sb 216763 has been researched along with Infarction, Middle Cerebral Artery in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bai, WJ; Hou, K; Hu, WY; Hu, YX; Jiao, Y; Kang, Q; Liu, SY; Liu, ZY; Lv, KY; Ma, N; Qiu, CW; Xiao, ZC; Zhang, FL; Zhang, L | 1 |
| Meng, C; Wang, Y; Wu, J; Zhang, J; Zhao, J | 1 |
| Bertolotti, P; Carruba, MO; De Simoni, MG; Delbarba, A; Dossena, M; Nisoli, E; Perego, C; Ragni, M; Spano, P; Valerio, A | 1 |
3 other study(ies) available for sb 216763 and Infarction, Middle Cerebral Artery
| Article | Year |
|---|---|
Wip1 knockout inhibits neurogenesis by affecting the Wnt/β-catenin signaling pathway in focal cerebral ischemia in mice.
Topics: Animals; beta Catenin; Brain Infarction; Bromodeoxyuridine; Disease Models, Animal; Doublecortin Domain Proteins; Enzyme Inhibitors; Gene Expression Regulation; In Situ Nick-End Labeling; Indoles; Infarction, Middle Cerebral Artery; Male; Maleimides; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microtubule-Associated Proteins; Neurogenesis; Neuropeptides; Protein Phosphatase 2C; Severity of Illness Index; Statistics, Nonparametric; Wnt Signaling Pathway | 2018 |
Inhibition of GSK-3β alleviates cerebral ischemia/reperfusion injury in rats by suppressing NLRP3 inflammasome activation through autophagy.
Topics: Animals; Autophagy; Cerebral Cortex; Glycogen Synthase Kinase 3 beta; Indoles; Infarction, Middle Cerebral Artery; Inflammasomes; Interleukin-18; Interleukin-1beta; Male; Maleimides; NLR Family, Pyrin Domain-Containing 3 Protein; Rats, Sprague-Dawley; Reperfusion Injury; RNA, Small Interfering | 2019 |
Glycogen synthase kinase-3 inhibition reduces ischemic cerebral damage, restores impaired mitochondrial biogenesis and prevents ROS production.
Topics: Animals; Cells, Cultured; Cerebral Cortex; Disease Models, Animal; DNA-Binding Proteins; DNA, Mitochondrial; Dose-Response Relationship, Drug; Embryo, Mammalian; Enzyme Inhibitors; Glucose; Glycogen Synthase Kinase 3; High Mobility Group Proteins; Hypoxia; Indoles; Infarction, Middle Cerebral Artery; L-Lactate Dehydrogenase; Maleimides; Mice; Mitochondria; Mutation; Neurons; Nuclear Respiratory Factor 1; Organelle Biogenesis; Reactive Oxygen Species; RNA, Messenger; Transfection | 2011 |