sevoflurane has been researched along with Brain Neoplasms in 22 studies
Sevoflurane: A non-explosive inhalation anesthetic used in the induction and maintenance of general anesthesia. It does not cause respiratory irritation and may also prevent PLATELET AGGREGATION.
sevoflurane : An ether compound having fluoromethyl and 1,1,1,3,3,3-hexafluoroisopropyl as the two alkyl groups.
Brain Neoplasms: Neoplasms of the intracranial components of the central nervous system, including the cerebral hemispheres, basal ganglia, hypothalamus, thalamus, brain stem, and cerebellum. Brain neoplasms are subdivided into primary (originating from brain tissue) and secondary (i.e., metastatic) forms. Primary neoplasms are subdivided into benign and malignant forms. In general, brain tumors may also be classified by age of onset, histologic type, or presenting location in the brain.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Sevoflurane (SEV) is a common anesthetic to inhibit glioma progression." | 8.12 | Sevoflurane represses the progression of glioma by the regulation of circ_0037655/miR-130a-5p/RPN2 axis. ( Huang, G; Li, Y; Liu, Z; Xie, B; Zhu, J, 2022) |
| "Sevoflurane alleviated proliferation, migration and invasion, but enhanced apoptosis of glioma cells through regulating circ_VCAN/miR-146b-5p/NFIB axis." | 8.12 | Sevoflurane inhibits the malignant phenotypes of glioma through regulating miR-146b-5p/NFIB axis. ( Cheng, G; Feng, Y; Li, X; Pan, H; Qu, H; Quan, L; Shi, X; Wang, H; Yang, A; Ye, J, 2022) |
| "Sevoflurane has been reported to have anti-tumorigenic effects in glioma." | 8.02 | Sevoflurane impedes glioma progression via regulating circ_0000215/miR-1200/NCR3LG1 axis. ( Gao, B; Gao, R; Zhao, Z; Zong, X, 2021) |
| "Sevoflurane is a common inhalational anesthetic, which has been revealed to have anticancer effect in glioma." | 8.02 | Sevoflurane suppresses glioma tumorigenesis via regulating circ_0079593/miR-633/ROCK1 axis. ( Cheng, J; Cheng, S, 2021) |
| "Sevoflurane (sev) was employed to treat glioma cells." | 7.96 | Sevoflurane impedes the progression of glioma through modulating the circular RNA has_circ_0012129/miR-761/TGIF2 axis. ( Kang, LY; Liu, WW; Tang, L; Wang, W; Wei, WT; Xia, R; Xu, W; Xue, R; Zheng, JW, 2020) |
| " In this study, we examined the effects of sevoflurane on human glioblastoma cells and determined the role of cluster of differentiation (CD) 44, a cell surface protein involved in cell growth, migration, and invasion, in sevoflurane's effects." | 7.91 | Sevoflurane promotes migration, invasion, and colony-forming ability of human glioblastoma cells possibly via increasing the expression of cell surface protein 44. ( Lai, RC; Pan, DF; Shan, WR; Zeng, WA; Zeng, XQ; Zhou, D; Zuo, K; Zuo, ZY, 2019) |
| " Volatile anesthetic sevoflurane inhibits the migration and invasion of multiple cancer cell lines; however, its effects on glioma cells are unclear." | 7.83 | Sevoflurane inhibits the migration and invasion of glioma cells by upregulating microRNA-637. ( Guo, Y; Huang, B; Li, D; Li, X; Yi, W; Zhang, Y, 2016) |
| "Sevoflurane (Sev) has been reported to inhibit cancer development, and sevoflurane treatment in cancers is implicated with the deregulation of specific non-coding RNAs (ncRNAs)." | 5.62 | Sevoflurane blocks glioma malignant development by upregulating circRELN through circRELN-mediated miR-1290/RORA axis. ( Kang, X; Li, H; Zhang, Z, 2021) |
| "Glioma is the most common brain malignancy and surgical resection is the primary option for patient with glioma." | 5.56 | Sevoflurane inhibits migration and invasion of glioma cells via regulating miR-34a-5p/MMP-2 axis. ( Li, Z; Xing, F; Yuan, J; Zhang, W; Zhao, H, 2020) |
| "Post-operative cerebral edema is a threat for patients performed gliomas resection." | 5.42 | Effects of propofol and sevoflurane on aquaporin-4 and aquaporin-9 expression in patients performed gliomas resection. ( Chang, FF; Dong, H; Lu, SJ; Qian, H; Song, CY; Yang, WC; Yue, ZY; Zhang, R; Zhou, LJ, 2015) |
| "The study indicates that before as well as during hyperventilation, subdural ICP and AVDo2 are lower and CPP higher in propofol-anesthetized patients compared with patients anesthetized with isoflurane or sevoflurane." | 5.10 | Intracranial pressure and cerebral hemodynamic in patients with cerebral tumors: a randomized prospective study of patients subjected to craniotomy in propofol-fentanyl, isoflurane-fentanyl, or sevoflurane-fentanyl anesthesia. ( Cold, GE; Hauerberg, J; Holst, P; Landsfeldt, U; Mau, S; Olsen, KS; Petersen, CB; Petersen, KD, 2003) |
| "Sevoflurane (SEV) is a common anesthetic to inhibit glioma progression." | 4.12 | Sevoflurane represses the progression of glioma by the regulation of circ_0037655/miR-130a-5p/RPN2 axis. ( Huang, G; Li, Y; Liu, Z; Xie, B; Zhu, J, 2022) |
| "Sevoflurane alleviated proliferation, migration and invasion, but enhanced apoptosis of glioma cells through regulating circ_VCAN/miR-146b-5p/NFIB axis." | 4.12 | Sevoflurane inhibits the malignant phenotypes of glioma through regulating miR-146b-5p/NFIB axis. ( Cheng, G; Feng, Y; Li, X; Pan, H; Qu, H; Quan, L; Shi, X; Wang, H; Yang, A; Ye, J, 2022) |
| "Sevoflurane has been reported to have anti-tumorigenic effects in glioma." | 4.02 | Sevoflurane impedes glioma progression via regulating circ_0000215/miR-1200/NCR3LG1 axis. ( Gao, B; Gao, R; Zhao, Z; Zong, X, 2021) |
| "Sevoflurane is a common inhalational anesthetic, which has been revealed to have anticancer effect in glioma." | 4.02 | Sevoflurane suppresses glioma tumorigenesis via regulating circ_0079593/miR-633/ROCK1 axis. ( Cheng, J; Cheng, S, 2021) |
| "Sevoflurane (sev) was employed to treat glioma cells." | 3.96 | Sevoflurane impedes the progression of glioma through modulating the circular RNA has_circ_0012129/miR-761/TGIF2 axis. ( Kang, LY; Liu, WW; Tang, L; Wang, W; Wei, WT; Xia, R; Xu, W; Xue, R; Zheng, JW, 2020) |
| " In this study, we examined the effects of sevoflurane on human glioblastoma cells and determined the role of cluster of differentiation (CD) 44, a cell surface protein involved in cell growth, migration, and invasion, in sevoflurane's effects." | 3.91 | Sevoflurane promotes migration, invasion, and colony-forming ability of human glioblastoma cells possibly via increasing the expression of cell surface protein 44. ( Lai, RC; Pan, DF; Shan, WR; Zeng, WA; Zeng, XQ; Zhou, D; Zuo, K; Zuo, ZY, 2019) |
| " Volatile anesthetic sevoflurane inhibits the migration and invasion of multiple cancer cell lines; however, its effects on glioma cells are unclear." | 3.83 | Sevoflurane inhibits the migration and invasion of glioma cells by upregulating microRNA-637. ( Guo, Y; Huang, B; Li, D; Li, X; Yi, W; Zhang, Y, 2016) |
| "Remifentanil was used as the analgetic component in all groups." | 2.71 | [Comparison between TCI-TIVA, manual TIVA and balanced anaesthesia for stereotactic biopsy of the brain]. ( Czerner, S; Steude, U; Weninger, B; Weninger, E, 2004) |
| "Sevoflurane (Sev) has been reported to inhibit cancer development, and sevoflurane treatment in cancers is implicated with the deregulation of specific non-coding RNAs (ncRNAs)." | 1.62 | Sevoflurane blocks glioma malignant development by upregulating circRELN through circRELN-mediated miR-1290/RORA axis. ( Kang, X; Li, H; Zhang, Z, 2021) |
| "Glioma is the most common brain malignancy and surgical resection is the primary option for patient with glioma." | 1.56 | Sevoflurane inhibits migration and invasion of glioma cells via regulating miR-34a-5p/MMP-2 axis. ( Li, Z; Xing, F; Yuan, J; Zhang, W; Zhao, H, 2020) |
| "Post-operative cerebral edema is a threat for patients performed gliomas resection." | 1.42 | Effects of propofol and sevoflurane on aquaporin-4 and aquaporin-9 expression in patients performed gliomas resection. ( Chang, FF; Dong, H; Lu, SJ; Qian, H; Song, CY; Yang, WC; Yue, ZY; Zhang, R; Zhou, LJ, 2015) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (4.55) | 18.2507 |
| 2000's | 5 (22.73) | 29.6817 |
| 2010's | 7 (31.82) | 24.3611 |
| 2020's | 9 (40.91) | 2.80 |
| Authors | Studies |
|---|---|
| Zhao, Z | 1 |
| Gao, B | 1 |
| Zong, X | 1 |
| Gao, R | 1 |
| Kang, X | 1 |
| Li, H | 2 |
| Zhang, Z | 1 |
| Bao, X | 1 |
| Peng, Y | 1 |
| Shen, J | 1 |
| Yang, L | 1 |
| Zhu, J | 1 |
| Xie, B | 1 |
| Huang, G | 1 |
| Li, Y | 2 |
| Liu, Z | 1 |
| Wang, H | 1 |
| Cheng, G | 1 |
| Quan, L | 1 |
| Qu, H | 1 |
| Yang, A | 1 |
| Ye, J | 1 |
| Feng, Y | 1 |
| Li, X | 2 |
| Shi, X | 1 |
| Pan, H | 1 |
| Xu, W | 1 |
| Xue, R | 1 |
| Xia, R | 1 |
| Liu, WW | 1 |
| Zheng, JW | 1 |
| Tang, L | 1 |
| Kang, LY | 1 |
| Wang, W | 1 |
| Wei, WT | 1 |
| Zhao, H | 1 |
| Xing, F | 1 |
| Yuan, J | 1 |
| Li, Z | 1 |
| Zhang, W | 1 |
| Qu, Y | 1 |
| Shi, C | 1 |
| Qian, M | 1 |
| Yang, N | 1 |
| Wang, L | 1 |
| Gao, X | 1 |
| Ni, C | 1 |
| Cheng, S | 1 |
| Cheng, J | 1 |
| Lai, RC | 1 |
| Shan, WR | 1 |
| Zhou, D | 1 |
| Zeng, XQ | 1 |
| Zuo, K | 1 |
| Pan, DF | 1 |
| Zeng, WA | 1 |
| Zuo, ZY | 1 |
| Zhang, L | 1 |
| Wang, J | 1 |
| Fu, Z | 1 |
| Ai, Y | 1 |
| Wang, Y | 2 |
| Kumar, N | 1 |
| Bindra, A | 1 |
| Mahajan, C | 1 |
| Yang, WC | 1 |
| Zhou, LJ | 1 |
| Zhang, R | 1 |
| Yue, ZY | 1 |
| Dong, H | 1 |
| Song, CY | 1 |
| Qian, H | 1 |
| Lu, SJ | 1 |
| Chang, FF | 1 |
| Yi, W | 1 |
| Li, D | 1 |
| Guo, Y | 1 |
| Zhang, Y | 1 |
| Huang, B | 1 |
| Tankó, B | 1 |
| Molnár, C | 1 |
| Budi, T | 1 |
| Peto, C | 1 |
| Novák, L | 1 |
| Fülesdi, B | 1 |
| Misra, S | 1 |
| Koshy, T | 1 |
| Unnikrishnan, KP | 1 |
| Suneel, PR | 1 |
| Chatterjee, N | 1 |
| Banevičius, G | 1 |
| Rugytė, D | 1 |
| Macas, A | 1 |
| Tamašauskas, A | 1 |
| Stankevičius, E | 1 |
| Petersen, KD | 1 |
| Landsfeldt, U | 1 |
| Cold, GE | 1 |
| Petersen, CB | 1 |
| Mau, S | 1 |
| Hauerberg, J | 1 |
| Holst, P | 1 |
| Olsen, KS | 1 |
| Weninger, B | 1 |
| Czerner, S | 1 |
| Steude, U | 1 |
| Weninger, E | 1 |
| Boztuğ, N | 1 |
| Bigat, Z | 1 |
| Akyuz, M | 1 |
| Demir, S | 1 |
| Ertok, E | 1 |
| Magni, G | 1 |
| La Rosa, I | 1 |
| Gimignani, S | 1 |
| Melillo, G | 1 |
| Imperiale, C | 1 |
| Rosa, G | 1 |
| Inada, T | 1 |
| Shingu, K | 1 |
| Uchida, M | 1 |
| Kawachi, S | 1 |
| Tsushima, K | 1 |
| Niitsu, T | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Influence of Vasopressors on Brain Oxygenation and Microcirculation in Anesthetized Patients With Cerebral Tumors[NCT02713087] | Phase 4 | 48 participants (Actual) | Interventional | 2015-09-30 | Completed | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
6 trials available for sevoflurane and Brain Neoplasms
| Article | Year |
|---|---|
Gabapentin premedication decreases the hemodynamic response to skull pin insertion in patients undergoing craniotomy.
Topics: Adolescent; Adult; Amines; Anesthesia, General; Anesthetics, Inhalation; Anesthetics, Local; Blood P | 2011 |
The effects of sevoflurane and propofol on cerebral hemodynamics during intracranial tumors surgery under monitoring the depth of anesthesia.
Topics: Adolescent; Adult; Anesthesia, Intravenous; Anesthetics, Intravenous; Blood Flow Velocity; Brain; Br | 2010 |
Intracranial pressure and cerebral hemodynamic in patients with cerebral tumors: a randomized prospective study of patients subjected to craniotomy in propofol-fentanyl, isoflurane-fentanyl, or sevoflurane-fentanyl anesthesia.
Topics: Adolescent; Adult; Aged; Anesthesia, General; Anesthesia, Inhalation; Anesthetics, Inhalation; Anest | 2003 |
[Comparison between TCI-TIVA, manual TIVA and balanced anaesthesia for stereotactic biopsy of the brain].
Topics: Analgesics; Anesthesia, Intravenous; Anesthetics, Intravenous; Biopsy; Blood Pressure; Brain Neoplas | 2004 |
Does using the bispectral index (BIS) during craniotomy affect the quality of recovery?
Topics: Adjuvants, Anesthesia; Adolescent; Adult; Aged; Anesthesia Recovery Period; Anesthesia, General; Ane | 2006 |
Changes in the cerebral arteriovenous oxygen content difference by surgical incision are similar during sevoflurane and isoflurane anaesthesia.
Topics: Adult; Anesthetics, Inhalation; Brain Neoplasms; Cerebrovascular Circulation; Ethers; Female; Hemody | 1996 |
16 other studies available for sevoflurane and Brain Neoplasms
| Article | Year |
|---|---|
Sevoflurane impedes glioma progression via regulating circ_0000215/miR-1200/NCR3LG1 axis.
Topics: Animals; B7 Antigens; Brain Neoplasms; Cell Movement; Glioma; Humans; Mice; Mice, Nude; MicroRNAs; R | 2021 |
Sevoflurane blocks glioma malignant development by upregulating circRELN through circRELN-mediated miR-1290/RORA axis.
Topics: Anesthetics, Inhalation; Apoptosis; Brain Neoplasms; Cell Cycle Checkpoints; Cell Line, Tumor; Cell | 2021 |
Sevoflurane inhibits progression of glioma via regulating the HMMR antisense RNA 1/microRNA-7/cyclin dependent kinase 4 axis.
Topics: Animals; Brain Neoplasms; Cell Line, Tumor; Cell Survival; Cyclin-Dependent Kinase 4; Disease Progre | 2021 |
Sevoflurane represses the progression of glioma by the regulation of circ_0037655/miR-130a-5p/RPN2 axis.
Topics: Brain Neoplasms; Cell Line, Tumor; Glioma; Hexosyltransferases; Humans; MicroRNAs; Proteasome Endope | 2022 |
Sevoflurane inhibits the malignant phenotypes of glioma through regulating miR-146b-5p/NFIB axis.
Topics: Animals; Brain Neoplasms; Cell Proliferation; Glioma; Humans; Mice; MicroRNAs; NFI Transcription Fac | 2022 |
Sevoflurane impedes the progression of glioma through modulating the circular RNA has_circ_0012129/miR-761/TGIF2 axis.
Topics: Animals; Antineoplastic Agents; Brain Neoplasms; Cell Movement; Cell Proliferation; Cell Survival; D | 2020 |
Sevoflurane inhibits migration and invasion of glioma cells via regulating miR-34a-5p/MMP-2 axis.
Topics: Base Sequence; Brain Neoplasms; Cell Line, Tumor; Cell Movement; Gene Expression Regulation, Neoplas | 2020 |
lncRNAs Are Involved in Sevoflurane Anesthesia-Related Brain Function Modulation through Affecting Mitochondrial Function and Aging Process.
Topics: Aging; Anesthesia; Anesthetics; Animals; Brain; Brain Neoplasms; Cell Line, Tumor; Cognition Disorde | 2020 |
Sevoflurane suppresses glioma tumorigenesis via regulating circ_0079593/miR-633/ROCK1 axis.
Topics: Animals; Apoptosis; Brain Neoplasms; Carcinogenesis; Cell Line, Tumor; Cell Movement; Cell Prolifera | 2021 |
Sevoflurane promotes migration, invasion, and colony-forming ability of human glioblastoma cells possibly via increasing the expression of cell surface protein 44.
Topics: Animals; Brain; Brain Neoplasms; Cell Line, Tumor; Cell Movement; Glioblastoma; Humans; Hyaluronan R | 2019 |
Sevoflurane suppresses migration and invasion of glioma cells by regulating miR-146b-5p and MMP16.
Topics: Base Sequence; Brain Neoplasms; Cell Line, Tumor; Cell Movement; Gene Knockdown Techniques; Glioma; | 2019 |
Anaesthetic management of a patient with brugada syndrome for craniotomy and tumor excision.
Topics: Adult; Anesthesia, General; Anesthetics, Inhalation; Brain Neoplasms; Brugada Syndrome; Craniotomy; | 2013 |
Effects of propofol and sevoflurane on aquaporin-4 and aquaporin-9 expression in patients performed gliomas resection.
Topics: Aquaporin 4; Aquaporins; Blotting, Western; Brain; Brain Edema; Brain Neoplasms; Female; Glioma; Hom | 2015 |
Sevoflurane inhibits the migration and invasion of glioma cells by upregulating microRNA-637.
Topics: Antineoplastic Agents; Brain Neoplasms; Cell Line, Tumor; Cell Movement; Cell Proliferation; Gene Ex | 2016 |
The relative exposure of the operating room staff to sevoflurane during intracerebral surgery.
Topics: Adolescent; Adult; Aged; Air Pollutants, Occupational; Air Pollution, Indoor; Anesthesiology; Anesth | 2009 |
Early postoperative complications after intracranial surgery: comparison between total intravenous and balanced anesthesia.
Topics: Adult; Aged; Anesthesia, Inhalation; Anesthesia, Intravenous; Anesthetics, Combined; Anesthetics, In | 2007 |