Page last updated: 2024-11-04

sb 239063 and Encephalopathy, Hepatic

sb 239063 has been researched along with Encephalopathy, Hepatic in 3 studies

SB 239063: structure in first source
SB-239063 : A member of the class of imidazoles carrying 4-hydroxycyclohexyl, 4-fluorophenyl and 2-methoxypyrimidin-4-yl substituents at positions 1, 4 and 5 respectively.

Research Excerpts

ExcerptRelevanceReference
"Besides, neuroinflammation has captured more attention in the process of HE, but the mechanism of neuroinflammation leading to HE remains unclear."2.52PET and MR imaging of neuroinflammation in hepatic encephalopathy. ( Lu, GM; Su, YY; Wu, S; Yang, GF; Zhang, LJ, 2015)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Agusti, A2
Dziedzic, JL1
Hernandez-Rabaza, V2
Guilarte, TR1
Felipo, V2
Su, YY1
Yang, GF1
Lu, GM1
Wu, S1
Zhang, LJ1
Cauli, O1
Rodrigo, R1
Llansola, M1

Reviews

1 review available for sb 239063 and Encephalopathy, Hepatic

ArticleYear
PET and MR imaging of neuroinflammation in hepatic encephalopathy.
    Metabolic brain disease, 2015, Volume: 30, Issue:1

    Topics: Amino Acid Transport System X-AG; Animals; Blood-Brain Barrier; Brain; Brain Edema; Cytokines; Disea

2015

Other Studies

2 other studies available for sb 239063 and Encephalopathy, Hepatic

ArticleYear
Rats with minimal hepatic encephalopathy due to portacaval shunt show differential increase of translocator protein (18 kDa) binding in different brain areas, which is not affected by chronic MAP-kinase p38 inhibition.
    Metabolic brain disease, 2014, Volume: 29, Issue:4

    Topics: Animals; Biomarkers; Brain; Carrier Proteins; Disease Models, Animal; Hepatic Encephalopathy; Imidaz

2014
p38 MAP kinase is a therapeutic target for hepatic encephalopathy in rats with portacaval shunts.
    Gut, 2011, Volume: 60, Issue:11

    Topics: Ammonia; Animals; Blotting, Western; Cerebellum; Cerebral Cortex; Cognition; Disease Models, Animal;

2011