Page last updated: 2024-09-05

sb 203580 and Leucocythaemia

sb 203580 has been researched along with Leucocythaemia in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's6 (85.71)29.6817
2010's1 (14.29)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Han, Z; Lu, S; Ma, F; Ren, Q; Tian, K; Yang, S; Zhang, L1
Araragi, S; Higashimoto, M; Kondoh, M; Sato, M; Takiguchi, M; Tasaki, E; Watanabe, Y1
Dent, P; Grant, S; Rahmani, M; Yu, C1
Gocek, E; Kiełbiński, M; Marcinkowska, E1
Caravatta, L; Cataldi, A; di Giacomo, V; Di Pietro, R; Rana, R; Sancilio, S1
Berger, F; Champelovier, P; El Atifi, M; Pautre, V; Rostaing, B; Seigneurin, D1
Fan, C; Liu, G; Liu, RY; Olashaw, NE; Zuckerman, KS1

Other Studies

7 other study(ies) available for sb 203580 and Leucocythaemia

ArticleYear
p38 MAPK contributes to the growth inhibition of leukemic tumor cells mediated by human umbilical cord mesenchymal stem cells.
    Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 2010, Volume: 26, Issue:6

    Topics: Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; Humans; Imidazoles; Leukemia; Mesenchymal Stem Cells; Mitogen-Activated Protein Kinase 14; Phosphorylation; Pyridines; RNA Interference; RNA, Small Interfering; Signal Transduction; Umbilical Cord

2010
Requirement of caspase and p38MAPK activation in zinc-induced apoptosis in human leukemia HL-60 cells.
    European journal of biochemistry, 2002, Volume: 269, Issue:24

    Topics: Apoptosis; Blotting, Western; Caspases; Cell Death; Cell Line; Dose-Response Relationship, Drug; Enzyme Inhibitors; HL-60 Cells; Humans; Imidazoles; In Situ Nick-End Labeling; Ionophores; Leukemia; Mitogen-Activated Protein Kinases; Necrosis; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Pyridines; Thiones; Time Factors; Zinc

2002
The hierarchical relationship between MAPK signaling and ROS generation in human leukemia cells undergoing apoptosis in response to the proteasome inhibitor Bortezomib.
    Experimental cell research, 2004, May-01, Volume: 295, Issue:2

    Topics: Anthracenes; Antineoplastic Agents; Apoptosis; Boronic Acids; Bortezomib; Caspases; Cytochromes c; Enzyme Activation; Enzyme Inhibitors; Humans; Imidazoles; JNK Mitogen-Activated Protein Kinases; Jurkat Cells; Leukemia; MAP Kinase Signaling System; Membrane Potentials; Mitochondria; Mitogen-Activated Protein Kinase Kinases; Mitogen-Activated Protein Kinases; Models, Biological; Protease Inhibitors; Pyrazines; Pyridines; Reactive Oxygen Species; Signal Transduction; U937 Cells

2004
Activation of intracellular signaling pathways is necessary for an increase in VDR expression and its nuclear translocation.
    FEBS letters, 2007, May-01, Volume: 581, Issue:9

    Topics: Active Transport, Cell Nucleus; Calcitriol; Cell Nucleus; Cells, Cultured; Chromones; Enzyme Inhibitors; Fatty Acids, Unsaturated; Flavonoids; Gene Expression Regulation; HL-60 Cells; Humans; Imidazoles; Leukemia; Models, Biological; Morpholines; Protein Biosynthesis; Pyridines; Receptors, Calcitriol; Signal Transduction

2007
PI3-K/Akt-dependent activation of cAMP-response element-binding (CREB) protein in Jurkat T leukemia cells treated with TRAIL.
    Journal of cellular physiology, 2008, Volume: 214, Issue:1

    Topics: Apoptosis; Caspase 3; Cell Culture Techniques; Chromones; Clone Cells; Cyclic AMP Response Element-Binding Protein; Dose-Response Relationship, Drug; Enzyme Inhibitors; Enzyme-Linked Immunosorbent Assay; Fluorescein-5-isothiocyanate; Fluorescent Antibody Technique, Indirect; Fluorescent Dyes; Histidine; Humans; Imidazoles; Indoles; Jurkat Cells; Leukemia; Morpholines; Necrosis; p38 Mitogen-Activated Protein Kinases; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Pyridines; Recombinant Proteins; RNA, Small Interfering; T-Lymphocytes; TNF-Related Apoptosis-Inducing Ligand; Transfection

2008
Specific inhibition of basal mitogen-activated protein kinases and phosphatidylinositol 3 kinase activities in leukemia cells: a possible therapeutic role for the kinase inhibitors.
    Experimental hematology, 2008, Volume: 36, Issue:1

    Topics: Anthracenes; Apoptosis; Cell Cycle; Cell Cycle Proteins; Cell Differentiation; Cell Division; Cell Line, Tumor; Chromones; Drug Screening Assays, Antitumor; Drug Synergism; Flavonoids; Gene Expression Regulation, Leukemic; HL-60 Cells; Humans; Imidazoles; Leukemia; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Monocytes; Morpholines; Neoplasm Proteins; Phosphoinositide-3 Kinase Inhibitors; Protein Kinase Inhibitors; Pyridines; U937 Cells

2008
Activation of p38 mitogen-activated protein kinase is required for tumor necrosis factor-alpha -supported proliferation of leukemia and lymphoma cell lines.
    The Journal of biological chemistry, 2000, Jul-14, Volume: 275, Issue:28

    Topics: Cell Division; Cell Nucleus; Cell Survival; Enzyme Activation; Enzyme Inhibitors; Genes, Reporter; Granulocyte-Macrophage Colony-Stimulating Factor; Humans; Imidazoles; Kinetics; Leukemia; Lymphoma; Mitogen-Activated Protein Kinases; NF-kappa B; p38 Mitogen-Activated Protein Kinases; Pyridines; Recombinant Proteins; Signal Transduction; Transfection; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha

2000