Page last updated: 2024-11-04

sb 239063 and Injury, Myocardial Reperfusion

sb 239063 has been researched along with Injury, Myocardial Reperfusion in 2 studies

SB 239063: structure in first source
SB-239063 : A member of the class of imidazoles carrying 4-hydroxycyclohexyl, 4-fluorophenyl and 2-methoxypyrimidin-4-yl substituents at positions 1, 4 and 5 respectively.

Research Excerpts

ExcerptRelevanceReference
"However, the effect of p38 MAPK on myocardial reperfusion injury, a pathologic condition involving a typical inflammatory response, has not been fully examined."1.31p38 MAPK inhibition reduces myocardial reperfusion injury via inhibition of endothelial adhesion molecule expression and blockade of PMN accumulation. ( Barone, FC; Christopher, TA; Gao, F; Lopez, BL; Ma, XL; Ohlstein, EH; Shi, DW; Yue, TL, 2002)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chai, W1
Wu, Y1
Li, G1
Cao, W1
Yang, Z1
Liu, Z1
Gao, F1
Yue, TL1
Shi, DW1
Christopher, TA1
Lopez, BL1
Ohlstein, EH1
Barone, FC1
Ma, XL1

Other Studies

2 other studies available for sb 239063 and Injury, Myocardial Reperfusion

ArticleYear
Activation of p38 mitogen-activated protein kinase abolishes insulin-mediated myocardial protection against ischemia-reperfusion injury.
    American journal of physiology. Endocrinology and metabolism, 2008, Volume: 294, Issue:1

    Topics: Animals; Anisomycin; Cardiotonic Agents; Drug Interactions; Enzyme Activation; Enzyme Inhibitors; Hy

2008
p38 MAPK inhibition reduces myocardial reperfusion injury via inhibition of endothelial adhesion molecule expression and blockade of PMN accumulation.
    Cardiovascular research, 2002, Feb-01, Volume: 53, Issue:2

    Topics: Analysis of Variance; Animals; Endothelium, Vascular; Imidazoles; Intercellular Adhesion Molecule-1;

2002