Page last updated: 2024-09-05

sb 216763 and Carcinoma, Medullary

sb 216763 has been researched along with Carcinoma, Medullary in 1 studies

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chen, H; Kunnimalaiyaan, M; Ndiaye, MA; Vaccaro, AM1

Other Studies

1 other study(ies) available for sb 216763 and Carcinoma, Medullary

ArticleYear
Inactivation of glycogen synthase kinase-3beta, a downstream target of the raf-1 pathway, is associated with growth suppression in medullary thyroid cancer cells.
    Molecular cancer therapeutics, 2007, Volume: 6, Issue:3

    Topics: Adjuvants, Immunologic; Animals; Apoptosis; Carcinoma, Medullary; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Chromogranin A; Cyclin D1; Cyclin-Dependent Kinase Inhibitor p15; Cyclin-Dependent Kinase Inhibitor p21; Cyclin-Dependent Kinase Inhibitor p27; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Humans; Indoles; Lithium Chloride; Maleimides; Mice; Mice, Nude; NIH 3T3 Cells; Phosphorylation; Proto-Oncogene Proteins c-raf; Signal Transduction; Thyroid Neoplasms; Xenograft Model Antitumor Assays

2007