Page last updated: 2024-11-04

sevoflurane and Brain Infarction

sevoflurane has been researched along with Brain Infarction in 2 studies

Sevoflurane: A non-explosive inhalation anesthetic used in the induction and maintenance of general anesthesia. It does not cause respiratory irritation and may also prevent PLATELET AGGREGATION.
sevoflurane : An ether compound having fluoromethyl and 1,1,1,3,3,3-hexafluoroisopropyl as the two alkyl groups.

Brain Infarction: Tissue NECROSIS in any area of the brain, including the CEREBRAL HEMISPHERES, the CEREBELLUM, and the BRAIN STEM. Brain infarction is the result of a cascade of events initiated by inadequate blood flow through the brain that is followed by HYPOXIA and HYPOGLYCEMIA in brain tissue. Damage may be temporary, permanent, selective or pan-necrosis.

Research Excerpts

ExcerptRelevanceReference
"Sevoflurane treatment also caused increased phosphorylation of p38 MAPK at 24 and 72 h after reperfusion."1.38Delayed neuroprotection induced by sevoflurane via opening mitochondrial ATP-sensitive potassium channels and p38 MAPK phosphorylation. ( Guo, Q; Wang, E; Wang, N; Xia, P; Ye, Z; Yuan, Y, 2012)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Li, B1
Sun, J1
Lv, G1
Yu, Y2
Wang, G1
Xie, K1
Jiao, Y1
Ye, Z1
Guo, Q1
Wang, N1
Xia, P1
Yuan, Y1
Wang, E1

Other Studies

2 other studies available for sevoflurane and Brain Infarction

ArticleYear
Sevoflurane postconditioning attenuates cerebral ischemia-reperfusion injury via protein kinase B/nuclear factor-erythroid 2-related factor 2 pathway activation.
    International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, 2014, Volume: 38

    Topics: Animals; Brain Infarction; Disease Models, Animal; Electrophoretic Mobility Shift Assay; Enzyme Inhi

2014
Delayed neuroprotection induced by sevoflurane via opening mitochondrial ATP-sensitive potassium channels and p38 MAPK phosphorylation.
    Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology, 2012, Volume: 33, Issue:2

    Topics: Analysis of Variance; Animals; Anti-Arrhythmia Agents; Brain Infarction; Cell Count; Decanoic Acids;

2012