Page last updated: 2024-09-05

sb 216763 and Cocaine Abuse

sb 216763 has been researched along with Cocaine Abuse in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (50.00)29.6817
2010's1 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Gould, TJ; Harper, LJ; Miller, JS; Poole, RL; Shi, X; Unterwald, EM1
Miller, JS; Tallarida, RJ; Unterwald, EM1

Other Studies

2 other study(ies) available for sb 216763 and Cocaine Abuse

ArticleYear
Reactivation of cocaine reward memory engages the Akt/GSK3/mTOR signaling pathway and can be disrupted by GSK3 inhibition.
    Psychopharmacology, 2014, Volume: 231, Issue:16

    Topics: Animals; Cocaine-Related Disorders; Conditioning, Operant; Cues; Enzyme Inhibitors; Fear; Glycogen Synthase Kinase 3; Hippocampus; Indoles; Limbic System; Male; Maleimides; Mechanistic Target of Rapamycin Complex 1; Memory; Mice; Multiprotein Complexes; Neostriatum; Oncogene Protein v-akt; Phosphorylation; Prefrontal Cortex; Reward; Signal Transduction; TOR Serine-Threonine Kinases

2014
Cocaine-induced hyperactivity and sensitization are dependent on GSK3.
    Neuropharmacology, 2009, Volume: 56, Issue:8

    Topics: Animals; Caudate Nucleus; Cocaine; Cocaine-Related Disorders; Enzyme Activation; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Hyperkinesis; Indoles; Locomotion; Male; Maleimides; Mice; Phosphorylation; Protein Kinase Inhibitors; Protein Processing, Post-Translational; Putamen; Stereotyped Behavior; Valproic Acid

2009