sb 203580 has been researched along with Injury, Myocardial Reperfusion in 19 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (10.53) | 18.2507 |
| 2000's | 7 (36.84) | 29.6817 |
| 2010's | 10 (52.63) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cha, L; Dai, Y; Han, B; Li, J; Li, X; Mu, J; Shi, Y; Yan, S | 1 |
| Chen, T; Liu, F; Wang, D | 1 |
| Fu, W; Hu, X; Jiang, H; Wang, J; Xie, J; Xu, W; Yang, H; Zhou, X | 1 |
| Hu, X; Jiang, H; Wang, J; Xie, J; Zhou, X | 1 |
| Chattipakorn, N; Chattipakorn, SC; Kumphune, S; Surinkaew, S | 1 |
| Chauski, EI; Khaliulin, IG; Maslov, LN; Mukhomedzyanov, AV; Portnichenko, AG; Tsibulnikov, SY | 1 |
| Li, G; Qian, W; Zhao, C | 1 |
| Li, D; Luo, Y; Ma, Y; Pan, D; Xu, T; Xuan, H; Zhang, Y; Zhu, H; Zhu, S | 1 |
| Hamilton, L; Keeven, J; Kurian, MV; Mehl, P; Mullins, JM | 1 |
| Chattipakorn, N; Chattipakorn, S; Kumphune, S; Surinkaew, S | 1 |
| Bouchard, JF; Lagneux, C; Lamontagne, D; Lépicier, P | 1 |
| Ballard-Croft, C; Keith, BJ; Kristo, G; Lasley, RD; Mentzer, RM; Reid, E; Yoshimura, Y | 1 |
| Hartley, S; Lochner, A; Marais, E; Moolman, JA; Van Wyk, J | 1 |
| Chilian, WM; Kolz, C; Potter, B; Reed, R; Rocic, P | 1 |
| Cheng, G; Padbury, JF; Tseng, YT; Zhang, LX; Zhao, TC | 1 |
| Ali, IS; Currie, RW; Li, G | 1 |
| Das, DK; Maulik, N; Price, BD; Sato, M | 1 |
| Christopher, TA; Feuerstein, GZ; Gao, F; Kumar, S; Lee, JC; Lopez, BL; Louden, CS; Ma, XL; Wang, C; Yue, TL | 1 |
| Feuerstein, GZ; Gu, JL; Kumar, S; Lee, JC; Ma, XL; Maleeff, B; Ohlstein, EH; Thomas, H; Wang, C; Yue, TL | 1 |
19 other study(ies) available for sb 203580 and Injury, Myocardial Reperfusion
| Article | Year |
|---|---|
Down-regulation of lncRNA KCNQ1OT1 protects against myocardial ischemia/reperfusion injury following acute myocardial infarction.
Topics: Acute Disease; Apoptosis; Cells, Cultured; Down-Regulation; Humans; Imidazoles; Myocardial Infarction; Myocardial Reperfusion Injury; Potassium Channels, Voltage-Gated; Pyridines; RNA, Long Noncoding | 2017 |
Betulinic acid alleviates myocardial hypoxia/reoxygenation injury via inducing Nrf2/HO-1 and inhibiting p38 and JNK pathways.
Topics: Animals; Anthracenes; Apoptosis; Betulinic Acid; Cell Hypoxia; Cell Survival; Heme Oxygenase-1; Imidazoles; JNK Mitogen-Activated Protein Kinases; Myocardial Reperfusion Injury; Myocytes, Cardiac; NF-E2-Related Factor 2; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Pentacyclic Triterpenes; Protective Agents; Pyridines; Rats; RNA, Small Interfering; Signal Transduction; Triterpenes | 2018 |
Dobutamine-mediated heme oxygenase-1 induction via PI3K and p38 MAPK inhibits high mobility group box 1 protein release and attenuates rat myocardial ischemia/reperfusion injury in vivo.
Topics: Adrenergic beta-1 Receptor Agonists; Animals; Chromones; Disease Models, Animal; Dobutamine; Enzyme Inhibitors; Heart; Heme Oxygenase-1; HMGB1 Protein; Imidazoles; Male; Morpholines; Myocardial Reperfusion Injury; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Phosphatidylinositol 3-Kinases; Protoporphyrins; Pyridines; Rats; Rats, Sprague-Dawley | 2013 |
[Selectively stimulating β1-adrenergic receptor attenuates rat myocardial ischemia/reperfusion injury in vivo by inhibiting high mobility group box 1 protein release].
Topics: Animals; Chromones; Dobutamine; HMGB1 Protein; Imidazoles; Interleukin-6; Male; Morpholines; Myocardial Infarction; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; NF-kappa B; Oxidative Stress; Pyridines; Rats; Rats, Sprague-Dawley; Receptors, Adrenergic; Reperfusion Injury; Superoxide Dismutase; Tumor Necrosis Factor-alpha | 2014 |
Inhibition of p38 MAPK activation protects cardiac mitochondria from ischemia/reperfusion injury.
Topics: Animals; Cardiotonic Agents; Enzyme Activation; Enzyme Inhibitors; Imidazoles; Male; Mitochondria, Heart; Myocardial Reperfusion Injury; Myocytes, Cardiac; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Wistar | 2015 |
Role of MEK, PI3, p38, Tyrosine, and mTOR Kinases in Regulation of Heart Resistance to the Arrhythmogenic Action of Short-Term Ischemia and Reperfusion.
Topics: Animals; Arrhythmias, Cardiac; Flavonoids; Imidazoles; MAP Kinase Kinase Kinases; Myocardial Reperfusion Injury; p38 Mitogen-Activated Protein Kinases; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Pyridines; Rats; Rats, Wistar; Sirolimus; TOR Serine-Threonine Kinases | 2015 |
Analyzing the anti-ischemia-reperfusion injury effects of ginsenoside Rb1 mediated through the inhibition of p38α MAPK.
Topics: Animals; Cardiotonic Agents; Caspase 3; Ginsenosides; Imidazoles; Male; Mitogen-Activated Protein Kinase 14; Myocardial Infarction; Myocardial Reperfusion Injury; Phosphorylation; Protein Kinase Inhibitors; Pyridines; Rats; Rats, Sprague-Dawley; Tumor Necrosis Factor-alpha | 2016 |
Luteolin Enhances Sarcoplasmic Reticulum Ca2+-ATPase Activity through p38 MAPK Signaling thus Improving Rat Cardiac Function after Ischemia/Reperfusion.
Topics: Animals; Calcium-Binding Proteins; Cardiotonic Agents; Gene Expression Regulation; Imidazoles; Luteolin; Male; Membrane Potential, Mitochondrial; Myocardial Reperfusion Injury; Myocardium; Myocytes, Cardiac; Organ Culture Techniques; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Primary Cell Culture; Pyridines; Rats; Rats, Wistar; Sarcoplasmic Reticulum; Sarcoplasmic Reticulum Calcium-Transporting ATPases; Signal Transduction | 2017 |
Enhanced cell survival and diminished apoptotic response to simulated ischemia-reperfusion in H9c2 cells by magnetic field preconditioning.
Topics: Animals; Apoptosis; Caspases; Cell Line; Cell Nucleus; Cell Survival; Cytosol; Fluorescent Antibody Technique; Heme Oxygenase (Decyclizing); HSP27 Heat-Shock Proteins; HSP72 Heat-Shock Proteins; Imidazoles; Ischemic Preconditioning; Magnetic Fields; Myocardial Reperfusion Injury; Phosphorylation; Proto-Oncogene Proteins c-bcl-2; Pyridines; Rats; Stress, Physiological | 2012 |
Inhibition of p38 MAPK during ischemia, but not reperfusion, effectively attenuates fatal arrhythmia in ischemia/reperfusion heart.
Topics: Administration, Intravenous; Animals; Arrhythmias, Cardiac; Cytochromes c; Disease Models, Animal; Enzyme Inhibitors; Imidazoles; Male; Myocardial Infarction; Myocardial Reperfusion Injury; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Pyridines; Rats; Rats, Wistar; Time Factors; Ventricular Function | 2013 |
Endocannabinoids protect the rat isolated heart against ischaemia.
Topics: Amides; Animals; Arachidonic Acids; Biomarkers; Blotting, Western; Camphanes; Cannabinoid Receptor Modulators; Endocannabinoids; Ethanolamines; Glycerides; Heart; Imidazoles; L-Lactate Dehydrogenase; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Myocardial Infarction; Myocardial Ischemia; Myocardial Reperfusion Injury; p38 Mitogen-Activated Protein Kinases; Palmitic Acids; Piperidines; Protein Kinase C; Pyrazoles; Pyridines; Rats; Rats, Sprague-Dawley; Rimonabant; Signal Transduction | 2003 |
Acute adenosine preconditioning is mediated by p38 MAPK activation in discrete subcellular compartments.
Topics: Adenosine; Animals; Blood Pressure; Cell Compartmentation; Enzyme Inhibitors; Heart Rate; Imidazoles; Ischemic Preconditioning, Myocardial; Male; Myocardial Reperfusion Injury; Myocardium; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Sprague-Dawley | 2005 |
Inhibition of myocardial apoptosis by ischaemic and beta-adrenergic preconditioning is dependent on p38 MAPK.
Topics: Adrenergic beta-Agonists; Animals; Anisomycin; Apoptosis; Enzyme Activators; Imidazoles; In Vitro Techniques; Ischemic Preconditioning, Myocardial; Isoproterenol; Models, Animal; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Necrosis; p38 Mitogen-Activated Protein Kinases; Protein Kinase Inhibitors; Pyridines; Rats | 2006 |
Optimal reactive oxygen species concentration and p38 MAP kinase are required for coronary collateral growth.
Topics: Acetophenones; Animals; Blood Flow Velocity; Cells, Cultured; Collateral Circulation; Coronary Circulation; Coronary Vessels; Disease Models, Animal; Ditiocarb; Endothelial Cells; Enzyme Inhibitors; Humans; Imidazoles; Ligation; Male; MAP Kinase Signaling System; Myocardial Reperfusion Injury; NADPH Oxidases; Neovascularization, Physiologic; Onium Compounds; Oxygenases; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Inbred WKY; Reactive Oxygen Species; Superoxide Dismutase; Vascular Endothelial Growth Factor A | 2007 |
Inhibition of histone deacetylases triggers pharmacologic preconditioning effects against myocardial ischemic injury.
Topics: Animals; Disease Models, Animal; Enzyme Inhibitors; Histone Deacetylase Inhibitors; Histone Deacetylases; Hydroxamic Acids; Imidazoles; Ischemic Preconditioning, Myocardial; Male; Mice; Mice, Inbred ICR; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; p38 Mitogen-Activated Protein Kinases; Pyridines; Signal Transduction; Ventricular Function, Left | 2007 |
Insulin-induced myocardial protection in isolated ischemic rat hearts requires p38 MAPK phosphorylation of Hsp27.
Topics: Animals; Blotting, Western; Densitometry; Disease Models, Animal; Dystrophin; Heat-Shock Proteins; HSP27 Heat-Shock Proteins; HSP70 Heat-Shock Proteins; Hypothermia, Induced; Imidazoles; Insulin; Male; Microscopy, Fluorescence; Myocardial Contraction; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; Neoplasm Proteins; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase Inhibitors; Pyridines; Rats; Rats, Sprague-Dawley; Signal Transduction; Time Factors; Tubulin; Ventricular Function, Left | 2008 |
An essential role of NFkappaB in tyrosine kinase signaling of p38 MAP kinase regulation of myocardial adaptation to ischemia.
Topics: Adaptation, Physiological; Animals; Calcium-Calmodulin-Dependent Protein Kinases; Creatine Kinase; Genistein; Heart; Imidazoles; In Vitro Techniques; Malondialdehyde; Mitogen-Activated Protein Kinases; Myocardial Ischemia; Myocardial Reperfusion Injury; NF-kappa B; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Peptides; Protein-Tyrosine Kinases; Pyridines; Rats; Rats, Sprague-Dawley; Signal Transduction | 1998 |
Inhibition of p38 mitogen-activated protein kinase decreases cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion.
Topics: Animals; Apoptosis; Calcium-Calmodulin-Dependent Protein Kinases; DNA Fragmentation; Dose-Response Relationship, Drug; Enzyme Activation; Enzyme Inhibitors; Imidazoles; In Situ Nick-End Labeling; In Vitro Techniques; Mitogen-Activated Protein Kinases; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; Necrosis; p38 Mitogen-Activated Protein Kinases; Pyridines; Rabbits; Time Factors | 1999 |
Inhibition of extracellular signal-regulated kinase enhances Ischemia/Reoxygenation-induced apoptosis in cultured cardiac myocytes and exaggerates reperfusion injury in isolated perfused heart.
Topics: Animals; Animals, Newborn; Apoptosis; Calcium-Calmodulin-Dependent Protein Kinases; Cells, Cultured; Enzyme Activation; Enzyme Inhibitors; Flavonoids; Heart; Imidazoles; In Vitro Techniques; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinases; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; p38 Mitogen-Activated Protein Kinases; Pyridines; Rats; Rats, Sprague-Dawley; Recovery of Function | 2000 |