | 1 |
| Pisano, PS | 1 |
| Guillet, BA | 1 |
| Wang, N | 1 |
| Xia, P | 1 |
| Yuan, Y | 1 |
| Zhang, FG | 1 |
| Meng, C | 2 |
| Tian, SY | 2 |
| Wang, YX | 2 |
| Zhao, W | 1 |
| Zhang, XS | 1 |
| Liang, Y | 2 |
| Zhang, SD | 1 |
| Xing, YJ | 1 |
| Gauberti, M | 1 |
| Obiang, P | 1 |
| Guedin, P | 1 |
| Balossier, A | 1 |
| Gakuba, C | 1 |
| Diependaele, AS | 1 |
| Chazalviel, L | 1 |
| Vivien, D | 1 |
| Young, AR | 1 |
| Agin, V | 1 |
| Orset, C | 1 |
| Huang, YM | 1 |
| Zuo, ZY | 1 |
| Guo, QL | 1 |
| Bedirli, N | 1 |
| Bagriacik, EU | 1 |
| Emmez, H | 1 |
| Yilmaz, G | 1 |
| Unal, Y | 1 |
| Ozkose, Z | 1 |
| Liu, HG | 1 |
| Hua, Z | 1 |
| Ma, YP | 1 |
| Wang, L | 1 |
| Wang, WS | 1 |
| Fernández Sánchez, LJ | 1 |
| Pérez González, R | 1 |
| Guasch Arévalo, E | 1 |
| Martín Reyes, R | 1 |
| Gilsanz Rodríguez, F | 1 |
23 other studies available for sevoflurane and Infarction, Middle Cerebral Artery
| Article | Year |
|---|---|
Sevoflurane protects mice from cerebral ischaemic injury by regulating microRNA-203-3p/HDAC4/Bcl-2 axis.
Topics: Animals; Apoptosis; Histone Deacetylases; Infarction, Middle Cerebral Artery; Inflammation; Mice; Mi | 2022 |
Sevoflurane up-regulates miR-7a to protect against ischemic brain injury in rats by down-regulating ATG7 and reducing neuronal autophagy.
Topics: Animals; Autophagy; Autophagy-Related Protein 7; Brain Injuries; Down-Regulation; Infarction, Middle | 2022 |
Role of Metallothionein-1 and Metallothionein-2 in the Neuroprotective Mechanism of Sevoflurane Preconditioning in Mice.
Topics: Animals; Apoptosis; Cells, Cultured; Glucose; Infarction, Middle Cerebral Artery; Metallothionein; M | 2020 |
A study of optimal concentration range and time window of sevoflurane preconditioning for brain protection in MCAO rats.
Topics: Animals; Brain; Brain Ischemia; Dose-Response Relationship, Drug; Infarction, Middle Cerebral Artery | 2020 |
Sevoflurane inhibits neuronal apoptosis and expressions of HIF-1 and HSP70 in brain tissues of rats with cerebral ischemia/reperfusion injury.
Topics: Animals; Apoptosis; Brain; Disease Models, Animal; HSP70 Heat-Shock Proteins; Hypoxia-Inducible Fact | 2020 |
Sevoflurane improves nerve regeneration and repair of neurological deficit in brain damage rats via microRNA-490-5p/CDK1 axis.
Topics: Animals; CDC2 Protein Kinase; Infarction, Middle Cerebral Artery; Male; MicroRNAs; Nerve Regeneratio | 2021 |
Sevoflurane preconditioning protects experimental ischemic stroke by enhancing anti-inflammatory microglia/macrophages phenotype polarization through GSK-3β/Nrf2 pathway.
Topics: Anesthetics, Inhalation; Animals; Glucose; Glycogen Synthase Kinase 3 beta; Hypoxia, Brain; Infarcti | 2021 |
Effect of sevoflurane on the ATPase activity of hippocampal neurons in a rat model of cerebral ischemia-reperfusion injury via the cAMP-PKA signaling pathway.
Topics: Animals; Brain-Derived Neurotrophic Factor; Colforsin; CREB-Binding Protein; Cyclic AMP; Cyclic AMP- | 2018 |
Sevoflurane prevents miR-181a-induced cerebral ischemia/reperfusion injury.
Topics: 3' Untranslated Regions; Animals; Antagomirs; Apoptosis; Base Sequence; Down-Regulation; Infarction, | 2019 |
Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore.
Topics: Animals; Apoptosis; Brain Edema; Brain Ischemia; Disease Models, Animal; Infarction, Middle Cerebral | 2015 |
Sevoflurane postconditioning attenuates cerebral ischemia-reperfusion injury via protein kinase B/nuclear factor-erythroid 2-related factor 2 pathway activation.
Topics: Animals; Brain Infarction; Disease Models, Animal; Electrophoretic Mobility Shift Assay; Enzyme Inhi | 2014 |
Sevoflurane preconditioning-induced neuroprotection is associated with Akt activation via carboxy-terminal modulator protein inhibition.
Topics: Activation, Metabolic; Anesthetics, Inhalation; Animals; Behavior, Animal; Carrier Proteins; Glycoge | 2015 |
Sevoflurane Preconditioning Confers Neuroprotection via Anti-apoptosis Effects.
Topics: Anesthetics, Inhalation; Animals; Apoptosis; bcl-X Protein; Blotting, Western; Brain; Fluorescent An | 2016 |
Protective effects of estrogen combined with sevoflurane in an experimental model of cerebral infarction and focal cerebral ischemia-reperfusion injury.
Topics: Animals; Brain Ischemia; Cerebral Infarction; Estrogens; Female; Infarction, Middle Cerebral Artery; | 2016 |
[Sevoflurane preconditioning induced delayed neuroprotection against focal cerebral ischemia in rats].
Topics: Animals; Hypoxia-Ischemia, Brain; Infarction, Middle Cerebral Artery; Interleukin-1beta; Ischemic Pr | 2009 |
Sevoflurane preconditioning against focal cerebral ischemia: inhibition of apoptosis in the face of transient improvement of neurological outcome.
Topics: Anesthetics, Inhalation; Animals; Apoptosis; Behavior, Animal; Brain Ischemia; DNA Fragmentation; In | 2009 |
Delayed neuroprotection induced by sevoflurane via opening mitochondrial ATP-sensitive potassium channels and p38 MAPK phosphorylation.
Topics: Analysis of Variance; Animals; Anti-Arrhythmia Agents; Brain Infarction; Cell Count; Decanoic Acids; | 2012 |
Inhibition of sevoflurane postconditioning against cerebral ischemia reperfusion-induced oxidative injury in rats.
Topics: Animals; Brain; Glutathione; Infarction, Middle Cerebral Artery; Inflammation Mediators; Interleukin | 2011 |
Thrombotic stroke in the anesthetized monkey (Macaca mulatta): characterization by MRI--a pilot study.
Topics: Anesthesia; Anesthetics, Dissociative; Anesthetics, Inhalation; Animals; Atracurium; Craniotomy; Dif | 2012 |
Sevoflurane-induced delayed neuroprotection involves mitoK(ATP) channel opening and PKC ε activation.
Topics: Animals; Enzyme Activation; Infarction, Middle Cerebral Artery; Ion Channel Gating; Ischemic Precond | 2012 |
Sevoflurane and isoflurane preconditioning provides neuroprotection by inhibition of apoptosis-related mRNA expression in a rat model of focal cerebral ischemia.
Topics: Anesthetics, Inhalation; Animals; Antioxidants; Apoptosis; Apoptosis Regulatory Proteins; Brain Edem | 2012 |
Effect of Sevoflurane postconditioning on gene expression in brain tissue of the middle cerebral artery occlusion rat model.
Topics: Animals; bcl-2-Associated X Protein; Brain; Brain Ischemia; Caspase 3; Catalase; Disease Models, Ani | 2012 |
[Perioperative treatment of a pregnant woman with recent cerebral infarction secondary to noncompaction cardiomyopathy].
Topics: Adrenergic beta-Antagonists; Androstanols; Anesthesia, General; Anesthesia, Inhalation; Anesthesia, | 2006 |