Page last updated: 2024-09-05

sb 216763 and Colorectal Neoplasms

sb 216763 has been researched along with Colorectal Neoplasms in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (40.00)29.6817
2010's3 (60.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Jung, DB; Jung, JH; Kang, SE; Kim, H; Kim, SH; Lee, H; Srivastava, SK; Yun, M1
Phyu, SM; Smith, TAD; Tseng, CC1
Feng, Y; Lu, H; Mao, D; Qiao, L1
Bruyneel, E; Dale, T; De Wever, O; Gespach, C; Le Floch, N; Mareel, M; Rivat, C1
Iyer, NG; Leong, HS; Liu, ET; Tan, J; Yu, Q; Zhuang, L1

Other Studies

5 other study(ies) available for sb 216763 and Colorectal Neoplasms

ArticleYear
Zinc finger protein 746 promotes colorectal cancer progression via c-Myc stability mediated by glycogen synthase kinase 3β and F-box and WD repeat domain-containing 7.
    Oncogene, 2018, Volume: 37, Issue:27

    Topics: Animals; Cell Line, Tumor; Colorectal Neoplasms; Cysteine Proteinase Inhibitors; Disease Progression; F-Box-WD Repeat-Containing Protein 7; G1 Phase Cell Cycle Checkpoints; Gene Expression Profiling; Glycogen Synthase Kinase 3 beta; HCT116 Cells; HEK293 Cells; HT29 Cells; Humans; Indoles; Leupeptins; Male; Maleimides; Mice; Mice, Inbred BALB C; Mice, Nude; Neoplasm Transplantation; Oligonucleotide Array Sequence Analysis; Phosphorylation; Proto-Oncogene Proteins c-myc; Repressor Proteins; RNA Interference; RNA, Small Interfering; Transplantation, Heterologous

2018
CDP-choline accumulation in breast and colorectal cancer cells treated with a GSK-3-targeting inhibitor.
    Magma (New York, N.Y.), 2019, Volume: 32, Issue:2

    Topics: Biomarkers, Tumor; Breast Neoplasms; Cell Line, Tumor; Choline; Colorectal Neoplasms; Cytidine Diphosphate Choline; Enzyme Inhibitors; Female; Glucose; Glycogen Synthase Kinase 3; Humans; Indoles; Lipid Metabolism; Magnetic Resonance Spectroscopy; Maleimides; Phosphatidylcholines

2019
B-cell translocation gene 3 overexpression inhibits proliferation and invasion of colorectal cancer SW480 cells via Wnt/β-catenin signaling pathway.
    Neoplasma, 2016, Volume: 63, Issue:5

    Topics: Caco-2 Cells; Cell Cycle Proteins; Cell Line, Tumor; Cell Movement; Cell Proliferation; Colorectal Neoplasms; Cyclin D1; Cyclin E; Epithelial-Mesenchymal Transition; G1 Phase Cell Cycle Checkpoints; Glycogen Synthase Kinase 3 beta; Humans; Indoles; Ki-67 Antigen; Maleimides; Neoplasm Invasiveness; Oncogene Proteins; Proliferating Cell Nuclear Antigen; Proteins; RNA Interference; RNA, Small Interfering; Wnt Signaling Pathway

2016
The proinvasive activity of Wnt-2 is mediated through a noncanonical Wnt pathway coupled to GSK-3beta and c-Jun/AP-1 signaling.
    FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2005, Volume: 19, Issue:1

    Topics: Animals; Antibodies, Monoclonal; Axin Protein; Cell Line; Cell Line, Transformed; Cell Line, Tumor; Colonic Neoplasms; Colorectal Neoplasms; Dogs; Epithelial Cells; Genetic Vectors; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Glycoproteins; Heterotrimeric GTP-Binding Proteins; HT29 Cells; Humans; Indoles; Intercellular Signaling Peptides and Proteins; Intracellular Signaling Peptides and Proteins; Kidney; Kidney Neoplasms; Ligands; Lithium Chloride; Maleimides; Matrix Metalloproteinase 7; Neoplasm Invasiveness; Peptide Fragments; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-jun; Repressor Proteins; Retroviridae; Signal Transduction; Transcription Factor AP-1; Transcription, Genetic; Wnt Proteins; Wnt2 Protein

2005
Pharmacologic modulation of glycogen synthase kinase-3beta promotes p53-dependent apoptosis through a direct Bax-mediated mitochondrial pathway in colorectal cancer cells.
    Cancer research, 2005, Oct-01, Volume: 65, Issue:19

    Topics: Aminophenols; Apoptosis; Apoptosis Regulatory Proteins; bcl-2-Associated X Protein; Colorectal Neoplasms; Enzyme Inhibitors; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; HCT116 Cells; Humans; Indoles; Maleimides; Mitochondria; Protein Conformation; Proto-Oncogene Proteins; RNA Interference; Transcriptional Activation; Tumor Suppressor Protein p53

2005