Page last updated: 2024-09-05

sb 203580 and Left Ventricular Dysfunction

sb 203580 has been researched along with Left Ventricular Dysfunction in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's6 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bassi, R; Bellahcene, M; Cao, X; Dighe, K; Gorog, DA; Kabir, AM; Marber, MS; Quinlan, RA; Tanno, M1
Birkle, D; Failinger, C; Finkel, MS; Jain, AC; Kan, H; Xie, S1
Akita, Y; Enoki, C; Fujiwara, H; Imamura, H; Iwasaka, T; Kamihata, H; Kyoi, S; Matsuhisa, S; Otani, H; Tatsumi, K1
Bücker-Gärtner, C; Linderer, A; Pauschinger, M; Riad, A; Rutschow, S; Schultheiss, HP; Sobirey, M; Tschöpe, C; Van Linthout, S; Westermann, D1
Adams, V; Döring, C; Erbs, S; Hambrecht, R; Kränkel, N; Labeit, S; Linke, A; Müller-Werdan, U; Schuler, G; Wisloff, U; Witt, CC1
Duan, J; Norby, FL; Relling, DP; Ren, J; Wold, LE1

Other Studies

6 other study(ies) available for sb 203580 and Left Ventricular Dysfunction

ArticleYear
Inhibition of p38 MAPK activity fails to attenuate contractile dysfunction in a mouse model of low-flow ischemia.
    Cardiovascular research, 2004, Jan-01, Volume: 61, Issue:1

    Topics: Animals; Blotting, Western; Enzyme Activation; Imidazoles; Male; Mice; Mice, Inbred C57BL; Mitogen-Activated Protein Kinases; Myocardial Contraction; Myocardial Stunning; Myocardium; p38 Mitogen-Activated Protein Kinases; Perfusion; Pyridines; Ventricular Dysfunction, Left

2004
p38 MAP kinase inhibitor reverses stress-induced cardiac myocyte dysfunction.
    Journal of applied physiology (Bethesda, Md. : 1985), 2005, Volume: 98, Issue:1

    Topics: Animals; Cells, Cultured; Female; Imidazoles; Male; Muscle Cells; Myocardial Contraction; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Sprague-Dawley; Stress, Physiological; Treatment Outcome; Ultrasonography; Ventricular Dysfunction, Left

2005
Opposing effect of p38 MAP kinase and JNK inhibitors on the development of heart failure in the cardiomyopathic hamster.
    Cardiovascular research, 2006, Mar-01, Volume: 69, Issue:4

    Topics: Animals; Anthracenes; Apoptosis; Cardiomyopathy, Dilated; Cricetinae; Fibrosis; Imidazoles; Immunoblotting; In Situ Nick-End Labeling; JNK Mitogen-Activated Protein Kinases; Male; Microscopy, Fluorescence; Myocardium; p38 Mitogen-Activated Protein Kinases; Pyrazoles; Pyridines; Ventricular Dysfunction, Left

2006
Inhibition of p38 mitogen-activated protein kinase attenuates left ventricular dysfunction by mediating pro-inflammatory cardiac cytokine levels in a mouse model of diabetes mellitus.
    Diabetologia, 2006, Volume: 49, Issue:10

    Topics: Animals; Body Weight; Cytokines; Diabetes Mellitus, Experimental; Diabetic Angiopathies; Enzyme Inhibitors; Heart; Imidazoles; Inflammation; Male; Mice; Mice, Inbred C57BL; Organ Size; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Pyridines; Systole; Ventricular Dysfunction, Left; Ventricular Function, Left

2006
Myocardial expression of Murf-1 and MAFbx after induction of chronic heart failure: Effect on myocardial contractility.
    Cardiovascular research, 2007, Jan-01, Volume: 73, Issue:1

    Topics: Animals; Animals, Newborn; Blotting, Western; Echocardiography; Flavonoids; Gene Expression; Heart Failure; Imidazoles; Mitogen-Activated Protein Kinase 1; Models, Animal; Muscle Proteins; Myocardium; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Inbred WKY; RNA Interference; RNA, Messenger; SKP Cullin F-Box Protein Ligases; Tripartite Motif Proteins; Troponin I; Tumor Necrosis Factor-alpha; Ubiquitin-Protein Ligases; Ventricular Dysfunction, Left

2007
Abrogated leptin-induced cardiac contractile response in ventricular myocytes under spontaneous hypertension: role of Jak/STAT pathway.
    Hypertension (Dallas, Tex. : 1979), 2002, Volume: 39, Issue:1

    Topics: Animals; Blood Pressure; Calcium; Carrier Proteins; Cell Size; DNA-Binding Proteins; Enzyme Inhibitors; Heart Ventricles; Hypertension; Imidazoles; Janus Kinase 2; Leptin; Mitogen-Activated Protein Kinase Kinases; Myocardial Contraction; NG-Nitroarginine Methyl Ester; Nitric Oxide; Nitric Oxide Synthase; Phosphorylation; Protein-Tyrosine Kinases; Proto-Oncogene Proteins; Pyridines; Rats; Rats, Inbred SHR; Rats, Inbred WKY; Receptors, Cell Surface; Receptors, Leptin; Signal Transduction; STAT3 Transcription Factor; Trans-Activators; Tyrphostins; Ventricular Dysfunction, Left

2002