sevoflurane has been researched along with Brain Edema in 9 studies
Sevoflurane: A non-explosive inhalation anesthetic used in the induction and maintenance of general anesthesia. It does not cause respiratory irritation and may also prevent PLATELET AGGREGATION.
sevoflurane : An ether compound having fluoromethyl and 1,1,1,3,3,3-hexafluoroisopropyl as the two alkyl groups.
Brain Edema: Increased intracellular or extracellular fluid in brain tissue. Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (From Childs Nerv Syst 1992 Sep; 8(6):301-6)
| Excerpt | Relevance | Reference |
|---|---|---|
| "5% sevoflurane for 60 min and 3% sevoflurane for 30 and 60 min significantly improved neurobehavioral function, brain edema, and attenuated neuronal cell death in the basal cortex at 24 h after SAH." | 7.96 | Subarachnoid Hemorrhage and Sevoflurane. ( Altay, O; Sorar, M, 2020) |
| "Postconditioning with sevoflurane has been shown to protect against focal cerebral ischemia and reperfusion injury." | 7.81 | Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore. ( Liu, XZ; Wang, JK; Wu, HF; Yang, B; Zhou, H, 2015) |
| "The first step to treat aneurysmal subarachnoid hemorrhage (SAH) is aneurysmal obliteration under general anesthesia but not treat the SAH itself and the secondary effects." | 5.56 | Isoflurane versus sevoflurane for early brain injury and expression of sphingosine kinase 1 after experimental subarachnoid hemorrhage. ( Altay, BN; Altay, O; Calisir, V; Suzuki, H; Tang, J; Zhang, JH, 2020) |
| "Post-operative cerebral edema is a threat for patients performed gliomas resection." | 5.42 | Effects of propofol and sevoflurane on aquaporin-4 and aquaporin-9 expression in patients performed gliomas resection. ( Chang, FF; Dong, H; Lu, SJ; Qian, H; Song, CY; Yang, WC; Yue, ZY; Zhang, R; Zhou, LJ, 2015) |
| "The study indicates that before as well as during hyperventilation, subdural ICP and AVDo2 are lower and CPP higher in propofol-anesthetized patients compared with patients anesthetized with isoflurane or sevoflurane." | 5.10 | Intracranial pressure and cerebral hemodynamic in patients with cerebral tumors: a randomized prospective study of patients subjected to craniotomy in propofol-fentanyl, isoflurane-fentanyl, or sevoflurane-fentanyl anesthesia. ( Cold, GE; Hauerberg, J; Holst, P; Landsfeldt, U; Mau, S; Olsen, KS; Petersen, CB; Petersen, KD, 2003) |
| "5% sevoflurane for 60 min and 3% sevoflurane for 30 and 60 min significantly improved neurobehavioral function, brain edema, and attenuated neuronal cell death in the basal cortex at 24 h after SAH." | 3.96 | Subarachnoid Hemorrhage and Sevoflurane. ( Altay, O; Sorar, M, 2020) |
| "Postconditioning with sevoflurane has been shown to protect against focal cerebral ischemia and reperfusion injury." | 3.81 | Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore. ( Liu, XZ; Wang, JK; Wu, HF; Yang, B; Zhou, H, 2015) |
| "The first step to treat aneurysmal subarachnoid hemorrhage (SAH) is aneurysmal obliteration under general anesthesia but not treat the SAH itself and the secondary effects." | 1.56 | Isoflurane versus sevoflurane for early brain injury and expression of sphingosine kinase 1 after experimental subarachnoid hemorrhage. ( Altay, BN; Altay, O; Calisir, V; Suzuki, H; Tang, J; Zhang, JH, 2020) |
| "Post-operative cerebral edema is a threat for patients performed gliomas resection." | 1.42 | Effects of propofol and sevoflurane on aquaporin-4 and aquaporin-9 expression in patients performed gliomas resection. ( Chang, FF; Dong, H; Lu, SJ; Qian, H; Song, CY; Yang, WC; Yue, ZY; Zhang, R; Zhou, LJ, 2015) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (22.22) | 29.6817 |
| 2010's | 4 (44.44) | 24.3611 |
| 2020's | 3 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Beck-Schimmer, B | 1 |
| Restin, T | 1 |
| Muroi, C | 1 |
| Roth Z'Graggen, B | 1 |
| Keller, E | 1 |
| Schläpfer, M | 1 |
| Altay, O | 2 |
| Suzuki, H | 1 |
| Altay, BN | 1 |
| Calisir, V | 1 |
| Tang, J | 1 |
| Zhang, JH | 1 |
| Sorar, M | 1 |
| Wang, JK | 1 |
| Wu, HF | 1 |
| Zhou, H | 1 |
| Yang, B | 1 |
| Liu, XZ | 1 |
| Yang, WC | 1 |
| Zhou, LJ | 1 |
| Zhang, R | 1 |
| Yue, ZY | 1 |
| Dong, H | 1 |
| Song, CY | 1 |
| Qian, H | 1 |
| Lu, SJ | 1 |
| Chang, FF | 1 |
| Bilotta, F | 1 |
| Doronzio, A | 1 |
| Cuzzone, V | 1 |
| Caramia, R | 1 |
| Rosa, G | 1 |
| Bedirli, N | 1 |
| Bagriacik, EU | 1 |
| Emmez, H | 1 |
| Yilmaz, G | 1 |
| Unal, Y | 1 |
| Ozkose, Z | 1 |
| Thal, SC | 1 |
| Luh, C | 1 |
| Schaible, EV | 1 |
| Timaru-Kast, R | 1 |
| Hedrich, J | 1 |
| Luhmann, HJ | 1 |
| Engelhard, K | 1 |
| Zehendner, CM | 1 |
| Petersen, KD | 1 |
| Landsfeldt, U | 1 |
| Cold, GE | 1 |
| Petersen, CB | 1 |
| Mau, S | 1 |
| Hauerberg, J | 1 |
| Holst, P | 1 |
| Olsen, KS | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Influence of Vasopressors on Brain Oxygenation and Microcirculation in Anesthetized Patients With Cerebral Tumors[NCT02713087] | Phase 4 | 48 participants (Actual) | Interventional | 2015-09-30 | Completed | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
2 trials available for sevoflurane and Brain Edema
| Article | Year |
|---|---|
Early postoperative cognitive recovery and gas exchange patterns after balanced anesthesia with sevoflurane or desflurane in overweight and obese patients undergoing craniotomy: a prospective randomized trial.
Topics: Adolescent; Adult; Aged; Anesthesia, Inhalation; Anesthetics, Inhalation; Brain Edema; Cognition; Cr | 2009 |
Intracranial pressure and cerebral hemodynamic in patients with cerebral tumors: a randomized prospective study of patients subjected to craniotomy in propofol-fentanyl, isoflurane-fentanyl, or sevoflurane-fentanyl anesthesia.
Topics: Adolescent; Adult; Aged; Anesthesia, General; Anesthesia, Inhalation; Anesthetics, Inhalation; Anest | 2003 |
7 other studies available for sevoflurane and Brain Edema
| Article | Year |
|---|---|
Sevoflurane sedation attenuates early cerebral oedema formation through stabilisation of the adherens junction protein beta catenin in a model of subarachnoid haemorrhage: A randomised animal study.
Topics: Adherens Junctions; Anesthesia; Animals; beta Catenin; Brain Edema; Male; Rats; Rats, Wistar; Sevofl | 2020 |
Isoflurane versus sevoflurane for early brain injury and expression of sphingosine kinase 1 after experimental subarachnoid hemorrhage.
Topics: Animals; Apoptosis; Brain Edema; Disease Models, Animal; Isoflurane; Male; Mice; Neuroprotective Age | 2020 |
Subarachnoid Hemorrhage and Sevoflurane.
Topics: Animals; Apoptosis; Brain; Brain Edema; Male; Mice; Mice, Inbred BALB C; Neuroprotective Agents; Pla | 2020 |
Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore.
Topics: Animals; Apoptosis; Brain Edema; Brain Ischemia; Disease Models, Animal; Infarction, Middle Cerebral | 2015 |
Effects of propofol and sevoflurane on aquaporin-4 and aquaporin-9 expression in patients performed gliomas resection.
Topics: Aquaporin 4; Aquaporins; Blotting, Western; Brain; Brain Edema; Brain Neoplasms; Female; Glioma; Hom | 2015 |
Sevoflurane and isoflurane preconditioning provides neuroprotection by inhibition of apoptosis-related mRNA expression in a rat model of focal cerebral ischemia.
Topics: Anesthetics, Inhalation; Animals; Antioxidants; Apoptosis; Apoptosis Regulatory Proteins; Brain Edem | 2012 |
Volatile anesthetics influence blood-brain barrier integrity by modulation of tight junction protein expression in traumatic brain injury.
Topics: Anesthetics, Inhalation; Animals; Blood-Brain Barrier; Brain Edema; Brain Injuries; Cell Line; Claud | 2012 |