sevoflurane has been researched along with Cerebral Infarction in 6 studies
Sevoflurane: A non-explosive inhalation anesthetic used in the induction and maintenance of general anesthesia. It does not cause respiratory irritation and may also prevent PLATELET AGGREGATION.
sevoflurane : An ether compound having fluoromethyl and 1,1,1,3,3,3-hexafluoroisopropyl as the two alkyl groups.
Cerebral Infarction: The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).
| Excerpt | Relevance | Reference |
|---|---|---|
| "We aimed to study the influence of sevoflurane on the nucleotide-binding domain and Leucine-rich repeat protein 3 (NLRP3) pathways in rats with cerebral ischemia/reperfusion (I/R) injury." | 8.31 | Sevoflurane preconditioning improves neuroinflammation in cerebral ischemia/reperfusion induced rats through ROS-NLRP3 pathway. ( Shang, S; Shao, W; Sun, F; Wang, Z; Yi, X; Yu, J; Yu, L; Zhu, Y, 2023) |
| "Potential additive effects of estrogens and sevoflurane against cerebral infarction after transient or permanent middle cerebral artery occlusion (MCAO) have not been addressed." | 7.83 | Protective effects of estrogen combined with sevoflurane in an experimental model of cerebral infarction and focal cerebral ischemia-reperfusion injury. ( Liu, JJ; Pan, SY, 2016) |
| "We aimed to study the influence of sevoflurane on the nucleotide-binding domain and Leucine-rich repeat protein 3 (NLRP3) pathways in rats with cerebral ischemia/reperfusion (I/R) injury." | 4.31 | Sevoflurane preconditioning improves neuroinflammation in cerebral ischemia/reperfusion induced rats through ROS-NLRP3 pathway. ( Shang, S; Shao, W; Sun, F; Wang, Z; Yi, X; Yu, J; Yu, L; Zhu, Y, 2023) |
| "Potential additive effects of estrogens and sevoflurane against cerebral infarction after transient or permanent middle cerebral artery occlusion (MCAO) have not been addressed." | 3.83 | Protective effects of estrogen combined with sevoflurane in an experimental model of cerebral infarction and focal cerebral ischemia-reperfusion injury. ( Liu, JJ; Pan, SY, 2016) |
| "Sevoflurane post-treatment decreased G9a and H3K9me2 levels, and G9a level was negatively correlated with NRF2 level." | 1.62 | Post-Treatment Sevoflurane Protects Against Hypoxic-Ischemic Brain Injury in Neonatal Rats by Downregulating Histone Methyltransferase G9a and Upregulating Nuclear Factor Erythroid 2-Related Factor 2 (NRF2). ( Li, X; Wang, H; Xu, Y; Zhang, H; Zhu, S, 2021) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (50.00) | 24.3611 |
| 2020's | 3 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Li, Y | 1 |
| Liang, Z | 1 |
| Lei, S | 1 |
| Wu, X | 1 |
| Yuan, T | 1 |
| Ma, K | 1 |
| Chi, K | 1 |
| Shang, S | 1 |
| Sun, F | 1 |
| Zhu, Y | 1 |
| Yu, J | 1 |
| Yu, L | 1 |
| Shao, W | 1 |
| Wang, Z | 1 |
| Yi, X | 1 |
| Wang, H | 1 |
| Xu, Y | 1 |
| Zhu, S | 1 |
| Li, X | 1 |
| Zhang, H | 2 |
| Tong, L | 1 |
| Cai, M | 1 |
| Huang, Y | 1 |
| Su, B | 1 |
| Li, Z | 1 |
| Dong, H | 1 |
| Liu, JJ | 1 |
| Pan, SY | 1 |
| Adamczyk, S | 1 |
| Robin, E | 1 |
| Simerabet, M | 1 |
| Kipnis, E | 1 |
| Tavernier, B | 1 |
| Vallet, B | 1 |
| Bordet, R | 1 |
| Lebuffe, G | 1 |
6 other studies available for sevoflurane and Cerebral Infarction
| Article | Year |
|---|---|
Sevoflurane Preconditioning Downregulates GRIA1 Expression to Attenuate Cerebral Ischemia-Reperfusion-Induced Neuronal Injury.
Topics: Animals; Apoptosis; Brain Ischemia; Cerebral Infarction; Glucose; Neurons; Neuroprotective Agents; O | 2023 |
Sevoflurane preconditioning improves neuroinflammation in cerebral ischemia/reperfusion induced rats through ROS-NLRP3 pathway.
Topics: Animals; Brain Ischemia; Caspase 1; Cerebral Infarction; Interleukin-18; Interleukin-6; Neuroinflamm | 2023 |
Post-Treatment Sevoflurane Protects Against Hypoxic-Ischemic Brain Injury in Neonatal Rats by Downregulating Histone Methyltransferase G9a and Upregulating Nuclear Factor Erythroid 2-Related Factor 2 (NRF2).
Topics: Animals; Animals, Newborn; Behavior, Animal; Cerebral Infarction; Disease Models, Animal; Gene Expre | 2021 |
Activation of K(2)P channel-TREK1 mediates the neuroprotection induced by sevoflurane preconditioning.
Topics: Animals; Brain; Brain Ischemia; Carbon Monoxide; Cell Differentiation; Cell Hypoxia; Cell Survival; | 2014 |
Protective effects of estrogen combined with sevoflurane in an experimental model of cerebral infarction and focal cerebral ischemia-reperfusion injury.
Topics: Animals; Brain Ischemia; Cerebral Infarction; Estrogens; Female; Infarction, Middle Cerebral Artery; | 2016 |
Sevoflurane pre- and post-conditioning protect the brain via the mitochondrial K ATP channel.
Topics: Anesthetics, Inhalation; Animals; Brain Ischemia; Carbon Dioxide; Cerebral Infarction; Hemodynamics; | 2010 |