sb 203580 has been researched along with Shock, Septic in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (16.67) | 18.2507 |
| 2000's | 4 (66.67) | 29.6817 |
| 2010's | 1 (16.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Guo, X; Liu, Y; Su, L; Wang, Z; Wu, F; Xu, Q | 1 |
| Khan, AA; Priya, S; Saha, B | 1 |
| Bland, KI; Chaudry, IH; Jarrar, D; Kuebler, JF; Matalon, S; Rue, LW; Wang, P | 1 |
| Huang, Q; Huang, X; Jiang, Y; Kan, W; Qin, Q; Wang, J; Wang, S; Yan, W; Zhao, KS | 1 |
| Adams, JL; Badger, AM; Bradbeer, JN; Griswold, DE; Lee, JC; Votta, B | 1 |
| Huang, Q; Jiang, Y; Kan, W; Wang, J; Wang, S; Yan, W; Zhao, K | 1 |
6 other study(ies) available for sb 203580 and Shock, Septic
| Article | Year |
|---|---|
[Mechanism of continuous venovenous hemofiltration combined with ulinastatin for the treatment of septic shock].
Topics: Actins; Cells, Cultured; Glycoproteins; Hemofiltration; Human Umbilical Vein Endothelial Cells; Humans; Imidazoles; MAP Kinase Signaling System; p38 Mitogen-Activated Protein Kinases; Pyridines; Shock, Septic | 2015 |
IL-2 regulates SEB induced toxic shock syndrome in BALB/c mice.
Topics: Animals; Disease Susceptibility; Enterotoxins; Enzyme Activation; Imidazoles; Interferon-gamma; Interleukin-12; Interleukin-2; Macrophages, Peritoneal; Mice; Mice, Inbred BALB C; Mice, Knockout; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; Pyridines; Recombinant Proteins; Shock, Septic; T-Lymphocytes; Tumor Necrosis Factor-alpha | 2009 |
Alveolar macrophage activation after trauma-hemorrhage and sepsis is dependent on NF-kappaB and MAPK/ERK mechanisms.
Topics: Animals; Cecum; Chemokine CXCL2; Enzyme Inhibitors; Extravascular Lung Water; Flavonoids; Hemorrhage; Imidazoles; In Vitro Techniques; Interleukin-6; Lipopolysaccharides; Macrophages, Alveolar; Male; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Monokines; NF-kappa B; Peroxidase; Pyridines; Rats; Rats, Sprague-Dawley; Respiratory Distress Syndrome; Shock, Septic; Tumor Necrosis Factor-alpha | 2002 |
Lung, spleen, and kidney are the major places for inducible nitric oxide synthase expression in endotoxic shock: role of p38 mitogen-activated protein kinase in signal transduction of inducible nitric oxide synthase expression.
Topics: Animals; Blood Pressure; Blotting, Western; DNA Primers; Dose-Response Relationship, Drug; Endotoxins; Enzyme Inhibitors; Imidazoles; Kidney; Lipopolysaccharides; Lung; Male; MAP Kinase Signaling System; Mice; Mice, Inbred BALB C; Mitogen-Activated Protein Kinases; Nitric Oxide; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; p38 Mitogen-Activated Protein Kinases; Polymerase Chain Reaction; Pyridines; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Shock, Septic; Signal Transduction; Spleen; Time Factors; Tissue Distribution; Up-Regulation | 2004 |
Pharmacological profile of SB 203580, a selective inhibitor of cytokine suppressive binding protein/p38 kinase, in animal models of arthritis, bone resorption, endotoxin shock and immune function.
Topics: Animals; Arachidonic Acid; Arthritis; Bone Resorption; Calcium-Calmodulin-Dependent Protein Kinases; Collagen; Disease Models, Animal; Enzyme Inhibitors; Female; Imidazoles; Male; Mice; Mice, Inbred Strains; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Inbred Lew; Shock, Septic; Tumor Necrosis Factor-alpha | 1996 |
Role of p38 MAPK in ICAM-1 expression of vascular endothelial cells induced by lipopolysaccharide.
Topics: Animals; Blood Pressure; Cell Line; Dose-Response Relationship, Drug; Endothelium, Vascular; Enzyme Inhibitors; Humans; Imidazoles; Intercellular Adhesion Molecule-1; Lipopolysaccharides; Lung; Male; Mice; Mice, Inbred BALB C; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Pyridines; RNA, Messenger; Shock, Septic; Time Factors | 2002 |