sb 216763 has been researched along with Cocaine Abuse in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (50.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Gould, TJ; Harper, LJ; Miller, JS; Poole, RL; Shi, X; Unterwald, EM | 1 |
Miller, JS; Tallarida, RJ; Unterwald, EM | 1 |
2 other study(ies) available for sb 216763 and Cocaine Abuse
Article | Year |
---|---|
Reactivation of cocaine reward memory engages the Akt/GSK3/mTOR signaling pathway and can be disrupted by GSK3 inhibition.
Topics: Animals; Cocaine-Related Disorders; Conditioning, Operant; Cues; Enzyme Inhibitors; Fear; Glycogen Synthase Kinase 3; Hippocampus; Indoles; Limbic System; Male; Maleimides; Mechanistic Target of Rapamycin Complex 1; Memory; Mice; Multiprotein Complexes; Neostriatum; Oncogene Protein v-akt; Phosphorylation; Prefrontal Cortex; Reward; Signal Transduction; TOR Serine-Threonine Kinases | 2014 |
Cocaine-induced hyperactivity and sensitization are dependent on GSK3.
Topics: Animals; Caudate Nucleus; Cocaine; Cocaine-Related Disorders; Enzyme Activation; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Hyperkinesis; Indoles; Locomotion; Male; Maleimides; Mice; Phosphorylation; Protein Kinase Inhibitors; Protein Processing, Post-Translational; Putamen; Stereotyped Behavior; Valproic Acid | 2009 |