Page last updated: 2024-11-08

serine and Death, Sudden, Cardiac

serine has been researched along with Death, Sudden, Cardiac in 2 studies

Serine: A non-essential amino acid occurring in natural form as the L-isomer. It is synthesized from GLYCINE or THREONINE. It is involved in the biosynthesis of PURINES; PYRIMIDINES; and other amino acids.
serine : An alpha-amino acid that is alanine substituted at position 3 by a hydroxy group.

Death, Sudden, Cardiac: Unexpected rapid natural death due to cardiovascular collapse within one hour of initial symptoms. It is usually caused by the worsening of existing heart diseases. The sudden onset of symptoms, such as CHEST PAIN and CARDIAC ARRHYTHMIAS, particularly VENTRICULAR TACHYCARDIA, can lead to the loss of consciousness and cardiac arrest followed by biological death. (from Braunwald's Heart Disease: A Textbook of Cardiovascular Medicine, 7th ed., 2005)

Research Excerpts

ExcerptRelevanceReference
"Ventricular arrhythmia is the most important risk factor for sudden cardiac death (SCD) after acute myocardial infarction (MI) worldwide."1.56Cardiac-Specific Caveolin-3 Overexpression Prevents Post-Myocardial Infarction Ventricular Arrhythmias by Inhibiting Ryanodine Receptor-2 Hyperphosphorylation. ( Chen, G; Du, T; Fang, Q; Wang, DW; Wang, Y; Zhang, Z, 2020)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's0 (0.00)24.3611
2020's1 (50.00)2.80

Authors

AuthorsStudies
Zhang, Z1
Fang, Q1
Du, T1
Chen, G1
Wang, Y1
Wang, DW1
Valdivia, CR1
Ueda, K1
Ackerman, MJ1
Makielski, JC1

Other Studies

2 other studies available for serine and Death, Sudden, Cardiac

ArticleYear
Cardiac-Specific Caveolin-3 Overexpression Prevents Post-Myocardial Infarction Ventricular Arrhythmias by Inhibiting Ryanodine Receptor-2 Hyperphosphorylation.
    Cardiology, 2020, Volume: 145, Issue:3

    Topics: Animals; Arrhythmias, Cardiac; Calcium; Caveolin 3; Death, Sudden, Cardiac; Male; Mice; Mice, Inbred

2020
GPD1L links redox state to cardiac excitability by PKC-dependent phosphorylation of the sodium channel SCN5A.
    American journal of physiology. Heart and circulatory physiology, 2009, Volume: 297, Issue:4

    Topics: Brugada Syndrome; Cell Line; Death, Sudden, Cardiac; Glycerolphosphate Dehydrogenase; Glycerophospha

2009